RAS transformation requires CUX1-dependent repair of oxidative DNA damage. - Wikidata - wikimedia - TriplyDB

RAS transformation requires CUX1-dependent repair of oxidative DNA damage.

RAS transformation requires CUX1-dependent repair of oxidative DNA damage.

http://www.wikidata.org/entity/Q35117739

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Molecular pathways: targeting the dependence of mutant RAS cancers on the DNA damage response How sleep and wakefulness influence circadian rhythmicity: effects of insufficient and mistimed sleep on the animal and human transcriptome Exploiting human and mouse transcriptomic data: Identification of circadian genes and pathways influencing health.CUX1, a haploinsufficient tumour suppressor gene overexpressed in advanced cancers.Autocrine Activation of the Wnt/β-Catenin Pathway by CUX1 and GLIS1 in Breast Cancers.Small Molecule Inhibitors of 8-Oxoguanine DNA Glycosylase-1 (OGG1).CUX2 protein functions as an accessory factor in the repair of oxidative DNA damage.Special AT-rich Sequence-binding Protein 1 (SATB1) Functions as an Accessory Factor in Base Excision Repair.Hypomorphism of Fto and Rpgrip1l causes obesity in mice.The transcription factor Cux1 in cerebellar granule cell development and medulloblastoma pathogenesis.Zinc finger oxidation of Fpg/Nei DNA glycosylases by 2-thioxanthine: biochemical and X-ray structural characterization.The function of CUX1 in oxidative DNA damage repair is needed to prevent premature senescence of mouse embryo fibroblasts.The DNA repair function of CUX1 contributes to radioresistance.8-Oxoguanine DNA glycosylase 1: Beyond repair of the oxidatively modified base lesions.CUX1 Stimulates APE1 Enzymatic Activity and Increases the Resistance of Glioblastoma Cells to the Mono-Alkylating Agent, Temozolomide.

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RAS transformation requires CUX1-dependent repair of oxidative DNA damage.

http://www.wikidata.org/entity/Q35117739

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2014 nî lūn-bûn

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2014 թուականի Մարտին հրատարակուած գիտական յօդուած

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2014 թվականի մարտին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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RAS transformation requires CUX1-dependent repair of oxidative DNA damage.

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RAS transformation requires CUX1-dependent repair of oxidative DNA damage.

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RAS transformation requires CUX1-dependent repair of oxidative DNA damage.

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RAS transformation requires CUX1-dependent repair of oxidative DNA damage.

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JOURNAL.PBIO.1001807

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RAS transformation requires CUX1-dependent repair of oxidative DNA damage

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Alain Nepveu

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Anthony N Karnezis

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Chantal Cadieux

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Charles Vadnais

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Guillaume Vandal

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Lam Leduy

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Lu Yao

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Marilène Paquet

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Sayeh Davoudi

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Zubaidah M Ramdzan

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P2860

Human CCAAT displacement protein is homologous to the Drosophila homeoprotein, cut

Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a

S phase-specific proteolytic cleavage is required to activate stable DNA binding by the CDP/Cut homeodomain protein

Complete nucleotide and deduced amino acid sequences of human and murine preprocathepsin L. An abundant transcript induced by transformation of fibroblasts

Prevalence and prognostic impact of allelic imbalances associated with leukemic transformation of Philadelphia chromosome-negative myeloproliferative neoplasms

Comprehensive molecular characterization of human colon and rectal cancer

A census of human cancer genes

A genome-wide RNAi screen identifies multiple synthetic lethal interactions with the Ras oncogene

Dimerization and opposite base-dependent catalytic impairment of polymorphic S326C OGG1 glycosylase

Oncogene-induced senescence relayed by an interleukin-dependent inflammatory network.

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10.1371/JOURNAL.PBIO.1001807

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