Inhibition of NAD(P)H oxidase activity blocks vascular endothelial growth factor overexpression and neovascularization during ischemic retinopathy.
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NADPH oxidase inhibitors: a decade of discovery from Nox2ds to HTSThe "two-faced" effects of reactive oxygen species and the lipid peroxidation product 4-hydroxynonenal in the hallmarks of cancerCellular and molecular mechanisms underlying alcohol-induced aggressiveness of breast cancerCYP1B1 expression promotes the proangiogenic phenotype of endothelium through decreased intracellular oxidative stress and thrombospondin-2 expressionDeletion of SPARC Enhances Retinal Vaso-Obliteration in Mouse Model of Oxygen-Induced Retinopathy.Hydroethidine- and MitoSOX-derived red fluorescence is not a reliable indicator of intracellular superoxide formation: another inconvenient truthReduced nitro-oxidative stress and neural cell death suggests a protective role for microglial cells in TNFalpha-/- mice in ischemic retinopathy.Vascular dysfunction in retinopathy-an emerging role for arginaseThe quest for selective nox inhibitors and therapeutics: challenges, triumphs and pitfallsNADPH oxidase, NOX1, mediates vascular injury in ischemic retinopathy.HMG-CoA reductase inhibitors (statin) prevents retinal neovascularization in a model of oxygen-induced retinopathy.Role of NADPH oxidase and Stat3 in statin-mediated protection against diabetic retinopathy.Novel role of NADPH oxidase in angiogenesis and stem/progenitor cell function.NAD(P)H oxidase-dependent regulation of CCL2 production during retinal inflammation.Studies on the pathogenesis of avascular retina and neovascularization into the vitreous in peripheral severe retinopathy of prematurity (an american ophthalmological society thesis)Nanoceria inhibit the development and promote the regression of pathologic retinal neovascularization in the Vldlr knockout mouseRole of IL-6 in angiotensin II-induced retinal vascular inflammation.Role of ROS in ischemia-induced lung angiogenesisIncreased expression and activity of 12-lipoxygenase in oxygen-induced ischemic retinopathy and proliferative diabetic retinopathy: implications in retinal neovascularization.12/15-Lipoxygenase-derived lipid metabolites induce retinal endothelial cell barrier dysfunction: contribution of NADPH oxidaseDocosahexaenoic acid improves the nitroso-redox balance and reduces VEGF-mediated angiogenic signaling in microvascular endothelial cells.Arginase in retinopathy.Targeting Neovascularization in Ischemic Retinopathy: Recent AdvancesOrgan specific optical imaging of mitochondrial redox state in a rodent model of hereditary hemorrhagic telangiectasia-1A lipidomic screen of hyperglycemia-treated HRECs links 12/15-Lipoxygenase to microvascular dysfunction during diabetic retinopathy via NADPH oxidase.NADPH oxidases in cardiovascular disease: insights from in vivo models and clinical studies.Nox2 B-loop peptide, Nox2ds, specifically inhibits the NADPH oxidase Nox2Neuroprotection from retinal ischemia/reperfusion injury by NOX2 NADPH oxidase deletionNox Inhibitors & Therapies: Rational Design of Peptidic and Small Molecule InhibitorsConstitutive NADPH oxidase 4 activity resides in the composition of the B-loop and the penultimate C terminusNeovascularization in an arterio-venous loop-containing tissue engineering chamber: role of NADPH oxidase.Damaged mitochondrial DNA replication system and the development of diabetic retinopathy.Cannabinoid 1 receptor activation contributes to vascular inflammation and cell death in a mouse model of diabetic retinopathy and a human retinal cell lineMEF2C ablation in endothelial cells reduces retinal vessel loss and suppresses pathologic retinal neovascularization in oxygen-induced retinopathyDownregulating p22phox ameliorates inflammatory response in Angiotensin II-induced oxidative stress by regulating MAPK and NF-κB pathways in ARPE-19 cellsPrediction of diabetic retinopathy: role of oxidative stress and relevance of apoptotic biomarkers.Hyperoxia-Induced Proliferative Retinopathy: Early Interruption of Retinal Vascular Development with Severe and Irreversible Neurovascular Disruption.The twisted survivin connection to angiogenesiseNOS overexpression exacerbates vascular closure in the obliterative phase of OIR and increases angiogenic drive in the subsequent proliferative stageAnti-VEGF therapy in the management of retinopathy of prematurity: what we learn from representative animal models of oxygen-induced retinopathy
P2860
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P2860
Inhibition of NAD(P)H oxidase activity blocks vascular endothelial growth factor overexpression and neovascularization during ischemic retinopathy.
description
2005 nî lūn-bûn
@nan
2005 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Inhibition of NAD(P)H oxidase ...... n during ischemic retinopathy.
@ast
Inhibition of NAD(P)H oxidase ...... n during ischemic retinopathy.
@en
type
label
Inhibition of NAD(P)H oxidase ...... n during ischemic retinopathy.
@ast
Inhibition of NAD(P)H oxidase ...... n during ischemic retinopathy.
@en
prefLabel
Inhibition of NAD(P)H oxidase ...... n during ischemic retinopathy.
@ast
Inhibition of NAD(P)H oxidase ...... n during ischemic retinopathy.
@en
P2093
P2860
P921
P1476
Inhibition of NAD(P)H oxidase ...... on during ischemic retinopathy
@en
P2093
Daniel H Platt
M Ali Behzadian
Manuela Bartoli
Mohamed Al-Shabrawey
Robert W Caldwell
Ruth B Caldwell
Sue Matragoon
P2860
P304
P356
10.1016/S0002-9440(10)63001-5
P407
P577
2005-08-01T00:00:00Z