Improvement of chloride transport defect by gonadotropin-releasing hormone (GnRH) in cystic fibrosis epithelial cells.
about
Gonadotropin-releasing hormone stimulates biliary proliferation by paracrine/autocrine mechanismsG551D-CFTR needs more bound actin than wild-type CFTR to maintain its presence in plasma membranes.Buserelin alleviates chloride transport defect in human cystic fibrosis nasal epithelial cells.Function and regulation of TRPM7, as well as intracellular magnesium content, are altered in cells expressing ΔF508-CFTR and G551D-CFTR.
P2860
Improvement of chloride transport defect by gonadotropin-releasing hormone (GnRH) in cystic fibrosis epithelial cells.
description
2014 nî lūn-bûn
@nan
2014 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Improvement of chloride transp ...... tic fibrosis epithelial cells.
@ast
Improvement of chloride transp ...... tic fibrosis epithelial cells.
@en
type
label
Improvement of chloride transp ...... tic fibrosis epithelial cells.
@ast
Improvement of chloride transp ...... tic fibrosis epithelial cells.
@en
prefLabel
Improvement of chloride transp ...... tic fibrosis epithelial cells.
@ast
Improvement of chloride transp ...... tic fibrosis epithelial cells.
@en
P2093
P2860
P1433
P1476
Improvement of chloride transp ...... tic fibrosis epithelial cells.
@en
P2093
Caroline Norez
Claude Férec
Marie-Laure Calvez
Mathieu Kerbiriou
Nathalie Benz
Pascal Trouvé
Sophie Le Hir
P2860
P304
P356
10.1371/JOURNAL.PONE.0088964
P407
P50
P577
2014-02-19T00:00:00Z