Gain of virulence caused by loss of a gene in murine cytomegalovirus - Wikidata - wikimedia - TriplyDB

Gain of virulence caused by loss of a gene in murine cytomegalovirus

Gain of virulence caused by loss of a gene in murine cytomegalovirus

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Ly49 receptors: innate and adaptive immune paradigms Cytomegalovirus antivirals and development of improved animal models Natural killer cells promote early CD8 T cell responses against cytomegalovirus Natural Killer Cell Sensing of Infected Cells Compensates for MyD88 Deficiency but Not IFN-I Activity in Resistance to Mouse Cytomegalovirus Nodular inflammatory foci are sites of T cell priming and control of murine cytomegalovirus infection in the neonatal lung NK cell receptor/H2-Dk-dependent host resistance to viral infection is quantitatively modulated by H2q inhibitory signals The SIRP family of receptors and immune regulation Know Thyself: NK-Cell Inhibitory Receptors Prompt Self-Tolerance, Education, and Viral Control.Random screening for dominant-negative mutants of the cytomegalovirus nuclear egress protein M50 Complete sequence determination of a novel reptile iridovirus isolated from soft-shelled turtle and evolutionary analysis of Iridoviridae HCMV spread and cell tropism are determined by distinct virus populations.A dominant role for the immunoproteasome in CD8+ T cell responses to murine cytomegalovirus Selective down-regulation of the NKG2D ligand H60 by mouse cytomegalovirus m155 glycoprotein M94 is essential for the secondary envelopment of murine cytomegalovirus.Deciphering the role of DC subsets in MCMV infection to better understand immune protection against viral infections Cytomegalovirus contributes to glioblastoma in the context of tumor suppressor mutations.Expression of m157, a murine cytomegalovirus-encoded putative major histocompatibility class I (MHC-I)-like protein, is independent of viral regulation of host MHC-I.Immune senescence: relative contributions of age and cytomegalovirus infection Antigen-specific expansion and differentiation of natural killer cells by alloantigen stimulation Natural killer cell dependent within-host competition arises during multiple MCMV infection: consequences for viral transmission and evolution.Crystal structure of the murine cytomegalovirus MHC-I homolog m144.Cytomegalovirus generates long-lived antigen-specific NK cells with diminished bystander activation to heterologous infection VIPhyb, an antagonist of vasoactive intestinal peptide receptor, enhances cellular antiviral immunity in murine cytomegalovirus infected mice.The Glycophosphatidylinositol Anchor of the MCMV Evasin, m157, Facilitates Optimal Cell Surface Expression and Ly49 Receptor Recognition.Distinct MHC class I-dependent NK cell-activating receptors control cytomegalovirus infection in different mouse strains.How the virus outsmarts the host: function and structure of cytomegalovirus MHC-I-like molecules in the evasion of natural killer cell surveillance Reversible silencing of cytomegalovirus genomes by type I interferon governs virus latency.Mast cells expedite control of pulmonary murine cytomegalovirus infection by enhancing the recruitment of protective CD8 T cells to the lungs.Reduced MCMV Δm157 viral clearance in the absence of TSAd.Virus progeny of murine cytomegalovirus bacterial artificial chromosome pSM3fr show reduced growth in salivary Glands due to a fixed mutation of MCK-2 Evaluating Human T-Cell Therapy of Cytomegalovirus Organ Disease in HLA-Transgenic Mice.Ly49h-deficient C57BL/6 mice: a new mouse cytomegalovirus-susceptible model remains resistant to unrelated pathogens controlled by the NK gene complex.The NK cell response to mouse cytomegalovirus infection affects the level and kinetics of the early CD8(+) T-cell response.Mice with asthma are more resistant to influenza virus infection and NK cells activated by the induction of asthma have potentially protective effects.A flow cytometry-based method for detecting antibody responses to murine cytomegalovirus infection.Mast cells as rapid innate sensors of cytomegalovirus by TLR3/TRIF signaling-dependent and -independent mechanisms Targeted deletion of regions rich in immune-evasive genes from the cytomegalovirus genome as a novel vaccine strategy.Antiviral T cell response triggers cytomegalovirus hepatitis in mice.NK cell activation through the NKG2D ligand MULT-1 is selectively prevented by the glycoprotein encoded by mouse cytomegalovirus gene m145.Cardif (MAVS) Regulates the Maturation of NK Cells

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P2860

Gain of virulence caused by loss of a gene in murine cytomegalovirus

http://www.wikidata.org/entity/Q35123311

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2004 nî lūn-bûn

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2004 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2004 թվականի հուլիսին հրատարակված գիտական հոդված

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2004年の論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年论文

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name

Gain of virulence caused by loss of a gene in murine cytomegalovirus

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Gain of virulence caused by loss of a gene in murine cytomegalovirus

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type

label

Gain of virulence caused by loss of a gene in murine cytomegalovirus

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Gain of virulence caused by loss of a gene in murine cytomegalovirus

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prefLabel

Gain of virulence caused by loss of a gene in murine cytomegalovirus

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Gain of virulence caused by loss of a gene in murine cytomegalovirus

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JVI.78.14.7536-7544.2004

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Journal of Virology

P1476

Gain of virulence caused by loss of a gene in murine cytomegalovirus

@en

P2093

Ivan Bubić

http://www.w3.org/2001/XMLSchema#string

Markus Wagner

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Sungjin Kim

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Tanja Saulig

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Ulrich H Koszinowski

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Wayne M Yokoyama

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P2860

Activation of NK cells and T cells by NKG2D, a receptor for stress-inducible MICA

The effect of the Cmv-1 resistance gene, which is linked to the natural killer cell gene complex, is mediated by natural killer cells

Gene structure, expression pattern, and biological activity of mouse killer cell activating receptor-associated protein (KARAP)/DAP-12

Class I MHC-binding characteristics of the 129/J Ly49 repertoire

Gene disruption in Escherichia coli: TcR and KmR cassettes with the option of Flp-catalyzed excision of the antibiotic-resistance determinant

Natural killer cells in antiviral defense: function and regulation by innate cytokines.

Natural killer cells: stress out, turn on, tune in.

Systematic excision of vector sequences from the BAC-cloned herpesvirus genome during virus reconstitution.

Recognition of a virus-encoded ligand by a natural killer cell activation receptor.

The role of the NKG2D immunoreceptor in immune cell activation and natural killing.

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7536-7544

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scholarly article

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10.1128/JVI.78.14.7536-7544.2004

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14

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78

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Astrid Krmpotic

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2004-07-01T00:00:00Z

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8488763

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15220428

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Cytomegalovirus

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434107

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