Wnt16 attenuates TGFβ-induced chondrogenic transformation in vascular smooth muscle. - Wikidata - wikimedia - TriplyDB

Wnt16 attenuates TGFβ-induced chondrogenic transformation in vascular smooth muscle.

Wnt16 attenuates TGFβ-induced chondrogenic transformation in vascular smooth muscle.

http://www.wikidata.org/entity/Q35130798

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Vascular smooth muscle LRP6 limits arteriosclerotic calcification in diabetic LDLR-/- mice by restraining noncanonical Wnt signals Bidirectional Translation in Cardiovascular Calcification.Vascular Smooth Muscle Cells.Experimental preeclampsia in rats affects vascular gene expression patterns.Notch Signaling in Development, Tissue Homeostasis, and Disease.Genome-wide association study of coronary artery calcified atherosclerotic plaque in African Americans with type 2 diabetes.Implication of molecular vascular smooth muscle cell heterogeneity among arterial beds in arterial calcification.Cell Phenotype Transitions in Cardiovascular Calcification.

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Wnt16 attenuates TGFβ-induced chondrogenic transformation in vascular smooth muscle.

http://www.wikidata.org/entity/Q35130798

description

2015 nî lūn-bûn

@nan

2015 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի հունվարին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

@zh-tw

2015年论文

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name

Wnt16 attenuates TGFβ-induced chondrogenic transformation in vascular smooth muscle.

@ast

Wnt16 attenuates TGFβ-induced chondrogenic transformation in vascular smooth muscle.

@en

type

label

Wnt16 attenuates TGFβ-induced chondrogenic transformation in vascular smooth muscle.

@ast

Wnt16 attenuates TGFβ-induced chondrogenic transformation in vascular smooth muscle.

@en

prefLabel

Wnt16 attenuates TGFβ-induced chondrogenic transformation in vascular smooth muscle.

@ast

Wnt16 attenuates TGFβ-induced chondrogenic transformation in vascular smooth muscle.

@en

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ATVBAHA.114.304393

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Arteriosclerosis, Thrombosis, and Vascular Biology

P1476

Wnt16 attenuates TGFβ-induced chondrogenic transformation in vascular smooth muscle.

@en

P2093

Chintan Gandhi

http://www.w3.org/2001/XMLSchema#string

Dmitry Nurminsky

http://www.w3.org/2001/XMLSchema#string

Florence Lima

http://www.w3.org/2001/XMLSchema#string

Kelly E Beazley

http://www.w3.org/2001/XMLSchema#string

Maria V Nurminskaya

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P2860

Mutations in the gene encoding the human matrix Gla protein cause Keutel syndrome

SB-431542 is a potent and specific inhibitor of transforming growth factor-beta superfamily type I activin receptor-like kinase (ALK) receptors ALK4, ALK5, and ALK7

WNT16B is a new marker of cellular senescence that regulates p53 activity and the phosphoinositide 3-kinase/AKT pathway

Role for WNT16B in human epidermal keratinocyte proliferation and differentiation

A role for the endothelium in vascular calcification

Spontaneous calcification of arteries and cartilage in mice lacking matrix GLA protein

Notch1 represses osteogenic pathways in aortic valve cells

Wnt/beta-catenin signaling is sufficient and necessary for synovial joint formation

TGFbeta2 knockout mice have multiple developmental defects that are non-overlapping with other TGFbeta knockout phenotypes

Runx2/Cbfa1, but not loss of myocardin, is required for smooth muscle cell lineage reprogramming toward osteochondrogenesis.

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573-579

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scholarly article

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10.1161/ATVBAHA.114.304393

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3

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2015-01-22T00:00:00Z

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25614285

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4344425

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