Ambient particulate air pollution induces oxidative stress and alterations of mitochondria and gene expression in brown and white adipose tissues. - Wikidata - wikimedia - TriplyDB

Ambient particulate air pollution induces oxidative stress and alterations of mitochondria and gene expression in brown and white adipose tissues.

Ambient particulate air pollution induces oxidative stress and alterations of mitochondria and gene expression in brown and white adipose tissues.

http://www.wikidata.org/entity/Q35154881

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Ambient particulate matter exposure and cardiovascular diseases: a focus on progenitor and stem cells Influencing Factors of Thermogenic Adipose Tissue Activity Placental DNA hypomethylation in association with particulate air pollution in early life Repeated ozone exposure exacerbates insulin resistance and activates innate immune response in genetically susceptible mice Carbon dioxide emissions and change in prevalence of obesity and diabetes in the United States: an ecological study Environmental endocrine disruption of energy metabolism and cardiovascular risk.Ambient Air Pollution and Type 2 Diabetes: A Systematic Review of Epidemiologic Research CD36-dependent 7-ketocholesterol accumulation in macrophages mediates progression of atherosclerosis in response to chronic air pollution exposure.Air pollution as a risk factor for type 2 diabetes.Ambient fine particulate matter induces apoptosis of endothelial progenitor cells through reactive oxygen species formation Long-term exposure to air pollution and type 2 diabetes mellitus in a multiethnic cohort.A longitudinal cohort study of body mass index and childhood exposure to secondhand tobacco smoke and air pollution: the Southern California Children's Health Study AMPK is required for PM2.5-induced autophagy in human lung epithelial A549 cells.Air-Pollution and Cardiometabolic Diseases (AIRCMD): a prospective study investigating the impact of air pollution exposure and propensity for type II diabetes Air pollution and type 2 diabetes: mechanistic insights Effect of co-exposure to nickel and particulate matter on insulin resistance and mitochondrial dysfunction in a mouse model.Exposure to fine airborne particulate matter induces macrophage infiltration, unfolded protein response, and lipid deposition in white adipose tissue Does near-roadway air pollution contribute to childhood obesity?Effects of airborne pollutants on mitochondrial DNA methylation.Exercise regulation of adipose tissue.Exposure to environmentally persistent free radicals during gestation lowers energy expenditure and impairs skeletal muscle mitochondrial function in adult mice.Ambient fine particulate matter and ozone exposures induce inflammation in epicardial and perirenal adipose tissues in rats fed a high fructose diet Predictive and Prognostic Biomarkers of Respiratory Diseases due to Particulate Matter Exposure.The mosaic theory revisited: common molecular mechanisms coordinating diverse organ and cellular events in hypertension Toxicological and epidemiological studies of cardiovascular effects of ambient air fine particulate matter (PM2.5) and its chemical components: coherence and public health implications.Ambient air pollution: an emerging risk factor for diabetes mellitus.Metabolomics analysis reveals that benzo[a]pyrene, a component of PM2.5, promotes pulmonary injury by modifying lipid metabolism in a phospholipase A2-dependent manner in vivo and in vitro.Lipidomic Adaptations in White and Brown Adipose Tissue in Response to Exercise Demonstrate Molecular Species-Specific Remodeling Pro-inflammatory response and oxidative stress induced by specific components in ambient particulate matter in human bronchial epithelial cells.Long-term exposure to traffic-related air pollution and diabetes-associated mortality: a cohort study.Traffic-related air pollution is associated with cardio-metabolic biomarkers in general residents.Residential proximity to major roadways, fine particulate matter, and adiposity: The framingham heart study.Flavone protects HBE cells from DNA double-strand breaks caused by PM2.5.Effect of Particulate Matter Air Pollution on Cardiovascular Oxidative Stress Pathways.The role of oxidative stress in citreoviridin-induced DNA damage in human liver-derived HepG2 cells.Inactivation of TNF/LT locus alters mouse metabolic response to concentrated ambient PM2.5.Residential Proximity to Major Roadways, Fine Particulate Matter, and Hepatic Steatosis: The Framingham Heart Study.Effects of prenatal exposure to ambient air pollutant PM10 on ultrasound-measured fetal growth.Mitochondrial toxicity of tobacco smoke and air pollution.Pollution and respiratory disease: can diet or supplements help? A review.

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P2860

Ambient particulate air pollution induces oxidative stress and alterations of mitochondria and gene expression in brown and white adipose tissues.

http://www.wikidata.org/entity/Q35154881

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2011 nî lūn-bûn

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2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2011 թվականի հուլիսին հրատարակված գիտական հոդված

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2011年の論文

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Ambient particulate air pollut ...... own and white adipose tissues.

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Ambient particulate air pollut ...... own and white adipose tissues.

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Particle and Fibre Toxicology

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Aixia Wang

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Ian P Hotchkiss

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Jack R Harkema

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James G Wagner

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Lori A Bramble

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Lung-Chi Chen

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Mianhua Zhong

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Morton Lippmann

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Qinghua Sun

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Ryan P Lewandowski

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P2860

Long-term inhalation exposure to nickel nanoparticles exacerbated atherosclerosis in a susceptible mouse model

Air pollution exposure potentiates hypertension through reactive oxygen species-mediated activation of Rho/ROCK

Mitochondrial complex III-generated oxidants activate ASK1 and JNK to induce alveolar epithelial cell death following exposure to particulate matter air pollution

Obesity as a medical problem

Long-term air pollution exposure and acceleration of atherosclerosis and vascular inflammation in an animal model

The hydrolipidic ratio in age-related maturation of adipose tissues.

UCP1 induction during recruitment of brown adipocytes in white adipose tissue is dependent on cyclooxygenase activity.

Gene expression of PPARgamma and PGC-1alpha in human omental and subcutaneous adipose tissues is related to insulin resistance markers and mediates beneficial effects of physical training.

Chronic peroxisome proliferator-activated receptor gamma (PPARgamma) activation of epididymally derived white adipocyte cultures reveals a population of thermogenically competent, UCP1-containing adipocytes molecularly distinct from classic brown ad

Association between fine particulate matter and diabetes prevalence in the U.S.

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scholarly article

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10.1186/1743-8977-8-20

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