Early potassium channel blockade improves sepsis-induced organ damage and cardiovascular dysfunction. - Wikidata - wikimedia - TriplyDB

Early potassium channel blockade improves sepsis-induced organ damage and cardiovascular dysfunction.

Early potassium channel blockade improves sepsis-induced organ damage and cardiovascular dysfunction.

http://www.wikidata.org/entity/Q35130561

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Fluid resuscitation therapy in endotoxemic hamsters improves survival and attenuates capillary perfusion deficits and inflammatory responses by a mechanism related to nitric oxide Inhibition of IκB Kinase Attenuates the Organ Injury and Dysfunction Associated with Hemorrhagic Shock Influence of arterial hypertension on colonic healing in rats.Smooth Muscle Ion Channels and Regulation of Vascular Tone in Resistance Arteries and Arterioles.A Protective Role of Glibenclamide in Inflammation-Associated Injury.Deletion of Nlrp3 Augments Survival during Polymicrobial Sepsis by Decreasing Autophagy and Enhancing Phagocytosis.Pneumonia-induced sepsis in mice: temporal study of inflammatory and cardiovascular parameters.Glucose homeostasis in two degrees of sepsis lethality induced by caecum ligation and puncture in mice.

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P2860

Early potassium channel blockade improves sepsis-induced organ damage and cardiovascular dysfunction.

http://www.wikidata.org/entity/Q35130561

description

2011 nî lūn-bûn

@nan

2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի հուլիսին հրատարակված գիտական հոդված

@hy

2011年の論文

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2011年論文

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2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

@wuu

name

Early potassium channel blocka ...... nd cardiovascular dysfunction.

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Early potassium channel blocka ...... nd cardiovascular dysfunction.

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type

label

Early potassium channel blocka ...... nd cardiovascular dysfunction.

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Early potassium channel blocka ...... nd cardiovascular dysfunction.

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prefLabel

Early potassium channel blocka ...... nd cardiovascular dysfunction.

@ast

Early potassium channel blocka ...... nd cardiovascular dysfunction.

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J.1476-5381.2011.01324.X

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British Journal of Pharmacology

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Early potassium channel blocka ...... and cardiovascular dysfunction

@en

P2093

B T Heckert

http://www.w3.org/2001/XMLSchema#string

J Assreuy

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P2860

Nitric oxide synthases in mammals

The ATP-sensitive K+ channel mediates hypotension in endotoxemia and hypoxic lactic acidosis in dog

Involvement of ATP-sensitive potassium channels in a model of a delayed vascular hyporeactivity induced by lipopolysaccharide in rats

Guanosine 5'-monophosphate modulates gating of high-conductance Ca2+-activated K+ channels in vascular smooth muscle cells.

Nitric oxide and cGMP cause vasorelaxation by activation of a charybdotoxin-sensitive K channel by cGMP-dependent protein kinase.

The pore-forming subunit of the K(ATP) channel is an important molecular target for LPS-induced vascular hyporeactivity in vitro.

Insulin therapy for the critically ill patient.

Multiple-center, randomized, placebo-controlled, double-blind study of the nitric oxide synthase inhibitor 546C88: effect on survival in patients with septic shock.

Animal models of sepsis: setting the stage.

Nitric oxide and cardiovascular dysfunction in sepsis.

P304

1289-1301

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scholarly article

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10.1111/J.1476-5381.2011.01324.X

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6

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163

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Daniel Fernandes

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2011-07-01T00:00:00Z

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50408062

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21410460

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3144541

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