Cell stress increases ATP release in NLRP3 inflammasome-mediated autoinflammatory diseases, resulting in cytokine imbalance - Wikidata - wikimedia - TriplyDB

Cell stress increases ATP release in NLRP3 inflammasome-mediated autoinflammatory diseases, resulting in cytokine imbalance

Cell stress increases ATP release in NLRP3 inflammasome-mediated autoinflammatory diseases, resulting in cytokine imbalance

http://www.wikidata.org/entity/Q35157045

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Innovative Target Therapies Are Able to Block the Inflammation Associated with Dysfunction of the Cholesterol Biosynthesis Pathway Molecular mechanisms regulating NLRP3 inflammasome activation NLRP3 inflammasome and its inhibitors: a review The NLRP3 inflammasome modulates sleep and NREM sleep delta power induced by spontaneous wakefulness, sleep deprivation and lipopolysaccharide.Functional Tissue Analysis Reveals Successful Cryopreservation of Human Osteoarthritic Synovium Extracellular ATP and other nucleotides-ubiquitous triggers of intercellular messenger release.Anti-inflammatory panacea? The expanding therapeutics of interleukin-1 blockade.Proton pump inhibitors protect mice from acute systemic inflammation and induce long-term cross-tolerance.T helper 1 immunity requires complement-driven NLRP3 inflammasome activity in CD4⁺ T cells.A novel "complement-metabolism-inflammasome axis" as a key regulator of immune cell effector function Redox stress unbalances the inflammatory cytokine network: role in autoinflammatory patients and healthy subjects.P2X7 receptor-pannexin 1 interaction mediates extracellular alpha-synuclein-induced ATP release in neuroblastoma SH-SY5Y cells.CD73-adenosine: a next-generation target in immuno-oncology.Genetic and Epigenetic Determinants in Autoinflammatory Diseases.Transplantation and Damage-Associated Molecular Patterns (DAMPs).The NLRP3 and Pyrin Inflammasomes: Implications in the Pathophysiology of Autoinflammatory Diseases.Cryopyrin-associated periodic syndrome in Australian children and adults: Epidemiological, clinical and treatment characteristics.Correlation of Secretory Activity of Neutrophils With Genotype in Patients With Familial Mediterranean Fever.Adenosine triphosphate inhibits melatonin synthesis in the rat pineal gland.Dysregulated IL-1β Secretion in Autoinflammatory Diseases: A Matter of Stress?Protection against lupus-like inflammatory disease is in the LAP of non-canonical autophagy.NLR in Human Diseases: Role and Laboratory Findings.TNF regulates transcription of NLRP3 inflammasome components and inflammatory molecules in cryopyrinopathies.Regulation of ATP utilization during metastatic cell migration by collagen architecture.Familial Mediterranean fever mutations are hypermorphic mutations that specifically decrease the activation threshold of the Pyrin inflammasome.Progressive waves of IL-1β release by primary human monocytes via sequential activation of vesicular and gasdermin D-mediated secretory pathways IL-6, IL-17 and Stat3 are required for auto-inflammatory syndrome development in mouse

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P2860

Cell stress increases ATP release in NLRP3 inflammasome-mediated autoinflammatory diseases, resulting in cytokine imbalance

http://www.wikidata.org/entity/Q35157045

description

2015 nî lūn-bûn

@nan

2015 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2015年の論文

@ja

2015年論文

@yue

2015年論文

@zh-hant

2015年論文

@zh-hk

2015年論文

@zh-mo

2015年論文

@zh-tw

2015年论文

@wuu

name

Cell stress increases ATP rele ...... esulting in cytokine imbalance

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Cell stress increases ATP rele ...... esulting in cytokine imbalance

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type

label

Cell stress increases ATP rele ...... esulting in cytokine imbalance

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Cell stress increases ATP rele ...... esulting in cytokine imbalance

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Cell stress increases ATP rele ...... esulting in cytokine imbalance

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Cell stress increases ATP rele ...... esulting in cytokine imbalance

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PNAS.1424741112

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Proceedings of the National Academy of Sciences of the United States of America

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Cell stress increases ATP rele ...... esulting in cytokine imbalance

@en

P2093

Alberto Martini

http://www.w3.org/2001/XMLSchema#string

Charles Anthony Dinarello

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Federica Penco

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Marco Gattorno

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Rosa Lavieri

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Sonia Carta

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P2860

Pannexin-1 mediates large pore formation and interleukin-1beta release by the ATP-gated P2X7 receptor

Interleukin-1 in the pathogenesis and treatment of inflammatory diseases

Cutting edge: direct interaction of TLR4 with NAD(P)H oxidase 4 isozyme is essential for lipopolysaccharide-induced production of reactive oxygen species and activation of NF-kappa B

ATP activates a reactive oxygen species-dependent oxidative stress response and secretion of proinflammatory cytokines in macrophages

Critical role for NALP3/CIAS1/Cryopyrin in innate and adaptive immunity through its regulation of caspase-1

The inflammasomes: guardians of the body

Src-dependent impairment of autophagy by oxidative stress in a mouse model of Duchenne muscular dystrophy

Altered redox state of monocytes from cryopyrin-associated periodic syndromes causes accelerated IL-1beta secretion.

Concerted action of wild-type and mutant TNF receptors enhances inflammation in TNF receptor 1-associated periodic fever syndrome.

Reactive oxygen species and redox compartmentalization

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2835-2840

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scholarly article

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10.1073/PNAS.1424741112

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9

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112

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Anna Rubartelli

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2015-02-17T00:00:00Z

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273501051

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25730877

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