Chronic intermittent hypoxia increases blood pressure and expression of FosB/DeltaFosB in central autonomic regions.
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Peripheral chemoreception and arterial pressure responses to intermittent hypoxiaNeural Control of Blood Pressure in Chronic Intermittent HypoxiaThe efficacy of antihypertensive drugs in chronic intermittent hypoxia conditions.Chronic intermittent hypoxia induces oxidative stress and inflammation in brain regions associated with early-stage neurodegeneration.Angiotensin converting enzyme 1 in the median preoptic nucleus contributes to chronic intermittent hypoxia hypertension.Role of chemoreception in cardiorespiratory acclimatization to, and deacclimatization from, hypoxiaCarotid body denervation prevents fasting hyperglycemia during chronic intermittent hypoxiaVisualization of oxytocin release that mediates paired pulse facilitation in hypothalamic pathways to brainstem autonomic neurons.Chemoreflexes, sleep apnea, and sympathetic dysregulationObstructive sleep apnea and hypertension: an update.Enhanced carotid body chemosensory activity and the cardiovascular alterations induced by intermittent hypoxiaAngiotensin II type 1a receptors in subfornical organ contribute towards chronic intermittent hypoxia-associated sustained increase in mean arterial pressure.Humans In Hypoxia: A Conspiracy Of Maladaptation?!Glucoregulatory consequences and cardiorespiratory parameters in rats exposed to chronic-intermittent hypoxia: effects of the duration of exposure and losartan.Brain stem activity changes associated with restored sympathetic drive following CPAP treatment in OSA subjects: a longitudinal investigation.Metabolic dysfunction in obstructive sleep apnea: A critical examination of underlying mechanismsAn Essential role for DeltaFosB in the median preoptic nucleus in the sustained hypertensive effects of chronic intermittent hypoxiaChronic and intermittent hypoxia differentially regulate left ventricular inflammatory and extracellular matrix responsesExercise training normalizes enhanced sympathetic activation from the paraventricular nucleus in chronic heart failure: role of angiotensin IISympatho-adrenal activation by chronic intermittent hypoxia.Neurogenic mechanisms underlying the rapid onset of sympathetic responses to intermittent hypoxia.The roles of sensitization and neuroplasticity in the long-term regulation of blood pressure and hypertension.Reduced c-Fos expression in medullary catecholaminergic neurons in rats 20 h after exposure to chronic intermittent hypoxia.Molecular mechanisms of chronic intermittent hypoxia and hypertensionOxytocin neuron activation prevents hypertension that occurs with chronic intermittent hypoxia/hypercapnia in rats.Rats selectively bred for differences in aerobic capacity have similar hypertensive responses to chronic intermittent hypoxia.Central losartan attenuates increases in arterial pressure and expression of FosB/ΔFosB along the autonomic axis associated with chronic intermittent hypoxia.Knockdown of tyrosine hydroxylase in the nucleus of the solitary tract reduces elevated blood pressure during chronic intermittent hypoxia.Chronic intermittent hypoxia increases sympathetic control of blood pressure: role of neuronal activity in the hypothalamic paraventricular nucleus.Parasympathetic preganglionic cardiac motoneurons labeled after voluntary diving.Attenuated dopaminergic tone in the paraventricular nucleus contributing to sympathoexcitation in rats with Type 2 diabetesNeural Control of the Upper Airway: Respiratory and State-Dependent Mechanisms.Parasympathetic Vagal Control of Cardiac Function.Effects of 12 Months Continuous Positive Airway Pressure on Sympathetic Activity Related Brainstem Function and Structure in Obstructive Sleep Apnea.Role of Carotid Body in Intermittent Hypoxia-Related Hypertension.Circulating exosomes in obstructive sleep apnea as phenotypic biomarkers and mechanistic messengers of end-organ morbidity.Transcription factor ΔFosB acts within the nucleus of the solitary tract to increase mean arterial pressure during exposures to intermittent hypoxia.Sex differences in sleep apnea and comorbid neurodegenerative diseases.Role of angiotensin-converting enzyme 1 within the median preoptic nucleus following chronic intermittent hypoxia.Cardiovascular consequences of sleep apnea.
P2860
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P2860
Chronic intermittent hypoxia increases blood pressure and expression of FosB/DeltaFosB in central autonomic regions.
description
2011 nî lūn-bûn
@nan
2011 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
name
Chronic intermittent hypoxia i ...... in central autonomic regions.
@ast
Chronic intermittent hypoxia i ...... in central autonomic regions.
@en
type
label
Chronic intermittent hypoxia i ...... in central autonomic regions.
@ast
Chronic intermittent hypoxia i ...... in central autonomic regions.
@en
prefLabel
Chronic intermittent hypoxia i ...... in central autonomic regions.
@ast
Chronic intermittent hypoxia i ...... in central autonomic regions.
@en
P2093
P2860
P1476
Chronic intermittent hypoxia i ...... in central autonomic regions.
@en
P2093
Flavia R Carreno
Glenn M Toney
J Thomas Cunningham
Joel T Little
Steven W Mifflin
W David Knight
P2860
P304
P356
10.1152/AJPREGU.00830.2010
P577
2011-05-04T00:00:00Z