Deficient IFN signaling by myeloid cells leads to MAVS-dependent virus-induced sepsis. - Wikidata - wikimedia - TriplyDB

Deficient IFN signaling by myeloid cells leads to MAVS-dependent virus-induced sepsis.

Deficient IFN signaling by myeloid cells leads to MAVS-dependent virus-induced sepsis.

http://www.wikidata.org/entity/Q35152251

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A Mouse Model of Zika Virus Pathogenesis Knocking on Closed Doors: Host Interferons Dynamically Regulate Blood-Brain Barrier Function during Viral Infections of the Central Nervous System Loss of Ifnar1 in Pancreatic Acinar Cells Ameliorates the Disease Course of Acute Pancreatitis Zika Virus Antagonizes Type I Interferon Responses during Infection of Human Dendritic Cells The Complement System in Flavivirus Infections Defining New Therapeutics Using a More Immunocompetent Mouse Model of Antibody-Enhanced Dengue Virus Infection.Viral pathogen-associated molecular patterns regulate blood-brain barrier integrity via competing innate cytokine signals.Bacterial programming of host responses: coordination between type I interferon and cell death.Virus-like particles activate type I interferon pathways to facilitate post-exposure protection against Ebola virus infection.Oropouche virus infection and pathogenesis are restricted by MAVS, IRF-3, IRF-7, and type I interferon signaling pathways in nonmyeloid cells.Type I interferons in viral control and immune regulation Systems biology: A tool for charting the antiviral landscape.Encephalitic Arboviruses: Emergence, Clinical Presentation, and Neuropathogenesis.Regional astrocyte IFN signaling restricts pathogenesis during neurotropic viral infection Viral sensing at the blood-brain barrier: new roles for innate immunity at the CNS vasculature.Innate Immune Basis for Rift Valley Fever Susceptibility in Mouse Models.Is the third interferon a charm?Distinct Effects of Type I and III Interferons on Enteric Viruses.Splenic macrophages are required for protective innate immunity against West Nile virus.

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P2860

Deficient IFN signaling by myeloid cells leads to MAVS-dependent virus-induced sepsis.

http://www.wikidata.org/entity/Q35152251

description

2014 nî lūn-bûn

@nan

2014 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2014年の論文

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2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

Deficient IFN signaling by myeloid cells leads to MAVS-dependent virus-induced sepsis.

@ast

Deficient IFN signaling by myeloid cells leads to MAVS-dependent virus-induced sepsis.

@en

type

label

Deficient IFN signaling by myeloid cells leads to MAVS-dependent virus-induced sepsis.

@ast

Deficient IFN signaling by myeloid cells leads to MAVS-dependent virus-induced sepsis.

@en

prefLabel

Deficient IFN signaling by myeloid cells leads to MAVS-dependent virus-induced sepsis.

@ast

Deficient IFN signaling by myeloid cells leads to MAVS-dependent virus-induced sepsis.

@en

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JOURNAL.PPAT.1004086

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Deficient IFN signaling by myeloid cells leads to MAVS-dependent virus-induced sepsis.

@en

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Hilario J Ramos

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John P Atkinson

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Kristin Y Kim

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Matthew Gorman

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Mehul S Suthar

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Shilpa Aggarwal

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Xiaobo Wu

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P2860

Characterization of reemerging chikungunya virus

A mouse model for Chikungunya: young age and inefficient type-I interferon signaling are risk factors for severe disease

Caspase-12 controls West Nile virus infection via the viral RNA receptor RIG-I.

The inflammasome: a molecular platform triggering activation of inflammatory caspases and processing of proIL-beta

STAT2 is a primary target for measles virus V protein-mediated alpha/beta interferon signaling inhibition

The adaptor MAVS promotes NLRP3 mitochondrial localization and inflammasome activation

Human dermal mast cells contain and release tumor necrosis factor alpha, which induces endothelial leukocyte adhesion molecule 1

A diverse range of gene products are effectors of the type I interferon antiviral response

NS5 of dengue virus mediates STAT2 binding and degradation

Development of a highly protective combination monoclonal antibody therapy against Chikungunya virus

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10.1371/JOURNAL.PPAT.1004086

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10

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Michael S. Diamond

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2014-04-17T00:00:00Z

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