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Ube3a imprinting impairs circadian robustness in Angelman syndrome models

Ube3a imprinting impairs circadian robustness in Angelman syndrome models

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Epigenetic mechanisms in diurnal cycles of metabolism and neurodevelopment Epigenetic regulation of UBE3A and roles in human neurodevelopmental disorders.Genomic Imprinting: A New Epigenetic Perspective of Sleep Regulation.Maternal Ube3a Loss Disrupts Sleep Homeostasis But Leaves Circadian Rhythmicity Largely Intact.Persistent neuronal Ube3a expression in the suprachiasmatic nucleus of Angelman syndrome model mice.New Perspectives on Genomic Imprinting, an Essential and Multifaceted Mode of Epigenetic Control in the Developing and Adult Brain.Aberrant Proteostasis of BMAL1 Underlies Circadian Abnormalities in a Paradigmatic mTOR-opathy.Meta-analysis of transcriptomic datasets identifies genes enriched in the mammalian circadian pacemaker.Abnormal coherence and sleep composition in children with Angelman syndrome: a retrospective EEG study.

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Ube3a imprinting impairs circadian robustness in Angelman syndrome models

http://www.wikidata.org/entity/Q35143079

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2015 nî lūn-bûn

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2015 թվականի փետրվարին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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Ube3a imprinting impairs circadian robustness in Angelman syndrome models

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Ube3a imprinting impairs circadian robustness in Angelman syndrome models

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type

label

Ube3a imprinting impairs circadian robustness in Angelman syndrome models

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Ube3a imprinting impairs circadian robustness in Angelman syndrome models

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prefLabel

Ube3a imprinting impairs circadian robustness in Angelman syndrome models

@ast

Ube3a imprinting impairs circadian robustness in Angelman syndrome models

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J.CUB.2014.12.047

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Current Biology

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Ube3a imprinting impairs circadian robustness in Angelman syndrome models

@en

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Carl Hirschie Johnson

http://www.w3.org/2001/XMLSchema#string

Shu-qun Shi

http://www.w3.org/2001/XMLSchema#string

P2860

Redundant function of REV-ERBalpha and beta and non-essential role for Bmal1 cycling in transcriptional regulation of intracellular circadian rhythms

The E3 ubiquitin ligase UBE3A is an integral component of the molecular circadian clock through regulating the BMAL1 transcription factor

SCFFbxl3 controls the oscillation of the circadian clock by directing the degradation of cryptochrome proteins

The basic-helix-loop-helix-PAS orphan MOP3 forms transcriptionally active complexes with circadian and hypoxia factors

Competing E3 ubiquitin ligases govern circadian periodicity by degradation of CRY in nucleus and cytoplasm

FBXL21 regulates oscillation of the circadian clock through ubiquitination and stabilization of cryptochromes

Dynamic circadian protein-protein interaction networks predict temporal organization of cellular functions

PERIOD2::LUCIFERASE real-time reporting of circadian dynamics reveals persistent circadian oscillations in mouse peripheral tissues.

Topoisomerase inhibitors unsilence the dormant allele of Ube3a in neurons

Disruption of the clock components CLOCK and BMAL1 leads to hypoinsulinaemia and diabetes

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537-545

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scholarly article

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10.1016/J.CUB.2014.12.047

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5

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25

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Rebecca A. Ihrie

Terry Jo V Bichell

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2015-02-05T00:00:00Z

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272099932

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25660546

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circadian rhythm

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4348236

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