BAFF selectively enhances the survival of plasmablasts generated from human memory B cells. - Wikidata - wikimedia - TriplyDB

BAFF selectively enhances the survival of plasmablasts generated from human memory B cells.

BAFF selectively enhances the survival of plasmablasts generated from human memory B cells.

http://www.wikidata.org/entity/Q35140484

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Graft-versus-tumor response in patients with multiple myeloma is associated with antibody response to BCMA, a plasma-cell membrane receptor The current status of targeting BAFF/BLyS for autoimmune diseases Dendritic Cells and Their Multiple Roles during Malaria Infection B-cell survival factors in autoimmune rheumatic disorders Unraveling the Role of Allo-Antibodies and Transplant Injury A phase III, randomized, placebo-controlled study of belimumab, a monoclonal antibody that inhibits B lymphocyte stimulator, in patients with systemic lupus erythematosus A therapeutic role for BLyS antagonists A soluble BAFF antagonist, BR3-Fc, decreases peripheral blood B cells and lymphoid tissue marginal zone and follicular B cells in cynomolgus monkeys Elevated pretransplantation soluble BAFF is associated with an increased risk of acute antibody-mediated rejection Functional and clinical aspects of the B-cell-activating factor (BAFF): a narrative review BCMA is essential for the survival of long-lived bone marrow plasma cells Regulatory roles of the tumor necrosis factor receptor BCMA.Local BAFF gene silencing suppresses Th17-cell generation and ameliorates autoimmune arthritis Human BLyS facilitates engraftment of human PBL derived B cells in immunodeficient mice.Raised expression of APRIL in Chinese patients with immune thrombocytopenia and its clinical implications.Long-lived plasma cells in autoimmunity: lessons from B-cell depleting therapy.Reduced tumour necrosis factor receptor superfamily 13C inversely correlated with tumour necrosis factor superfamily 13B in patients with immune thrombocytopenia.APRIL is overexpressed in cancer: link with tumor progression Autoantibody-dependent and autoantibody-independent roles for B cells in systemic lupus erythematosus: past, present, and future Novel anti-B-cell maturation antigen antibody-drug conjugate (GSK2857916) selectively induces killing of multiple myeloma.B cell activating factor (BAFF) and T cells cooperate to breach B cell tolerance in lupus-prone New Zealand Black (NZB) mice B cell abnormalities in systemic lupus erythematosus Immunopathogenesis of thyroid eye disease: emerging paradigms Malaria infection alters the expression of B-cell activating factor resulting in diminished memory antibody responses and survival.The role of B cells in solid organ transplantation.Paucity of clinical disease despite serological autoimmunity and kidney pathology in lupus-prone New Zealand mixed 2328 mice deficient in BAFF High levels of B-cell activating factor in patients with active chronic graft-versus-host disease.Marginal zone B cells: virtues of innate-like antibody-producing lymphocytes.Similarities and differences between selective and nonselective BAFF blockade in murine SLE Systems vaccinology.Innate signaling networks in mucosal IgA class switching Interleukin 6 signaling promotes anti-aquaporin 4 autoantibody production from plasmablasts in neuromyelitis optica.Enhanced immunogenicity of pneumococcal surface adhesin A (PsaA) in mice via fusion to recombinant human B lymphocyte stimulator (BLyS)Recovery of B-cell homeostasis after rituximab in chronic graft-versus-host disease.B lymphocyte stimulator (BLyS) isoforms in systemic lupus erythematosus: disease activity correlates better with blood leukocyte BLyS mRNA levels than with plasma BLyS protein levels.Innate control of B cell responses.Evaluation of B lymphocyte stimulator and a proliferation inducing ligand as candidate biomarkers in lupus nephritis based on clinical and histopathological outcome following induction therapy.Dual B cell immunotherapy is superior to individual anti-CD20 depletion or BAFF blockade in murine models of spontaneous or accelerated lupus.Systems biology of vaccination for seasonal influenza in humans.Characterization of B cells in muscle-specific kinase antibody myasthenia gravis.

P2860

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P2860

BAFF selectively enhances the survival of plasmablasts generated from human memory B cells.

http://www.wikidata.org/entity/Q35140484

description

2003 nî lūn-bûn

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2003 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2003 թվականի հուլիսին հրատարակված գիտական հոդված

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

@zh-hk

2003年論文

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2003年論文

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2003年论文

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name

BAFF selectively enhances the ...... ted from human memory B cells.

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BAFF selectively enhances the ...... ted from human memory B cells.

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type

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BAFF selectively enhances the ...... ted from human memory B cells.

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BAFF selectively enhances the ...... ted from human memory B cells.

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BAFF selectively enhances the ...... ted from human memory B cells.

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BAFF selectively enhances the ...... ted from human memory B cells.

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P1433

Journal of Clinical Investigation

P1476

BAFF selectively enhances the ...... ated from human memory B cells

@en

P2093

Christine Ambrose

http://www.w3.org/2001/XMLSchema#string

Danielle T Avery

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Julia I Ellyard

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Marilyn Thien

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Robert Brink

http://www.w3.org/2001/XMLSchema#string

Sarah A Bixler

http://www.w3.org/2001/XMLSchema#string

Stuart G Tangye

http://www.w3.org/2001/XMLSchema#string

Susan L Kalled

http://www.w3.org/2001/XMLSchema#string

P2860

BAFF, a novel ligand of the tumor necrosis factor family, stimulates B cell growth

TACI and BCMA are receptors for a TNF homologue implicated in B-cell autoimmune disease

B cell maturation protein is a receptor for the tumor necrosis factor family member TALL-1

NF-AT activation induced by a CAML-interacting member of the tumor necrosis factor receptor superfamily

Cross-linking of OX40 ligand, a member of the TNF/NGF cytokine family, induces proliferation and differentiation in murine splenic B cells

BLyS: member of the tumor necrosis factor family and B lymphocyte stimulator

TALL-1 is a novel member of the TNF family that is down-regulated by mitogens

Identification and characterization of a novel cytokine, THANK, a TNF homologue that activates apoptosis, nuclear factor-kappaB, and c-Jun NH2-terminal kinase

Molecular interactions mediating T-B lymphocyte collaboration in human lymphoid follicles. Roles of T cell-B-cell-activating molecule (5c8 antigen) and CD40 in contact-dependent help

Identification of a novel receptor for B lymphocyte stimulator that is mutated in a mouse strain with severe B cell deficiency

P304

286-297

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scholarly article

P356

10.1172/JCI18025

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10.1172/JCI200318025

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P407

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2

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112

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P50

Philip D Hodgkin

Fabienne Mackay

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2003-07-01T00:00:00Z

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10659980

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12865416

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164292

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