EAE: a model for immune intervention with synthetic peptides. - Wikidata - wikimedia - TriplyDB

EAE: a model for immune intervention with synthetic peptides.

EAE: a model for immune intervention with synthetic peptides.

http://www.wikidata.org/entity/Q35173105

about

Peripherally Induced Regulatory T Cells: Recruited Protectors of the Central Nervous System against Autoimmune Neuroinflammation.Peripherally Induced Tolerance Depends on Peripheral Regulatory T Cells That Require Hopx To Inhibit Intrinsic IL-2 Expression.

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EAE: a model for immune intervention with synthetic peptides.

http://www.wikidata.org/entity/Q35173105

description

1992 nî lūn-bûn

@nan

1992 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

1992 թվականի հունվարին հրատարակված գիտական հոդված

@hy

1992年の論文

@ja

1992年論文

@yue

1992年論文

@zh-hant

1992年論文

@zh-hk

1992年論文

@zh-mo

1992年論文

@zh-tw

1992年论文

@wuu

name

EAE: a model for immune intervention with synthetic peptides.

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EAE: a model for immune intervention with synthetic peptides.

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type

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EAE: a model for immune intervention with synthetic peptides.

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EAE: a model for immune intervention with synthetic peptides.

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prefLabel

EAE: a model for immune intervention with synthetic peptides.

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EAE: a model for immune intervention with synthetic peptides.

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08830189209061792

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International Reviews of Immunology

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EAE: a model for immune intervention with synthetic peptides.

@en

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Gautam AM

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McDevitt HO

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Pearson C

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Smilek DE

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Steinman L

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P2860

Involvement of distinct murine T-cell receptors in the autoimmune encephalitogenic response to nested epitopes of myelin basic protein.

Specific inhibition of the T-cell response to myelin basic protein by the synthetic copolymer Cop 1

Prevention of experimental encephalomyelitis with peptides that block interaction of T cells with major histocompatibility complex proteins.

Regulation of the immune response to peptide antigens: differential induction of immediate-type hypersensitivity and T cell proliferation due to changes in either peptide structure or major histocompatibility complex haplotype

Analysis of T cell receptor beta chains in Lewis rats with experimental allergic encephalomyelitis: conserved complementarity determining region 3

An antibody to lymphotoxin and tumor necrosis factor prevents transfer of experimental allergic encephalomyelitis.

Two minor determinants of myelin basic protein induce experimental allergic encephalomyelitis in SJL/J mice.

Multiple discrete encephalitogenic epitopes of the autoantigen myelin basic protein include a determinant for I-E class II-restricted T cells

Protection from experimental allergic encephalomyelitis conferred by a monoclonal antibody directed against a shared idiotype on rat T cell receptors specific for myelin basic protein.

In vivo effects of antibodies to immune response gene products: prevention of experimental allergic encephalitis.

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223-230

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scholarly article

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10.3109/08830189209061792

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3

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9

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1992-01-01T00:00:00Z

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1285062

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