Inhibition of interferon-gamma signaling in oligodendroglia delays coronavirus clearance without altering demyelination. - Wikidata - wikimedia - TriplyDB

Inhibition of interferon-gamma signaling in oligodendroglia delays coronavirus clearance without altering demyelination.

Inhibition of interferon-gamma signaling in oligodendroglia delays coronavirus clearance without altering demyelination.

http://www.wikidata.org/entity/Q35088074

about

Keeping it in check: chronic viral infection and antiviral immunity in the brain MicroRNA-155 enhances T cell trafficking and antiviral effector function in a model of coronavirus-induced neurologic disease RNase L mediated protection from virus induced demyelination.A protective role for ELR+ chemokines during acute viral encephalomyelitis.CXCR2 signaling protects oligodendrocytes and restricts demyelination in a mouse model of viral-induced demyelination.Gamma interferon signaling in oligodendrocytes is critical for protection from neurotropic coronavirus infection The pathogenesis of murine coronavirus infection of the central nervous system.Target-dependent B7-H1 regulation contributes to clearance of central nervous system infection and dampens morbidity.FTY720 (fingolimod) modulates the severity of viral-induced encephalomyelitis and demyelination.IFN-γ signaling to astrocytes protects from autoimmune mediated neurological disability Promoting remyelination: utilizing a viral model of demyelination to assess cell-based therapies Analysis of the host transcriptome from demyelinating spinal cord of murine coronavirus-infected mice.Enhanced antiviral T cell function in the absence of B7-H1 is insufficient to prevent persistence but exacerbates axonal bystander damage during viral encephalomyelitis.CD4 T cells promote CD8 T cell immunity at the priming and effector site during viral encephalitis CXCR2 signaling and host defense following coronavirus-induced encephalomyelitis.Myd88 Initiates Early Innate Immune Responses and Promotes CD4 T Cells during Coronavirus Encephalomyelitis.Oligodendroglia are limited in type I interferon induction and responsiveness in vivo The chemokine receptor CXCR2 and coronavirus-induced neurologic disease.CD4 T cells contribute to virus control and pathology following central nervous system infection with neurotropic mouse hepatitis virus.NKG2D receptor signaling enhances cytolytic activity by virus-specific CD8+ T cells: evidence for a protective role in virus-induced encephalitis.Generation of a protective T-cell response following coronavirus infection of the central nervous system is not dependent on IL-12/23 signaling.Memory CD4+ T-cell-mediated protection from lethal coronavirus encephalomyelitis.Neuroimmunology of central nervous system viral infections: the cells, molecules and mechanisms involved.Interleukin-12 (IL-12), but not IL-23, deficiency ameliorates viral encephalitis without affecting viral control.IL-21 optimizes T cell and humoral responses in the central nervous system during viral encephalitis.Mouse hepatitis virus infection of the CNS: a model for defense, disease, and repair.IFN-gamma-mediated suppression of coronavirus replication in glial-committed progenitor cells.The Biology of Persistent Infection: Inflammation and Demyelination following Murine Coronavirus Infection of the Central Nervous System Ifit2 deficiency results in uncontrolled neurotropic coronavirus replication and enhanced encephalitis via impaired alpha/beta interferon induction in macrophages.Immune responses to non-tumor antigens in the central nervous system.Sulfocerebrosides upregulate liposome uptake in human astrocytes without inducing a proinflammatory response.Distinct CD4 T-cell effects on primary versus recall CD8 T-cell responses during viral encephalomyelitis.Microglia are required for protection against lethal coronavirus encephalitis in mice.

P2860

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P2860

Inhibition of interferon-gamma signaling in oligodendroglia delays coronavirus clearance without altering demyelination.

http://www.wikidata.org/entity/Q35088074

description

2006 nî lūn-bûn

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2006 թվականի մարտին հրատարակված գիտական հոդված

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Inhibition of interferon-gamma ...... ithout altering demyelination.

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Inhibition of interferon-gamma ...... ithout altering demyelination.

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Inhibition of interferon-gamma ...... ithout altering demyelination.

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Inhibition of interferon-gamma ...... ithout altering demyelination.

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The American Journal of Pathology

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Inhibition of interferon-gamma ...... ithout altering demyelination.

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Chandran Ramakrishna

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David R Hinton

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Jason Hoskin

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Roscoe Atkinson

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Stephen A Stohlman

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P2860

In vivo depletion of CD8+ T cells prevents lesions of demyelination in Semliki Forest virus infection

Relapsing and remitting multiple sclerosis: pathology of the newly forming lesion

Induction of apoptosis in murine coronavirus-infected cultured cells and demonstration of E protein as an apoptosis inducer.

Expression of matrix metalloproteinases and their tissue inhibitor during viral encephalitis.

Contributions of Fas-Fas ligand interactions to the pathogenesis of mouse hepatitis virus in the central nervous system.

Role of viral persistence in retaining CD8(+) T cells within the central nervous system

Macrophage infiltration, but not apoptosis, is correlated with immune-mediated demyelination following murine infection with a neurotropic coronavirus.

Axonal damage is T cell mediated and occurs concomitantly with demyelination in mice infected with a neurotropic coronavirus.

Fas and perforin pathways as major mechanisms of T cell-mediated cytotoxicity.

MHV infection of the CNS: mechanisms of immune-mediated control.

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