Mice with cardiac-restricted angiotensin-converting enzyme (ACE) have atrial enlargement, cardiac arrhythmia, and sudden death. - Wikidata - wikimedia - TriplyDB

Mice with cardiac-restricted angiotensin-converting enzyme (ACE) have atrial enlargement, cardiac arrhythmia, and sudden death.

Mice with cardiac-restricted angiotensin-converting enzyme (ACE) have atrial enlargement, cardiac arrhythmia, and sudden death.

http://www.wikidata.org/entity/Q35103181

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Angiotensin II-induced sudden arrhythmic death and electrical remodeling MURC, a muscle-restricted coiled-coil protein that modulates the Rho/ROCK pathway, induces cardiac dysfunction and conduction disturbance Renal angiotensin-converting enzyme and blood pressure control Endothelin-converting enzymes and related metalloproteases in Alzheimer's disease Rediscovering ACE: novel insights into the many roles of the angiotensin-converting enzyme Cross talk between cardiac myocytes and fibroblasts: from multiscale investigative approaches to mechanisms and functional consequences Mechanisms of sudden cardiac death: oxidants and metabolism Key roles of endothelin-1 and p38 MAPK in the regulation of atrial stretch response Caveolin-1 modulates cardiac gap junction homeostasis and arrhythmogenecity by regulating cSrc tyrosine kinase.American Heart Association atrial fibrillation research summit: a conference report from the American Heart Association Elastase-2, a Tissue Alternative Pathway for Angiotensin II Generation, Plays a Role in Circulatory Sympathovagal Balance in Mice Demographic determinants and effect of pre-operative angiotensin converting enzyme inhibitors and angiotensin receptor blockers on the occurrence of atrial fibrillation after CABG surgery Redox regulation, NF-kappaB, and atrial fibrillation.Extracellular matrix remodeling in atrial fibrosis: mechanisms and implications in atrial fibrillation.Circulating fibrosis biomarkers and risk of atrial fibrillation: The Cardiovascular Health Study (CHS)Impact of aldosterone antagonists on the substrate for atrial fibrillation: aldosterone promotes oxidative stress and atrial structural/electrical remodeling Myeloperoxidase acts as a profibrotic mediator of atrial fibrillation Mitochondria and arrhythmias.Molecular determinants of cardiac fibroblast electrical function and therapeutic implications for atrial fibrillation.Atrial fibrosis and the mechanisms of atrial fibrillation The intracellular renin-angiotensin system in the heart.Atrial remodeling is directly related to end-diastolic left ventricular pressure in a mouse model of ventricular pressure overload.Genetic models provide unique insight into angiotensin and bradykinin peptides in the extravascular compartment of the heart in vivo.Inhibition of renin-angiotensin system (RAS) reduces ventricular tachycardia risk by altering connexin43.Cardiac-restricted angiotensin-converting enzyme overexpression causes conduction defects and connexin dysregulation.Aerobic exercise training-induced left ventricular hypertrophy involves regulatory MicroRNAs, decreased angiotensin-converting enzyme-angiotensin ii, and synergistic regulation of angiotensin-converting enzyme 2-angiotensin (1-7).Novel pharmacological targets for the rhythm control management of atrial fibrillation.Inhibition of c-Src tyrosine kinase prevents angiotensin II-mediated connexin-43 remodeling and sudden cardiac death.Is angiotensin II a direct mediator of left ventricular hypertrophy? Time for another look.New fACEs to the renin-angiotensin system.Role of the MAPKs/TGF-β1/TRAF6 signaling pathway in postoperative atrial fibrillation Role of angiotensin system and effects of its inhibition in atrial fibrillation: clinical and experimental evidence.A Novel Transgenic Mouse Model of Cardiac Hypertrophy and Atrial Fibrillation.Tissue specific expression of angiotensin converting enzyme: a new way to study an old friend Atrial fibrillation: insights from clinical trials and novel treatment options.Mitochondria oxidative stress, connexin43 remodeling, and sudden arrhythmic death Non-antiarrhythmic medications for atrial fibrillation: from bench to clinical practice.Update on tissue renin-angiotensin systems.New insights into the role of angiotensin-converting enzyme obtained from the analysis of genetically modified mice.Relaxin suppresses atrial fibrillation by reversing fibrosis and myocyte hypertrophy and increasing conduction velocity and sodium current in spontaneously hypertensive rat hearts.

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P2860

Mice with cardiac-restricted angiotensin-converting enzyme (ACE) have atrial enlargement, cardiac arrhythmia, and sudden death.

http://www.wikidata.org/entity/Q35103181

description

2004 nî lūn-bûn

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2004 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2004 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2004年の論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年论文

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Mice with cardiac-restricted a ...... arrhythmia, and sudden death.

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Mice with cardiac-restricted a ...... arrhythmia, and sudden death.

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Mice with cardiac-restricted a ...... arrhythmia, and sudden death.

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The American Journal of Pathology

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Mice with cardiac-restricted a ...... c arrhythmia, and sudden death

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Brian D Hoit

http://www.w3.org/2001/XMLSchema#string

Duncan J Campbell

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George Keshelava

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Hong D Xiao

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Hui Zhao

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Mario R Capecchi

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Sebastien Fuchs

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Vijaykumar S Kasi

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William Lewis

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P2860

Effects of an angiotensin-converting-enzyme inhibitor, ramipril, on cardiovascular events in high-risk patients

The cardiac renin-angiotensin system: conceptual, or a regulator of cardiac function?

Effects of enalapril on mortality in severe congestive heart failure. Results of the Cooperative North Scandinavian Enalapril Survival Study (CONSENSUS).

Overexpression of angiotensin II type I receptor in cardiomyocytes induces cardiac hypertrophy and remodeling.

Role of the local renin-angiotensin system in cardiac damage: a minireview focussing on transgenic animal models.

Angiotensin-converting enzyme and male fertility

Overexpression of angiotensin AT1 receptor transgene in the mouse myocardium produces a lethal phenotype associated with myocyte hyperplasia and heart block.

Transcriptional mapping and genomic analysis of the cardiac atria and ventricles.

Overview of randomized trials of angiotensin-converting enzyme inhibitors on mortality and morbidity in patients with heart failure. Collaborative Group on ACE Inhibitor Trials.

Peptidyl dipeptidase A: angiotensin I-converting enzyme.

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1019-1032

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Samuel C. Dudley

Kenneth Bernstein

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8381726

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