AKT phosphorylation is essential for insulin-induced relaxation of rat vascular smooth muscle cells
about
Upregulation of AT2 receptor and iNOS impairs angiotensin II-induced contraction without endothelium influence in young normotensive diabetic ratsAdipocytokines in atherothrombosis: focus on platelets and vascular smooth muscle cells.The involvement of aldosterone on vascular insulin resistance: implications in obesity and type 2 diabetes.Thyroid hormone stimulates NO production via activation of the PI3K/Akt pathway in vascular myocytesImpaired coronary microvascular dilation correlates with enhanced vascular smooth muscle MLC phosphorylation in diabetesIncreased availability of angiotensin AT 1 receptors leads to sustained arterial constriction to angiotensin II in diabetes - role for Rho-kinase activationMolecular signatures for obesity and associated disorders identified through partial least square regression models.Impaired insulin-stimulated myosin phosphatase Rho-interacting protein signaling in diabetic Goto-Kakizaki vascular smooth muscle cells.Impaired insulin-mediated vasorelaxation in diabetic Goto-Kakizaki rats is caused by impaired Akt phosphorylationInsulin signalling in the heart.Contribution of insulin resistance to vascular dysfunction.Examining the role of insulin in the regulation of cardiovascular health.Abnormal Rho-associated kinase activity contributes to the dysfunctional myogenic response of cerebral arteries in type 2 diabetes.Skeletal Muscle Vascular Function: A Counterbalance of Insulin Action.Hyperglycemia- and hyperinsulinemia-induced insulin resistance causes alterations in cellular bioenergetics and activation of inflammatory signaling in lymphatic muscle.Airway epithelial-derived factor relaxes pulmonary vascular smooth muscle.A practical approach to parameter estimation applied to model predicting heart rate regulation.Insulin relaxes bladder via PI3K/AKT/eNOS pathway activation in mucosa: unfolded protein response-dependent insulin resistance as a cause of obesity-associated overactive bladder.Omega-3E treatment regulates matrix metalloproteinases and prevents vascular reactivity alterations in diabetic rat aorta.Serine racemase deficiency attenuates choroidal neovascularization and reduces nitric oxide and VEGF levels by retinal pigment epithelial cells.The Biased G-Protein-Coupled Receptor Agonism Bridges the Gap between the Insulin Receptor and the Metabolic Syndrome.Inhibition of CXCL12/CXCR4 suppresses pulmonary arterial smooth muscle cell proliferation and cell cycle progression via PI3K/Akt pathway under hypoxia.Increase in insulin-induced relaxation of consecutive arterial segments toward the periphery: Role of vascular oxidative state.Determinants of evolving metabolic and cardiovascular benefit/risk profiles of rosiglitazone therapy during the natural history of diabetes: molecular mechanisms in the context of integrated pathophysiology
P2860
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P2860
AKT phosphorylation is essential for insulin-induced relaxation of rat vascular smooth muscle cells
description
2006 nî lūn-bûn
@nan
2006 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2006年の論文
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2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年學術文章
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name
AKT phosphorylation is essenti ...... t vascular smooth muscle cells
@ast
AKT phosphorylation is essenti ...... t vascular smooth muscle cells
@en
type
label
AKT phosphorylation is essenti ...... t vascular smooth muscle cells
@ast
AKT phosphorylation is essenti ...... t vascular smooth muscle cells
@en
prefLabel
AKT phosphorylation is essenti ...... t vascular smooth muscle cells
@ast
AKT phosphorylation is essenti ...... t vascular smooth muscle cells
@en
P2860
P1476
AKT phosphorylation is essenti ...... t vascular smooth muscle cells
@en
P2093
Jin Hee Lee
Louis Ragolia
P2860
P304
P356
10.1152/AJPCELL.00125.2006
P577
2006-07-19T00:00:00Z