Ca²⁺ entry via Trpm2 is essential for cardiac myocyte bioenergetics maintenance. - Wikidata - wikimedia - TriplyDB

Ca²⁺ entry via Trpm2 is essential for cardiac myocyte bioenergetics maintenance.

Ca²⁺ entry via Trpm2 is essential for cardiac myocyte bioenergetics maintenance.

http://www.wikidata.org/entity/Q35178670

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TRP ion channels in thermosensation, thermoregulation and metabolism Detrimental or beneficial: the role of TRPM2 in ischemia/reperfusion injury Targeting TRPM2 in ROS-Coupled Diseases Mitochondrial calcium overload is a key determinant in heart failure.Meeting report from the 2nd International Symposium on New Frontiers in Cardiovascular Research. Protecting the cardiovascular system from ischemia: between bench and bedside Depletion of the Human Ion Channel TRPM2 in Neuroblastoma Demonstrates Its Key Role in Cell Survival through Modulation of Mitochondrial Reactive Oxygen Species and Bioenergetics.Altered Mitochondrial Metabolism and Mechanosensation in the Failing Heart: Focus on Intracellular Calcium Signaling.TRPM2 protects against tissue damage following oxidative stress and ischaemia-reperfusion.Transient Receptor Potential-Melastatin Channel Family Member 2: Friend or Foe Mitochondrial Mechanosensor Microdomains in Cardiovascular Disorders.Methylene Blue Counteracts H2S-Induced Cardiac Ion Channel Dysfunction and ATP Reduction.

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Ca²⁺ entry via Trpm2 is essential for cardiac myocyte bioenergetics maintenance.

http://www.wikidata.org/entity/Q35178670

description

2015 nî lūn-bûn

@nan

2015 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի հունվարին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

Ca²⁺ entry via Trpm2 is essential for cardiac myocyte bioenergetics maintenance.

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Ca²⁺ entry via Trpm2 is essential for cardiac myocyte bioenergetics maintenance.

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Ca²⁺ entry via Trpm2 is essential for cardiac myocyte bioenergetics maintenance.

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Ca²⁺ entry via Trpm2 is essential for cardiac myocyte bioenergetics maintenance.

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Ca²⁺ entry via Trpm2 is essential for cardiac myocyte bioenergetics maintenance.

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Ca²⁺ entry via Trpm2 is essential for cardiac myocyte bioenergetics maintenance.

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AJPHEART.00720.2014

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American Journal of Physiology - Heart and Circulatory Physiology

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Ca²⁺ entry via Trpm2 is essential for cardiac myocyte bioenergetics maintenance.

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Arthur M Feldman

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Barbara A Miller

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Iwona Hirschler-Laszkiewicz

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Jianliang Song

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Joseph Y Cheung

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JuFang Wang

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Muniswamy Madesh

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Nicholas E Hoffman

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Sudasan Rajan

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Walter J Koch

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P2860

Subunit composition of mammalian transient receptor potential channels in living cells

Functional characterization of homo- and heteromeric channel kinases TRPM6 and TRPM7

LTRPC2 Ca2+-permeable channel activated by changes in redox status confers susceptibility to cell death

Transient receptor potential cation channels in disease

Mitochondrial autophagy is an HIF-1-dependent adaptive metabolic response to hypoxia

TRP channels as cellular sensors

Mitofusin 2 tethers endoplasmic reticulum to mitochondria

Regulation of in vivo cardiac contractility by phospholemman: role of Na+/Ca2+ exchange.

Regulation of cardiac myocyte contractility by phospholemman: Na+/Ca2+ exchange versus Na+ -K+ -ATPase

Altered contractility and [Ca2+]i homeostasis in phospholemman-deficient murine myocytes: role of Na+/Ca2+ exchange

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H637-50

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10.1152/AJPHEART.00720.2014

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6

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308

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Santhanam Shanmughapriya

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2015-01-09T00:00:00Z

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270764890

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25576627

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