In utero exposure to diesel exhaust air pollution promotes adverse intrauterine conditions, resulting in weight gain, altered blood pressure, and increased susceptibility to heart failure in adult mice.
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The shared pathoetiological effects of particulate air pollution and the social environment on fetal-placental developmentPrenatal and postnatal mothering by diesel exhaust PM2.5-exposed dams differentially program mouse energy metabolismProgramming of mouse obesity by maternal exposure to concentrated ambient fine particlesEarly life exposure to air pollution induces adult cardiac dysfunction.Air pollution and adverse cardiac remodeling: clinical effects and basic mechanisms.Prenatal Ambient Air Pollution, Placental Mitochondrial DNA Content, and Birth Weight in the INMA (Spain) and ENVIRONAGE (Belgium) Birth CohortsEffects of Fine Particulate Matter (PM2.5) on Systemic Oxidative Stress and Cardiac Function in ApoE(-/-) Mice.Maternal exposure to diluted diesel engine exhaust alters placental function and induces intergenerational effects in rabbitsNeonatal Diesel Exhaust Particulate Exposure Does Not Predispose Mice to Adult Cardiac Hypertrophy or Heart Failure.Lower Placental Leptin Promoter Methylation in Association with Fine Particulate Matter Air Pollution during Pregnancy and Placental Nitrosative Stress at Birth in the ENVIRONAGE Cohort.Basic mechanisms for adverse cardiovascular events associated with air pollution.PM2.5 exposure in utero contributes to neonatal cardiac dysfunction in mice.Maternal-engineered nanomaterial exposure disrupts progeny cardiac function and bioenergetics.Biomarkers used in studying air pollution exposure during pregnancy and perinatal outcomes: a review.Chronic exposure to air pollution particles increases the risk of obesity and metabolic syndrome: findings from a natural experiment in Beijing.Air Pollution and Other Environmental Modulators of Cardiac Function.In Utero Exposure of Hyperlipidemic Mice to Diesel Exhaust: Lack of Effects on Atherosclerosis in Adult Offspring Fed a Regular Chow Diet.PM2.5 induces embryonic growth retardation: Potential involvement of ROS-MAPKs-apoptosis and G0/G1 arrest pathways.In utero exposure to diesel exhaust particulates is associated with an altered cardiac transcriptional response to transverse aortic constriction and altered DNA methylation.Deletion of HP1γ in cardiac myocytes affects H4K20me3 levels but does not impact cardiac growth.Maternal Exposure to Ambient Particulate Matter ≤2.5 µm During Pregnancy and the Risk for High Blood Pressure in ChildhoodMeta-Prediction of MTHFR Gene Polymorphism and Air Pollution on the Risks of Congenital Heart Defects Worldwide: A Transgenerational Analysis
P2860
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P2860
In utero exposure to diesel exhaust air pollution promotes adverse intrauterine conditions, resulting in weight gain, altered blood pressure, and increased susceptibility to heart failure in adult mice.
description
2014 nî lūn-bûn
@nan
2014 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
In utero exposure to diesel ex ...... o heart failure in adult mice.
@ast
In utero exposure to diesel ex ...... o heart failure in adult mice.
@en
type
label
In utero exposure to diesel ex ...... o heart failure in adult mice.
@ast
In utero exposure to diesel ex ...... o heart failure in adult mice.
@en
prefLabel
In utero exposure to diesel ex ...... o heart failure in adult mice.
@ast
In utero exposure to diesel ex ...... o heart failure in adult mice.
@en
P2093
P2860
P921
P1433
P1476
In utero exposure to diesel ex ...... o heart failure in adult mice.
@en
P2093
Chad S Weldy
H Denny Liggitt
Michael T Chin
Yonggang Liu
P2860
P304
P356
10.1371/JOURNAL.PONE.0088582
P407
P577
2014-02-12T00:00:00Z