Neuroinflammation triggered by β-glucan/dectin-1 signaling enables CNS axon regeneration.
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Molecular Interactions of β-(1→3)-Glucans with Their ReceptorsRescue of neurodegeneration in the Fig4 null mouse by a catalytically inactive FIG4 transgene.Enhancing Nervous System Recovery through Neurobiologics, Neural Interface Training, and Neurorehabilitation.Overexpression of the monocyte chemokine CCL2 in dorsal root ganglion neurons causes a conditioning-like increase in neurite outgrowth and does so via a STAT3 dependent mechanism.Mincle Activation and the Syk/Card9 Signaling Axis Are Central to the Development of Autoimmune Disease of the Eye.GIT1 enhances neurite outgrowth by stimulating microtubule assembly.Reassembly of Excitable Domains after CNS Axon Regeneration.Reconnecting Eye to Brain.Mobile zinc increases rapidly in the retina after optic nerve injury and regulates ganglion cell survival and optic nerve regeneration.Neuroinflammation as Fuel for Axonal Regeneration in the Injured Vertebrate Central Nervous System.Profiling of Glycan Alterations in Regrowing Limb Tissues of Cynops orientalis.Microglia in central nervous system repair after injury.Hyper-IL-6: a potent and efficacious stimulator of RGC regeneration.Protective and Pathological Immunity during Central Nervous System Infections.Myeloid Cells in the Central Nervous System.Boosting Central Nervous System Axon Regeneration by Circumventing Limitations of Natural Cytokine Signaling.Reassembly of the axon initial segment and nodes of Ranvier in regenerated axons of the central nervous system.Pharmacological Tools to Activate Microglia and their Possible use to Study Neural Network Patho-physiology.La Deletion from Mouse Brain Alters Pre-tRNA Metabolism and Accumulation of Pre-5.8S rRNA, with Neuron Death and Reactive Astrocytosis.Optic nerve regeneration in the mouse is a complex trait modulated by genetic background.Critical Role of the CXCL10/C-X-C Chemokine Receptor 3 Axis in Promoting Leukocyte Recruitment and Neuronal Injury during Traumatic Optic Neuropathy Induced by Optic Nerve Crush.Optimizing Tumor Microenvironment for Cancer Immunotherapy: β-Glucan-Based Nanoparticles.Nogo receptor 1 is expressed by nearly all retinal ganglion cells.Wedelolactone from Vietnamese Eclipta prostrata (L) L. Protected Zymosan-induced shock in Mice.Low-Intensity Pulsed Ultrasound Protects Retinal Ganglion Cell From Optic Nerve Injury Induced Apoptosis via Yes Associated Protein.Damage-associated molecular pattern recognition is required for induction of retinal neuroprotective pathways in a sex-dependent manner.
P2860
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P2860
Neuroinflammation triggered by β-glucan/dectin-1 signaling enables CNS axon regeneration.
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2015 nî lūn-bûn
@nan
2015 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2015 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
name
Neuroinflammation triggered by β-glucan/dectin-1 signaling enables CNS axon regeneration.
@ast
Neuroinflammation triggered by β-glucan/dectin-1 signaling enables CNS axon regeneration.
@en
type
label
Neuroinflammation triggered by β-glucan/dectin-1 signaling enables CNS axon regeneration.
@ast
Neuroinflammation triggered by β-glucan/dectin-1 signaling enables CNS axon regeneration.
@en
prefLabel
Neuroinflammation triggered by β-glucan/dectin-1 signaling enables CNS axon regeneration.
@ast
Neuroinflammation triggered by β-glucan/dectin-1 signaling enables CNS axon regeneration.
@en
P2093
P2860
P356
P1476
Neuroinflammation triggered by β-glucan/dectin-1 signaling enables CNS axon regeneration.
@en
P2093
Katherine T Baldwin
Kevin S Carbajal
Roman J Giger
P2860
P304
P356
10.1073/PNAS.1423221112
P407
P577
2015-02-09T00:00:00Z