Calcium-permeable AMPA/kainate receptors mediate toxicity and preconditioning by oxygen-glucose deprivation in oligodendrocyte precursors. - Wikidata - wikimedia - TriplyDB

Calcium-permeable AMPA/kainate receptors mediate toxicity and preconditioning by oxygen-glucose deprivation in oligodendrocyte precursors.

Calcium-permeable AMPA/kainate receptors mediate toxicity and preconditioning by oxygen-glucose deprivation in oligodendrocyte precursors.

http://www.wikidata.org/entity/Q35144325

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Neurotransmitter receptors in the life and death of oligodendrocytes Systematic Review of Pharmacological Properties of the Oligodendrocyte Lineage AMPA-Kainate Receptor Inhibition Promotes Neurologic Recovery in Premature Rabbits with Intraventricular Hemorrhage Rat cerebellar slice cultures exposed to bilirubin evidence reactive gliosis, excitotoxicity and impaired myelinogenesis that is prevented by AMPA and TNF-α inhibitors Effect of MK-801 and Clozapine on the Proteome of Cultured Human Oligodendrocytes.Pathophysiology of glia in perinatal white matter injury.Neonatal Hypoxia Ischaemia: Mechanisms, Models, and Therapeutic Challenges.Glutamate receptors on myelinated spinal cord axons: I. GluR6 kainate receptors The encephalopathy of prematurity--brain injury and impaired brain development inextricably intertwined.Imaging selective vulnerability in the developing nervous system.NMDA receptor blockade with memantine attenuates white matter injury in a rat model of periventricular leukomalacia.Hypoxic-ischemic encephalopathy in the term infant.Glutamate transporter EAAT2 expression is up-regulated in reactive astrocytes in human periventricular leukomalacia.RIP1 kinase mediates arachidonic acid-induced oxidative death of oligodendrocyte precursors.Regulation of glutamate transport in developing rat oligodendrocytes.The challenge of understanding cerebral white matter injury in the premature infant.Intranasal administration of aTf protects and repairs the neonatal white matter after a cerebral hypoxic-ischemic event.Protective Effects of Berberine on Oxygen-Glucose Deprivation/Reperfusion on Oligodendrocyte Cell Line (OLN-93).Developmental regulation of alpha-amino-3-hydroxy-5-methyl-4-isoxazole-propionic acid receptor subunit expression in forebrain and relationship to regional susceptibility to hypoxic/ischemic injury. II. Human cerebral white matter and cortex.Role of metabotropic glutamate receptors in oligodendrocyte excitotoxicity and oxidative stress Early proliferation does not prevent the loss of oligodendrocyte progenitor cells during the chronic phase of secondary degeneration in a CNS white matter tract.The developing oligodendrocyte: key cellular target in brain injury in the premature infant Extracellular acidic pH inhibits oligodendrocyte precursor viability, migration, and differentiation Developmental regulation of alpha-amino-3-hydroxy-5-methyl-4-isoxazole-propionic acid receptor subunit expression in forebrain and relationship to regional susceptibility to hypoxic/ischemic injury. I. Rodent cerebral white matter and cortex.Oligodendroglial differentiation induces mitochondrial genes and inhibition of mitochondrial function represses oligodendroglial differentiation.Axon-glia synapses are highly vulnerable to white matter injury in the developing brain Systemic prenatal insults disrupt telencephalon development: implications for potential interventions.Synaptic lability after experience-dependent plasticity is not mediated by calcium-permeable AMPARs Oligodendrocytes Are Targets of HIV-1 Tat: NMDA and AMPA Receptor-Mediated Effects on Survival and Development.Myelin Proteolipid Protein Complexes with αv Integrin and AMPA Receptors In Vivo and Regulates AMPA-Dependent Oligodendrocyte Progenitor Cell Migration through the Modulation of Cell-Surface GluR2 Expression.Autocrine glutamate signaling promotes glioma cell invasion.Derivation of glial restricted precursors from E13 mice.Neuropathologic substrate of cerebral palsy.Role of glutamate receptors in periventricular leukomalacia.The pathophysiology of ischemic injury to developing white matter Pathogenesis of cerebral white matter injury of prematurity.Rodent brain slice model for the study of white matter injury.Molecular mechanisms involved in injury to the preterm brain.Tumor necrosis factor alpha mediates lipopolysaccharide-induced microglial toxicity to developing oligodendrocytes when astrocytes are present.Inhibition of cystine uptake disrupts the growth of primary brain tumors.

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P2860

Calcium-permeable AMPA/kainate receptors mediate toxicity and preconditioning by oxygen-glucose deprivation in oligodendrocyte precursors.

http://www.wikidata.org/entity/Q35144325

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2003 nî lūn-bûn

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2003 թուականի Մայիսին հրատարակուած գիտական յօդուած

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Calcium-permeable AMPA/kainate ...... in oligodendrocyte precursors.

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Calcium-permeable AMPA/kainate ...... in oligodendrocyte precursors.

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Calcium-permeable AMPA/kainate ...... in oligodendrocyte precursors.

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Proceedings of the National Academy of Sciences of the United States of America

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Calcium-permeable AMPA/kainate ...... in oligodendrocyte precursors.

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Frances E Jensen

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Joseph J Volpe

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Paul A Rosenberg

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Wenbin Deng

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P2860

Phosphorylation of the AMPA receptor subunit GluR2 differentially regulates its interaction with PDZ domain-containing proteins

Molecular mechanisms of calcium-dependent excitotoxicity

Preparation of separate astroglial and oligodendroglial cell cultures from rat cerebral tissue

Cloned glutamate receptors

The oligodendrocyte precursor cell in health and disease.

Non-N-methyl-D-aspartate glutamate receptors mediate oxygen--glucose deprivation-induced oligodendroglial injury.

Cooperation between two growth factors promotes extended self-renewal and inhibits differentiation of oligodendrocyte-type-2 astrocyte (O-2A) progenitor cells.

Ca(2+)-permeable AMPA receptors induce phosphorylation of cAMP response element-binding protein through a phosphatidylinositol 3-kinase-dependent stimulation of the mitogen-activated protein kinase signaling cascade in neurons.

Biology of oligodendrocyte and myelin in the mammalian central nervous system.

Ca2+-Zn2+ permeable AMPA or kainate receptors: possible key factors in selective neurodegeneration.

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oligodendrocyte

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