Asthmatic airway epithelium is intrinsically inflammatory and mitotically dyssynchronous. - Wikidata - wikimedia - TriplyDB

Asthmatic airway epithelium is intrinsically inflammatory and mitotically dyssynchronous.

Asthmatic airway epithelium is intrinsically inflammatory and mitotically dyssynchronous.

http://www.wikidata.org/entity/Q35103899

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Epithelial Anion Transport as Modulator of Chemokine Signaling The genetic and epigenetic landscapes of the epithelium in asthma VBP15, a glucocorticoid analogue, is effective at reducing allergic lung inflammation in mice Synthetic response of stimulated respiratory epithelium: modulation by prednisolone and iKK2 inhibition.Transcription factor p63 regulates key genes and wound repair in human airway epithelial basal cells Mitotic asynchrony induces transforming growth factor-β1 secretion from airway epithelium Asynchronous remodeling is a driver of failed regeneration in Duchenne muscular dystrophy.A microRNA network dysregulated in asthma controls IL-6 production in bronchial epithelial cells Dexamethasone inhibits repair of human airway epithelial cells mediated by glucocorticoid-induced leucine zipper (GILZ).Gene expression changes associated with the airway wall response to injury.Directional secretory response of double stranded RNA-induced thymic stromal lymphopoetin (TSLP) and CCL11/eotaxin-1 in human asthmatic airways.Glutathione S-transferase M1 modulates allergen-induced NF-κB activation in asthmatic airway epithelium Adipocyte-derived exosomal miRNAs: a novel mechanism for obesity-related disease.Cigarette smoke causes caspase-independent apoptosis of bronchial epithelial cells from asthmatic donors.Conserved steroid hormone homology converges on nuclear factor κB to modulate inflammation in asthma.Pharmacologically-induced mitotic synchrony in airway epithelial cells as a mechanism of action of anti-inflammatory drugs Epigenetic modifying enzyme expression in asthmatic airway epithelial cells and fibroblasts Exercise and asthma: an overview.Asthmatic airway neutrophilia after allergen challenge is associated with the glutathione S-transferase M1 genotype The human secretome atlas initiative: implications in health and disease conditions.Directional secretomes reflect polarity-specific functions in an in vitro model of human bronchial epithelium.Endothelin-1 directs airway remodeling and hyper-reactivity in a murine asthma model.Epithelial function and dysfunction in asthma.Response of airway epithelial cells to double-stranded RNA in an allergic environment.Using multiple online databases to help identify microRNAs regulating the airway epithelial cell response to a virus-like stimulus.Intrinsic phenotypic differences of asthmatic epithelium and its inflammatory responses to respiratory syncytial virus and air pollution.A Heterotopic Xenograft Model of Human Airways for Investigating Fibrosis in Asthma.Anti-inflammatory effects of embelin in A549 cells and human asthmatic airway epithelial tissues.Modeling the Influence of Seasonal Differences in the HPA Axis on Synchronization of the Circadian Clock and Cell Cycle.Chemokine expression in the early response to injury in human airway epithelial cells.Prevalidation of an Acute Inhalation Toxicity Test Using the EpiAirway In Vitro Human Airway Model.

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P2860

Asthmatic airway epithelium is intrinsically inflammatory and mitotically dyssynchronous.

http://www.wikidata.org/entity/Q35103899

description

2010 nî lūn-bûn

@nan

2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

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2010年論文

@zh-tw

2010年论文

@wuu

name

Asthmatic airway epithelium is intrinsically inflammatory and mitotically dyssynchronous.

@ast

Asthmatic airway epithelium is intrinsically inflammatory and mitotically dyssynchronous.

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type

label

Asthmatic airway epithelium is intrinsically inflammatory and mitotically dyssynchronous.

@ast

Asthmatic airway epithelium is intrinsically inflammatory and mitotically dyssynchronous.

@en

prefLabel

Asthmatic airway epithelium is intrinsically inflammatory and mitotically dyssynchronous.

@ast

Asthmatic airway epithelium is intrinsically inflammatory and mitotically dyssynchronous.

@en

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P356

RCMB.2010-0029OC

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American Journal of Respiratory Cell and Molecular Biology

P1476

Asthmatic airway epithelium is intrinsically inflammatory and mitotically dyssynchronous.

@en

P2093

Alan M Watson

http://www.w3.org/2001/XMLSchema#string

Angela S Benton

http://www.w3.org/2001/XMLSchema#string

Dinesh K Pillai

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Eric P Hoffman

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Mary C Rose

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Sabah F Iqbal

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P2860

The gene encoding interleukin-13: a susceptibility locus for asthma and related traits

Delineation of a gene network underlying the pulmonary response to oxidative stress in asthma

Expert Panel Report 3 (EPR-3): Guidelines for the Diagnosis and Management of Asthma-Summary Report 2007

Resetting of circadian time in peripheral tissues by glucocorticoid signaling

Epithelial cell proliferation contributes to airway remodeling in severe asthma.

IL-10 gene knockout attenuates allergen-induced airway hyperresponsiveness in C57BL/6 mice.

The bronchial epithelium as a key regulator of airway inflammation and remodelling in asthma.

Asthma: the importance of epithelial mesenchymal communication in pathogenesis. Inflammation and the airway epithelium in asthma.

Airway remodeling in asthma: new insights.

Circadian timing in the lung; a specific role for bronchiolar epithelial cells

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