Macrophage contact dependent and independent TLR4 mechanisms induce β-cell dysfunction and apoptosis in a mouse model of type 2 diabetes.

Macrophage contact dependent and independent TLR4 mechanisms induce β-cell dysfunction and apoptosis in a mouse model of type 2 diabetes.

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http://www.wikidata.org/entity/Q35110994
Minireview: Emerging Concepts in Islet Macrophage Biology in Type 2 DiabetesThe Role of the Microbial Metabolites Including Tryptophan Catabolites and Short Chain Fatty Acids in the Pathophysiology of Immune-Inflammatory and Neuroimmune DiseaseEvidence for the gut microbiota short-chain fatty acids as key pathophysiological molecules improving diabetes.Reduction of specific circulating lymphocyte populations with metabolic risk factors in patients at risk to develop type 2 diabetesNeutralizing Anti-IL20 Antibody Treatment Significantly Modulates Low Grade Inflammation without Affecting HbA1c in Type 2 Diabetic db/db MiceImpaired coronary microcirculation in type 2 diabetic patients is associated with elevated circulating regulatory T cells and reduced number of IL-21R⁺ T cells.Mitochondrial toxicity of triclosan on mammalian cells.Low dose doxycycline decreases systemic inflammation and improves glycemic control, lipid profiles, and islet morphology and function in db/db mice.Ageing potentiates diet-induced glucose intolerance, β-cell failure and tissue inflammation through TLR4.
P2860

Macrophage contact dependent and independent TLR4 mechanisms induce β-cell dysfunction and apoptosis in a mouse model of type 2 diabetes.

Item
http://www.wikidata.org/entity/Q35110994
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
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name
Macrophage contact dependent a ...... ouse model of type 2 diabetes.
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Macrophage contact dependent a ...... ouse model of type 2 diabetes.
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Macrophage contact dependent a ...... ouse model of type 2 diabetes.
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Macrophage contact dependent a ...... ouse model of type 2 diabetes.
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Macrophage contact dependent a ...... ouse model of type 2 diabetes.
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Macrophage contact dependent a ...... ouse model of type 2 diabetes.
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10.1371/JOURNAL.PONE.0090685
P407
P433
P478
P577
P5875
P698
P921
P932