Nuclear translocation of AMPK-alpha1 potentiates striatal neurodegeneration in Huntington's disease. - Wikidata - wikimedia - TriplyDB

Nuclear translocation of AMPK-alpha1 potentiates striatal neurodegeneration in Huntington's disease.

Nuclear translocation of AMPK-alpha1 potentiates striatal neurodegeneration in Huntington's disease.

http://www.wikidata.org/entity/Q35130191

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Defining the role of the Bcl-2 family proteins in Huntington's disease High-content chemical and RNAi screens for suppressors of neurotoxicity in a Huntington's disease model A novel manganese-dependent ATM-p53 signaling pathway is selectively impaired in patient-based neuroprogenitor and murine striatal models of Huntington's disease A dual role for AMP-activated protein kinase (AMPK) during neonatal hypoxic-ischaemic brain injury in mice Metabolism in HD: still a relevant mechanism?c-Myc and AMPK Control Cellular Energy Levels by Cooperatively Regulating Mitochondrial Structure and Function A neuroprotective phase precedes striatal degeneration upon nucleolar stress.CCL5/RANTES contributes to hypothalamic insulin signaling for systemic insulin responsiveness through CCR5.Dissecting the role of AMP-activated protein kinase in human diseases.AMPK signaling in the nucleus accumbens core mediates cue-induced reinstatement of cocaine seeking Metformin intake associates with better cognitive function in patients with Huntington's disease.Adenosine monophosphate-activated protein kinase activation enhances embryonic neural stem cell apoptosis in a mouse model of amyotrophic lateral sclerosis.Forkhead transcription factor FOXO3a levels are increased in Huntington disease because of overactivated positive autofeedback loop.AMPK at the nexus of energetics and aging.Suppression of the HSF1-mediated proteotoxic stress response by the metabolic stress sensor AMPK Detecting apoptosis of leukocytes in mouse lymph nodes.Small molecule adenosine 5'-monophosphate activated protein kinase (AMPK) modulators and human diseases Bioenergy sensing in the brain: the role of AMP-activated protein kinase in neuronal metabolism, development and neurological diseases Huntington's disease: Neural dysfunction linked to inositol polyphosphate multikinase.AMP-Activated Protein Kinase (AMPK) and Energy-Sensing in the Brain SUMOylation of AMPKα1 by PIAS4 specifically regulates mTORC1 signalling.N-cadherin coordinates AMP kinase-mediated lung vascular repair.AMPK activation protects from neuronal dysfunction and vulnerability across nematode, cellular and mouse models of Huntington's disease.AMPK Signaling in the Dorsal Hippocampus Negatively Regulates Contextual Fear Memory Formation.Alternative mitochondrial electron transfer for the treatment of neurodegenerative diseases and cancers: Methylene blue connects the dots.AMP-activated protein kinase modulates tau phosphorylation and tau pathology in vivo.The CAMKK2-AMPK kinase pathway mediates the synaptotoxic effects of Aβ oligomers through Tau phosphorylation Latrepirdine is a potent activator of AMP-activated protein kinase and reduces neuronal excitability.(ADP-ribose) polymerase 1 and AMP-activated protein kinase mediate progressive dopaminergic neuronal degeneration in a mouse model of Parkinson's disease.Role and Therapeutic Potential of the Pro-Longevity Factor FOXO and Its Regulators in Neurodegenerative Disease.Roles of AMP-activated protein kinase in Alzheimer's disease.Energy dysfunction in Huntington's disease: insights from PGC-1α, AMPK, and CKB.AMP-activated protein kinase: new regulation, new roles?Function of the master energy regulator adenosine monophosphate-activated protein kinase in stroke.Involvement of 'stress-response' kinase pathways in Alzheimer's disease progression.Insulin as a Bridge between Type 2 Diabetes and Alzheimer Disease - How Anti-Diabetics Could be a Solution for Dementia.Activators of AMPK: not just for type II diabetes.The metabolic response to excitotoxicity - lessons from single-cell imaging.From pathways to targets: understanding the mechanisms behind polyglutamine disease.Activation of mTOR: a culprit of Alzheimer's disease?

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P2860

Nuclear translocation of AMPK-alpha1 potentiates striatal neurodegeneration in Huntington's disease.

http://www.wikidata.org/entity/Q35130191

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2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2011 թվականի հուլիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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Nuclear translocation of AMPK- ...... ation in Huntington's disease.

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Nuclear translocation of AMPK- ...... ation in Huntington's disease.

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Nuclear translocation of AMPK- ...... ation in Huntington's disease.

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Nuclear translocation of AMPK- ...... ation in Huntington's disease.

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Nuclear translocation of AMPK- ...... ation in Huntington's disease.

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Chen Chang

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Cheng-Pu Sun

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Ching-Pang Chang

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Hui-Mei Chen

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Jheng-Jie Kang

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Jiun-Tsai Lin

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Mi-Hua Tao

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Tz-Chuen Ju

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Wei-Cheng Chang

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P2860

Phosphorylation of CREB at Ser-133 induces complex formation with CREB-binding protein via a direct mechanism

Mammalian AMP-activated protein kinase subfamily

Production of high-titer recombinant adeno-associated virus vectors in the absence of helper adenovirus

The regulation of AMP-activated protein kinase by phosphorylation

Huntingtin and the molecular pathogenesis of Huntington's disease. Fourth in molecular medicine review series

Longitudinal evaluation of an N-ethyl-N-nitrosourea-created murine model with normal pressure hydrocephalus

A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes.

Pharmacological inhibition of AMP-activated protein kinase provides neuroprotection in stroke

CCR7-dependent stimulation of survival in dendritic cells involves inhibition of GSK3beta

Excitotoxic neuronal death and the pathogenesis of Huntington's disease

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209-227

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10.1083/JCB.201105010

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2

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51501395

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21768291

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corpus striatum

Huntington disease

Protein kinase AMP-activated catalytic subunit alpha 2

Protein kinase AMP-activated catalytic subunit alpha 1

Protein kinase, AMP-activated, alpha 1 catalytic subunit

energy homeostasis

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3144412

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