The Drosophila melanogaster toll pathway participates in resistance to infection by the gram-negative human pathogen Pseudomonas aeruginosa. - Wikidata - wikimedia - TriplyDB

The Drosophila melanogaster toll pathway participates in resistance to infection by the gram-negative human pathogen Pseudomonas aeruginosa.

The Drosophila melanogaster toll pathway participates in resistance to infection by the gram-negative human pathogen Pseudomonas aeruginosa.

http://www.wikidata.org/entity/Q35106121

about

Secreted Bacterial Effectors and Host-Produced Eiger/TNF Drive Death in aSalmonella-Infected Fruit Fly The bacterial redox signaller pyocyanin as an antiplasmodial agent: comparisons with its thioanalog methylene blue Unique biofilm signature, drug susceptibility and decreased virulence in Drosophila through the Pseudomonas aeruginosa two-component system PprAB Quorum sensing differentially regulates Pseudomonas aeruginosa type VI secretion locus I and homologous loci II and III, which are required for pathogenesis Genome-wide identification of Pseudomonas aeruginosa virulence-related genes using a Caenorhabditis elegans infection model Identification of anti-virulence compounds that disrupt quorum-sensing regulated acute and persistent pathogenicity An ordered, nonredundant library of Pseudomonas aeruginosa strain PA14 transposon insertion mutants Dual-seq transcriptomics reveals the battle for iron during Pseudomonas aeruginosa acute murine pneumonia.Exploiting amoeboid and non-vertebrate animal model systems to study the virulence of human pathogenic fungi.Isotopolog perturbation techniques for metabolic networks: metabolic recycling of nutritional glucose in Drosophila melanogaster Genomic analysis reveals that Pseudomonas aeruginosa virulence is combinatorial.Involvement of skeletal muscle gene regulatory network in susceptibility to wound infection following trauma.A signaling protease required for melanization in Drosophila affects resistance and tolerance of infections.Effective but costly, evolved mechanisms of defense against a virulent opportunistic pathogen in Drosophila melanogaster The role of anorexia in resistance and tolerance to infections in Drosophila Homeostatic interplay between bacterial cell-cell signaling and iron in virulence.A conserved suppressor mutation in a tryptophan auxotroph results in dysregulation of Pseudomonas quinolone signal synthesis.Pseudomonas aeruginosa OxyR is required for full virulence in rodent and insect models of infection and for resistance to human neutrophils A Small Wolbachia Protein Directly Represses Phage Lytic Cycle Genes in "Candidatus Liberibacter asiaticus" within Psyllids Profiling early infection responses: Pseudomonas aeruginosa eludes host defenses by suppressing antimicrobial peptide gene expression.Sugar fatty acid esters inhibit biofilm formation by food-borne pathogenic bacteria Drosophila host defense after oral infection by an entomopathogenic Pseudomonas species.Blocking of Plasmodium transmission by cooperative action of Cecropin A and Defensin A in transgenic Aedes aegypti mosquitoes.A quorum sensing regulated small volatile molecule reduces acute virulence and promotes chronic infection phenotypes.Bacterial communities of diverse Drosophila species: ecological context of a host-microbe model system.Drosophila melanogaster S2 cells: a model system to study Chlamydia interaction with host cells.Peroxiredoxin 5 modulates immune response in Drosophila The Pseudomonas aeruginosa reference strain PA14 displays increased virulence due to a mutation in ladS.Drosophila as a model system to unravel the layers of innate immunity to infection.Assessment of virulence diversity of methicillin-resistant Staphylococcus aureus strains with a Drosophila melanogaster infection model.Reactive oxygen species mediate inflammatory cytokine release and EGFR-dependent mucin secretion in airway epithelial cells exposed to Pseudomonas pyocyanin.Circadian regulation in the ability of Drosophila to combat pathogenic infections.Bacterial infection of fly ovaries reduces egg production and induces local hemocyte activation.Pseudomonas aeruginosa RhlR is required to neutralize the cellular immune response in a Drosophila melanogaster oral infection model.The Drosophila melanogaster host model.Down-regulation of glutatione S-transferase α 4 (hGSTA4) in the muscle of thermally injured patients is indicative of susceptibility to bacterial infection.Major Transcriptome Changes Accompany the Growth of Pseudomonas aeruginosa in Blood from Patients with Severe Thermal Injuries.Midgut immune responses induced by bacterial infection in the silkworm, Bombyx mori.Are Drosophila a useful model for understanding the toxicity of inhaled oxidative pollutants: a review.Disruption of the Aspergillus fumigatus gene encoding nucleolar protein CgrA impairs thermotolerant growth and reduces virulence.

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The Drosophila melanogaster toll pathway participates in resistance to infection by the gram-negative human pathogen Pseudomonas aeruginosa.

http://www.wikidata.org/entity/Q35106121

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2003 nî lūn-bûn

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2003 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2003 թվականի հուլիսին հրատարակված գիտական հոդված

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2003年の論文

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2003年論文

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2003年論文

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The Drosophila melanogaster to ...... thogen Pseudomonas aeruginosa.

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The Drosophila melanogaster to ...... thogen Pseudomonas aeruginosa.

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IAI.71.7.4059-4066.2003

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Infection and Immunity

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The Drosophila melanogaster to ...... thogen Pseudomonas aeruginosa.

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P2093

Boyan C Goumnerov

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Cynthia L Walendziewicz

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Gee W Lau

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Jennifer Hewitson

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Laurence G Rahme

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Lizabeth A Perkins

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Ronald G Tompkins

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Shalina Mahajan-Miklos

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P2860

Drosophila as a model host for Pseudomonas aeruginosa infection

Plants and animals share functionally common bacterial virulence factors

Use of model plant hosts to identify Pseudomonas aeruginosa virulence factors

Phylogenetic perspectives in innate immunity

The dorsoventral regulatory gene cassette spätzle/Toll/cactus controls the potent antifungal response in Drosophila adults

Common virulence factors for bacterial pathogenicity in plants and animals

Molecular mechanisms of bacterial virulence elucidated using a Pseudomonas aeruginosa-Caenorhabditis elegans pathogenesis model

Mutations in the hemolytic-phospholipase C operon result in decreased virulence of Pseudomonas aeruginosa PAO1 grown under phosphate-limiting conditions

DsbA: a protein-folding catalyst contributing to bacterial virulence.

Toll signaling pathways in the innate immune response.

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4059-4066

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10.1128/IAI.71.7.4059-4066.2003

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Drosophila melanogaster

Pseudomonas aeruginosa

Gram-negative bacteria

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162001

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