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Cardiovascular disease in diabetes: where does glucose fit in?

Cardiovascular disease in diabetes: where does glucose fit in?

http://www.wikidata.org/entity/Q35137148

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Diabetes and cardiovascular disease: changing the focus from glycemic control to improving long-term survival Protective mechanisms of mitochondria and heart function in diabetes Endothelial cell and platelet bioenergetics: effect of glucose and nutrient composition.High-intensity endurance training results in faster vessel-specific rate of vasorelaxation in type 1 diabetic rats Hyperglycemia impairs atherosclerosis regression in mice.Effect of low dose mineralocorticoid receptor antagonist eplerenone on glucose and lipid metabolism in healthy adult males Timed Bromocriptine-QR Therapy Reduces Progression of Cardiovascular Disease and Dysglycemia in Subjects with Well-Controlled Type 2 Diabetes Mellitus.Is it time to screen for the haptoglobin genotype to assess the cardiovascular risk profile and vitamin E therapy responsiveness in patients with diabetes?Clinical Update: Cardiovascular Disease in Diabetes Mellitus: Atherosclerotic Cardiovascular Disease and Heart Failure in Type 2 Diabetes Mellitus - Mechanisms, Management, and Clinical Considerations Vitamin E in the prevention of cardiovascular disease: the importance of proper patient selection.Is glucose control important for prevention of cardiovascular disease in diabetes?Glucose control in diabetes: which target level to aim for?The vascular smooth muscle cell: a therapeutic target in Type 2 diabetes?Understanding the type 2 diabetes mellitus and cardiovascular disease risk paradox.Targeting Select Cellular Stress Pathways to Prevent Hyperglycemia-Related Complications: Shifting the Paradigm.Therapeutic Targeting of Cellular Stress to Prevent Cardiovascular Disease: A Review of the Evidence.Low nanomolar caffeic acid attenuates high glucose-induced endothelial dysfunction in primary human umbilical-vein endothelial cells by affecting NF-κB and Nrf2 pathways.Vessel-specific rate of vasorelaxation is slower in diabetic rats.Glutathione prevents free fatty acids-induced oxidative stress and apoptosis in human brain vascular endothelial cells through Akt pathway.Hyperglycemia accelerates ATP-binding cassette transporter A1 degradation via an ERK-dependent pathway in macrophages.Interferon regulatory factor-1 as a positive regulator for high glucose-induced proliferation of vascular smooth muscle cells.

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Cardiovascular disease in diabetes: where does glucose fit in?

http://www.wikidata.org/entity/Q35137148

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2011 nî lūn-bûn

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2011 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2011 թվականի մայիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Cardiovascular disease in diabetes: where does glucose fit in?

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Cardiovascular disease in diabetes: where does glucose fit in?

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Cardiovascular disease in diabetes: where does glucose fit in?

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The Journal of Clinical Endocrinology and Metabolism

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Cardiovascular disease in diabetes: where does glucose fit in?

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Cecilia C Low Wang

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Glycated hemoglobin, diabetes, and cardiovascular risk in nondiabetic adults

Intensive Diabetes Treatment and Cardiovascular Disease in Patients with Type 1 Diabetes

Diabetes mellitus, fasting blood glucose concentration, and risk of vascular disease: a collaborative meta-analysis of 102 prospective studies

Intensive blood-glucose control with sulphonylureas or insulin compared with conventional treatment and risk of complications in patients with type 2 diabetes (UKPDS 33). UK Prospective Diabetes Study (UKPDS) Group

The effect of intensive treatment of diabetes on the development and progression of long-term complications in insulin-dependent diabetes mellitus. The Diabetes Control and Complications Trial Research Group

The platelet in diabetes: focus on prevention of ischemic events

Intensive blood glucose control and vascular outcomes in patients with type 2 diabetes

Effects of intensive glucose lowering in type 2 diabetes

10-year follow-up of intensive glucose control in type 2 diabetes

The pathobiology of diabetic complications: a unifying mechanism

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2367-2376

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10.1210/JC.2010-3011

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cardiovascular disease

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