A role for ethanol-induced oxidative stress in controlling lineage commitment of mesenchymal stromal cells through inhibition of Wnt/beta-catenin signaling - Wikidata - wikimedia - TriplyDB

A role for ethanol-induced oxidative stress in controlling lineage commitment of mesenchymal stromal cells through inhibition of Wnt/beta-catenin signaling

A role for ethanol-induced oxidative stress in controlling lineage commitment of mesenchymal stromal cells through inhibition of Wnt/beta-catenin signaling

http://www.wikidata.org/entity/Q35156190

about

Targeting disease through novel pathways of apoptosis and autophagy Osteoblast-adipocyte lineage plasticity in tissue development, maintenance and pathology Calcium-mediated repression of β-catenin and its transcriptional signaling mediates neural crest cell death in an avian model of fetal alcohol syndrome Novel Akt activator SC-79 is a potential treatment for alcohol-induced osteonecrosis of the femoral head Exogenous activation of Wnt/β-catenin signaling attenuates binge alcohol-induced deficient bone fracture healing.Loss of functional NADPH oxidase 2 protects against alcohol-induced bone resorption in female p47phox-/- mice.Diabetic osteopenia by decreased β-catenin signaling is partly induced by epigenetic derepression of sFRP-4 gene Wnt1, FoxO3a, and NF-kappaB oversee microglial integrity and activation during oxidant stress.Wnt1 neuroprotection translates into improved neurological function during oxidant stress and cerebral ischemia through AKT1 and mitochondrial apoptotic pathways.WISP1: Clinical insights for a proliferative and restorative member of the CCN family Inhibition of NADPH oxidases prevents chronic ethanol-induced bone loss in female rats.Osteoporosis and bone fractures in alcoholic liver disease: a meta-analysis.Effects of nutrition and alcohol consumption on bone loss Osteogenesis of Adipose-Derived Stem Cells.Effects of Combined Exposure to Lead and High-Fat Diet on Bone Quality in Juvenile Male Mice Vitamin D supplementation protects against bone loss associated with chronic alcohol administration in female mice.Heavy metal lead exposure, osteoporotic-like phenotype in an animal model, and depression of Wnt signaling Alcohol: A Simple Nutrient with Complex Actions on Bone in the Adult Skeleton.Reactive Oxygen Species Differentially Regulate Bone Turnover in an Age-Specific Manner in Catalase Transgenic Female Mice.Influence of fat/carbohydrate ratio on progression of fatty liver disease and on development of osteopenia in male rats fed alcohol via total enteral nutrition (TEN).A look behind the scenes: the risk and pathogenesis of primary osteoporosis.Effect of alendronate on healing of extraction sockets and healing around implants.Lgr4 Expression in Osteoblastic Cells Is Suppressed by Hydrogen Peroxide Treatment.N-acetylcysteine supplementation decreases osteoclast differentiation and increases bone mass in mice fed a high-fat diet.Partial Protection by Dietary Antioxidants Against Ethanol-Induced Osteopenia and Changes in Bone Morphology in Female Mice.Activation of Wnt/β-catenin pathway by exogenous Wnt1 protects SH-SY5Y cells against 6-hydroxydopamine toxicity.Cellular and molecular mechanisms of alcohol-induced osteopenia.Titanium topography controls FoxO/beta-catenin signaling.CaMKII represses transcriptionally active β-catenin to mediate acute ethanol neurodegeneration and can phosphorylate β-catenin.PPARγ and Oxidative Stress: Con(β) Catenating NRF2 and FOXO Oxidative Stress in Stem Cell Aging.Chronic High Dose Alcohol Induces Osteopenia via Activation of mTOR Signaling in Bone Marrow Mesenchymal Stem Cells.Alcohol-related deficient fracture healing is associated with activation of FoxO transcription factors in mice.Female mice lacking p47phox have altered adipose tissue gene expression and are protected against high fat-induced obesity.Vitamin E improved bone strength and bone minerals in male rats given alcohol.The protective effect of PFTα on alcohol-induced osteonecrosis of the femoral head.The Impact of Lipid Types and Liposomal Formulations on Osteoblast Adiposity and Mineralization.Diet-derived phenolic acids regulate osteoblast and adipocyte lineage commitment and differentiation in young mice.

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P2860

A role for ethanol-induced oxidative stress in controlling lineage commitment of mesenchymal stromal cells through inhibition of Wnt/beta-catenin signaling

http://www.wikidata.org/entity/Q35156190

description

2010 nî lūn-bûn

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2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

A role for ethanol-induced oxi ...... of Wnt/beta-catenin signaling

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A role for ethanol-induced oxi ...... of Wnt/beta-catenin signaling

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A role for ethanol-induced oxi ...... of Wnt/beta-catenin signaling

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A role for ethanol-induced oxi ...... of Wnt/beta-catenin signaling

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A role for ethanol-induced oxi ...... of Wnt/beta-catenin signaling

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A role for ethanol-induced oxi ...... of Wnt/beta-catenin signaling

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Journal of Bone and Mineral Research

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A role for ethanol-induced oxi ...... of Wnt/beta-catenin signaling

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P2093

Jin-Ran Chen

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Martin J Ronis

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Michael L Blackburn

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Oxana P Lazarenko

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Thomas M Badger

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P2860

Cytokines promote Wnt signaling and inflammation and impair the normal differentiation and lipid accumulation in 3T3-L1 preadipocytes

Control of beta-catenin phosphorylation/degradation by a dual-kinase mechanism

Nongenotropic, sex-nonspecific signaling through the estrogen or androgen receptors: dissociation from transcriptional activity

Alcohol alters whole body composition, inhibits bone formation, and increases bone marrow adiposity in rats

Protective effects of estradiol on ethanol-induced bone loss involve inhibition of reactive oxygen species generation in osteoblasts and downstream activation of the extracellular signal-regulated kinase/signal transducer and activator of transcript

Attenuation of the self-renewal of transit-amplifying osteoblast progenitors in the murine bone marrow by 17 beta-estradiol

Marrow stromal cells as stem cells for nonhematopoietic tissues

Different molecular mechanisms underlie ethanol-induced bone loss in cycling and pregnant rats.

Chronic ethanol consumption inhibits postlactational anabolic bone rebuilding in female rats.

Short term effects on bone quality associated with consumption of soy protein isolate and other dietary protein sources in rapidly growing female rats.

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5

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