Remodeling of the myocardium and potential targets in the collagen degradation and synthesis pathways.
about
Physiological Implications of Myocardial Scar StructureSecreted Frizzled-related protein 2 is a procollagen C proteinase enhancer with a role in fibrosis associated with myocardial infarctionTransforming growth factor β1 induces the expression of collagen type I by DNA methylation in cardiac fibroblastsWhy Is Infarct Expansion Such an Elusive Therapeutic Target?Mechanisms of Post-Infarct Left Ventricular RemodelingExercise training reduces fibrosis and matrix metalloproteinase dysregulation in the aging rat heart.Curcumin suppresses gelatinase B mediated norepinephrine induced stress in H9c2 cardiomyocytes.Cardiac mesenchymal stem cells contribute to scar formation after myocardial infarction.Increased collagen, per se, may not affect left ventricular function in spontaneously hypertensive rats.Diabetes influences cardiac extracellular matrix remodelling after myocardial infarction and subsequent development of cardiac dysfunction.Decreased Laminin Expression by Human Lung Epithelial Cells and Fibroblasts Cultured in Acellular Lung Scaffolds from Aged Mice.Cardiac-Restricted IGF-1Ea Overexpression Reduces the Early Accumulation of Inflammatory Myeloid Cells and Mediates Expression of Extracellular Matrix Remodelling Genes after Myocardial InfarctionHyperoxia and transforming growth factor β1 signaling in the post-ischemic mouse heart.Skeletal Myoblast Cell Sheet Implantation Ameliorates Both Systolic and Diastolic Cardiac Performance in Canine Dilated Cardiomyopathy Model.MicroRNA-503 promotes angiotensin II-induced cardiac fibrosis by targeting Apelin-13.Direct Renin Inhibitor Attenuates Left Ventricular Remodeling in Post-Myocardial Infarction Heart Failure MiceValsartan in the treatment of heart attack survivorsThe origin and arrhythmogenic potential of fibroblasts in cardiac disease.Mechanical Regulation of Cardiac Aging in Model Systems.The effect of polypyrrole on arteriogenesis in an acute rat infarct modelAging, exercise, and extracellular matrix in the heartEmbryonic stem cells in cardiac repair and regeneration.The cardiac fibroblast: functional and electrophysiological considerations in healthy and diseased hearts.Atrial fibrillation and heart failure in the elderly.The paradox of left ventricular assist device unloading and myocardial recovery in end-stage dilated cardiomyopathy: implications for heart failure in the elderly.Thrombospondin-4, tumour necrosis factor-like weak inducer of apoptosis (TWEAK) and its receptor Fn14: novel extracellular matrix modulating factors in cardiac remodelling.Pacing-induced cardiomyopathy: pathophysiological insights through matrix metalloproteinases.Cardiac Physiology of Aging: Extracellular Considerations.Loss or inhibition of uPA or MMP-9 attenuates LV remodeling and dysfunction after acute pressure overload in mice.Extracellular matrix-mediated cellular communication in the heart.Apelin prevents cardiac fibroblast activation and collagen production through inhibition of sphingosine kinase 1.Transplanted embryonic stem cells following mouse myocardial infarction inhibit apoptosis and cardiac remodeling.Expression of Ca-activated K Channels and Their Role in Proliferation of Rat Cardiac Fibroblasts.Effects of cholesterol on proliferation and functional protein expression in rabbit bile duct fibroblasts.Increasing cardiac contractility after myocardial infarction exacerbates cardiac injury and pump dysfunctionUse of Heart Failure Medical Therapies Among Patients With Left Ventricular Assist Devices: Insights From INTERMACS.Direct intracardiac injection of umbilical cord-derived stromal cells and umbilical cord blood-derived endothelial cells for the treatment of ischemic cardiomyopathy.Attraction and repulsion of spiral waves by inhomogeneity of conduction anisotropy--a model of spiral wave interaction with electrical remodeling of heart tissue.Myocardial expression of PDECGF is associated with extracellular matrix remodeling in experimental myocardial infarction in rats.Effect of acute and chronic exercise on plasma matrix metalloproteinase and total antioxidant levels.
P2860
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P2860
Remodeling of the myocardium and potential targets in the collagen degradation and synthesis pathways.
description
2003 nî lūn-bûn
@nan
2003 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի մարտին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Remodeling of the myocardium a ...... dation and synthesis pathways.
@ast
Remodeling of the myocardium a ...... dation and synthesis pathways.
@en
type
label
Remodeling of the myocardium a ...... dation and synthesis pathways.
@ast
Remodeling of the myocardium a ...... dation and synthesis pathways.
@en
prefLabel
Remodeling of the myocardium a ...... dation and synthesis pathways.
@ast
Remodeling of the myocardium a ...... dation and synthesis pathways.
@en
P356
P1476
Remodeling of the myocardium a ...... dation and synthesis pathways.
@en
P2093
Bodh I Jugdutt
P356
10.2174/1568006033337276
P577
2003-03-01T00:00:00Z