The pathobiological mechanisms of emphysema models: what do they have in common?
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Bronchoscopic lung volume reduction procedures for chronic obstructive pulmonary diseaseLung volume reduction surgery for diffuse emphysemaAnimal models of chronic obstructive pulmonary diseaseEarly emphysema in the tight skin and pallid mice: roles of microfibril-associated glycoproteins, collagen, and mechanical forces.Fibroblast growth factor receptors control epithelial-mesenchymal interactions necessary for alveolar elastogenesis.Inflammatory changes in the airways of mice caused by cigarette smoke exposure are only partially reversed after smoking cessation.HIV and chronic obstructive pulmonary disease: is it worse and why?Vascular remodeling in the circulations of the lung.Airway epithelial cell PPARγ modulates cigarette smoke-induced chemokine expression and emphysema susceptibility in mice.Pathogenesis of chronic obstructive pulmonary diseaseAdvances in the pathology of COPD.Role of lung maintenance program in the heterogeneity of lung destruction in emphysema.Current concepts in mechanisms of emphysema.Lung volumes and respiratory mechanics in elastase-induced emphysema in mice.Cigarette smoke triggers code red: p21CIP1/WAF1/SDI1 switches on danger responses in the lung.Pathogenesis of emphysema: from the bench to the bedside.Breakdown of lung framework and an increase in pores of Kohn as initial events of emphysema and a cause of reduction in diffusing capacity.Lung disease associated with alpha1-antitrypsin deficiency.Animal models in chronic obstructive pulmonary disease-an overview.Computational modeling helps uncover mechanisms related to the progression of emphysema.Biological effects and mechanisms of action of mesenchymal stem cell therapy in chronic obstructive pulmonary disease.Bronchoscopic lung volume reduction procedures for chronic obstructive pulmonary disease.Pathobiology of cigarette smoke-induced chronic obstructive pulmonary disease.Characterization of human pre-elafin mutants: full antipeptidase activity is essential to preserve lung tissue integrity in experimental emphysema.Airway inflammation in cadmium-exposed rats is associated with pulmonary oxidative stress and emphysema.Effects of exercise training on papain-induced pulmonary emphysema in Wistar rats.Assessing Structure-Function Relations in Mice Using the Forced Oscillation Technique and Quantitative Histology.Cell-based tissue engineering for lung regeneration.Reactive Oxygen Species in Chronic Obstructive Pulmonary Disease.
P2860
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P2860
The pathobiological mechanisms of emphysema models: what do they have in common?
description
2003 nî lūn-bûn
@nan
2003 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
The pathobiological mechanisms of emphysema models: what do they have in common?
@ast
The pathobiological mechanisms of emphysema models: what do they have in common?
@en
type
label
The pathobiological mechanisms of emphysema models: what do they have in common?
@ast
The pathobiological mechanisms of emphysema models: what do they have in common?
@en
prefLabel
The pathobiological mechanisms of emphysema models: what do they have in common?
@ast
The pathobiological mechanisms of emphysema models: what do they have in common?
@en
P2093
P1476
The pathobiological mechanisms of emphysema models: what do they have in common?
@en
P2093
Enid Neptune
Rubin M Tuder
Sharon McGrath
P356
10.1016/S1094-5539(02)00099-8
P577
2003-01-01T00:00:00Z