Increased complement C1q level marks active disease in human tuberculosis. - Wikidata - wikimedia - TriplyDB

Increased complement C1q level marks active disease in human tuberculosis.

Increased complement C1q level marks active disease in human tuberculosis.

http://www.wikidata.org/entity/Q35125344

about

Diagnostic 'omics' for active tuberculosis Antibodies and tuberculosis xCT increases tuberculosis susceptibility by regulating antimicrobial function and inflammation Tricks to translating TB transcriptomics.Host sirtuin 1 regulates mycobacterial immunopathogenesis and represents a therapeutic target against tuberculosis.Genome-wide expression for diagnosis of pulmonary tuberculosis: a multicohort analysis.Temporal Expression of Peripheral Blood Leukocyte Biomarkers in a Macaca fascicularis Infection Model of Tuberculosis; Comparison with Human Datasets and Analysis with Parametric/Non-parametric Tools for Improved Diagnostic Biomarker Identification The Transcriptional Signature of Active Tuberculosis Reflects Symptom Status in Extra-Pulmonary and Pulmonary Tuberculosis Sputum endothelin-1 level is associated with active pulmonary tuberculosis and effectiveness of anti-tuberculosis chemotherapy.Proteomic profiling of eccrine sweat reveals its potential as a diagnostic biofluid for active tuberculosis.Q-GDEMAR: a general method for the identification of differentially expressed genes in microarrays with unbalanced groups.Extracellular ADP facilitates monocyte recruitment in bacterial infection via ERK signaling.The lectin Siglec-G inhibits dendritic cell cross-presentation by impairing MHC class I-peptide complex formation.Discriminating Active Tuberculosis from Latent Tuberculosis Infection by flow cytometric measurement of CD161-expressing T cells.Complement pathway gene activation and rising circulating immune complexes characterize early disease in HIV-associated tuberculosis.Complement C1q expression in Erythema nodosum leprosum.Amino acid starvation sensing dampens IL-1β production by activating riboclustering and autophagy.Type I interferons in tuberculosis: Foe and occasionally friend.Data Mining Mycobacterium tuberculosis Pathogenic Gene Transcription Factors and Their Regulatory Network Nodes.Genetic Resistance to Infection and Disease Complement Component C1q as Serum Biomarker to Detect Active Tuberculosis

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P2860

Increased complement C1q level marks active disease in human tuberculosis.

http://www.wikidata.org/entity/Q35125344

description

2014 nî lūn-bûn

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2014 թուականի Մարտին հրատարակուած գիտական յօդուած

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2014 թվականի մարտին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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Increased complement C1q level marks active disease in human tuberculosis.

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Increased complement C1q level marks active disease in human tuberculosis.

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Increased complement C1q level marks active disease in human tuberculosis.

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Increased complement C1q level marks active disease in human tuberculosis.

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Increased complement C1q level marks active disease in human tuberculosis.

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Increased complement C1q level marks active disease in human tuberculosis.

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Increased complement C1q level marks active disease in human tuberculosis

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Boping Zhou

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Guoliang Zhang

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Haiying Liu

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Jian Huang

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Mingxia Zhang

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Qianting Yang

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Qunyi Deng

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Xinchun Chen

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Yueqiang Tang

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Zhiliang Gao

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P2860

Interferon-γ release assays for active pulmonary tuberculosis diagnosis in adults in low- and middle-income countries: systematic review and meta-analysis

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

An interferon-inducible neutrophil-driven blood transcriptional signature in human tuberculosis

Restoration of C1q levels by bone marrow transplantation attenuates autoimmune disease associated with C1q deficiency in mice.

Neutrophils are the predominant infected phagocytic cells in the airways of patients with active pulmonary TB.

High gene expression of inflammatory markers and IL-17A correlates with severity of injection site reactions of Atlantic salmon vaccinated with oil-adjuvanted vaccines.

Intranasal Poly-IC treatment exacerbates tuberculosis in mice through the pulmonary recruitment of a pathogen-permissive monocyte/macrophage population.

Enhanced contact tracing and spatial tracking of Mycobacterium tuberculosis infection by enumeration of antigen-specific T cells.

Efficacy of BCG vaccine in the prevention of tuberculosis. Meta-analysis of the published literature.

Different patterns of cytokines and chemokines combined with IFN-γ production reflect Mycobacterium tuberculosis infection and disease.

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