Aspartyl-(asparaginyl) beta-hydroxylase, hypoxia-inducible factor-alpha and Notch cross-talk in regulating neuronal motility
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Effects of Hyperoxia on Oxygen-Related Inflammation with a Focus on ObesityComplex network analysis of CA3 transcriptome reveals pathogenic and compensatory pathways in refractory temporal lobe epilepsyMotor Function Deficits Following Chronic Prenatal Ethanol Exposure are Linked to Impairments in Insulin/IGF, Notch and Wnt Signaling in the Cerebellum.Dose effect of gestational ethanol exposure on placentation and fetal growthRegulation of Aspartyl-(Asparaginyl)-β-Hydroxylase Protein Expression and Function by Phosphorylation in Hepatocellular Carcinoma Cells.Ceramide inhibitor myriocin restores insulin/insulin growth factor signaling for liver remodeling in experimental alcohol-related steatohepatitis.Potential Role of Phosphorylation as a Regulator of Aspartyl-(asparaginyl)-β-hydroxylase: Relevance to Infiltrative Spread of Human Hepatocellular Carcinoma.Optimized Expression and Purification of Humbug in Pichia pastoris and Its Monoclonal Antibody Preparation.siRNA inhibition of aspartyl-asparaginyl β-hydroxylase expression impairs cell motility, Notch signaling, and fetal growth.Evidence that collaboration between HIF-1α and Notch-1 promotes neuronal cell death in ischemic stroke.Aspartate-β-hydroxylase (ASPH): A potential therapeutic target in human malignant gliomas.A Radiolabeled Fully Human Antibody to Human Aspartyl (Asparaginyl) β-Hydroxylase Is a Promising Agent for Imaging and Therapy of Metastatic Breast Cancer.Tobacco Smoke Exposure Impairs Brain Insulin/IGF Signaling: Potential Co-Factor Role in Neurodegeneration.Aspartate β-hydroxylase disrupts mitochondrial DNA stability and function in hepatocellular carcinoma.Potential Co-Factor Role of Tobacco Specific Nitrosamine Exposures in the Pathogenesis of Fetal Alcohol Spectrum DisorderAcetaldehyde-Mediated Neurotoxicity: Relevance to Fetal Alcohol Spectrum Disorders.Tobacco Nitrosamine Exposures Contribute to Fetal Alcohol Spectrum Disorder Associated Cerebellar Dysgenesis.Ethanol-Induced White Matter Atrophy Is Associated with Impaired Expression of Aspartyl-Asparaginyl-β-Hydroxylase (ASPH) and Notch Signaling in an Experimental Rat Model.Insulin-like growth factor-1 attenuates apoptosis and protects neurochemical phenotypes of dorsal root ganglion neurons with paclitaxel-induced neurotoxicity in vitro.Phosphorylation Modulates Aspartyl-(Asparaginyl)-β Hydroxylase Protein Expression, Catalytic Activity and Migration in Human Immature Neuronal Cerebellar Cells.Hydroxylase Activity of ASPH Promotes Hepatocellular Carcinoma Metastasis Through Epithelial-to-Mesenchymal Transition Pathway.
P2860
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P2860
Aspartyl-(asparaginyl) beta-hydroxylase, hypoxia-inducible factor-alpha and Notch cross-talk in regulating neuronal motility
description
2010 nî lūn-bûn
@nan
2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Aspartyl-(asparaginyl) beta-hy ...... n regulating neuronal motility
@ast
Aspartyl-(asparaginyl) beta-hy ...... n regulating neuronal motility
@en
type
label
Aspartyl-(asparaginyl) beta-hy ...... n regulating neuronal motility
@ast
Aspartyl-(asparaginyl) beta-hy ...... n regulating neuronal motility
@en
prefLabel
Aspartyl-(asparaginyl) beta-hy ...... n regulating neuronal motility
@ast
Aspartyl-(asparaginyl) beta-hy ...... n regulating neuronal motility
@en
P2093
P2860
P356
P1476
Aspartyl-(asparaginyl) beta-hy ...... n regulating neuronal motility
@en
P2093
Fusun Gundogan
Jack R Wands
Margot Lawton
Suzanne M de la Monte
P2860
P304
P356
10.4161/OXIM.3.5.13296
P577
2010-09-01T00:00:00Z