The vitronectin-binding function of PAI-1 exacerbates lung fibrosis in mice.
about
Always cleave up your mess: targeting collagen degradation to treat tissue fibrosisSoy biodiesel and petrodiesel emissions differ in size, chemical composition and stimulation of inflammatory responses in cells and animalsPeriostin promotes fibrosis and predicts progression in patients with idiopathic pulmonary fibrosis.Plasminogen activator-1 overexpression decreases experimental postthrombotic vein wall fibrosis by a non-vitronectin-dependent mechanism.Vitronectin-binding PAI-1 protects against the development of cardiac fibrosis through interaction with fibroblastsInhibition of myocardin-related transcription factor/serum response factor signaling decreases lung fibrosis and promotes mesenchymal cell apoptosis.Plasminogen activator inhibitor 1, fibroblast apoptosis resistance, and aging-related susceptibility to lung fibrosisInhibition of Plasminogen Activator Inhibitor-1 Attenuates Transforming Growth Factor-β-Dependent Epithelial Mesenchymal Transition and Differentiation of Fibroblasts to Myofibroblasts.HSP47 regulates ECM accumulation in renal proximal tubular cells induced by TGF-β1 through ERK1/2 and JNK MAPK pathways.PAI-1 promotes the accumulation of exudate macrophages and worsens pulmonary fibrosis following type II alveolar epithelial cell injury.Plasminogen Activator Inhibitor Type-1 as a Regulator of Fibrosis.The vitronectin RGD motif regulates TGF-β-induced alveolar epithelial cell apoptosis.Plasminogen activator inhibitor-1 regulates the vascular expression of vitronectin.Signalling pathways regulating muscle mass in ageing skeletal muscle: the role of the IGF1-Akt-mTOR-FoxO pathway.Lung epithelial cell focal adhesion kinase signaling inhibits lung injury and fibrosis.Fibrinolytic system related to pulmonary arterial pressure and lung function of patients with idiopathic pulmonary fibrosis.The positive role of vitronectin in radiation induced lung toxicity: the in vitro and in vivo mechanism study.Serpine1 Knockdown Enhances MMP Activity after Flexor Tendon Injury in Mice: Implications for Adhesions Therapy.
P2860
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P2860
The vitronectin-binding function of PAI-1 exacerbates lung fibrosis in mice.
description
2011 nî lūn-bûn
@nan
2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
The vitronectin-binding function of PAI-1 exacerbates lung fibrosis in mice.
@ast
The vitronectin-binding function of PAI-1 exacerbates lung fibrosis in mice.
@en
type
label
The vitronectin-binding function of PAI-1 exacerbates lung fibrosis in mice.
@ast
The vitronectin-binding function of PAI-1 exacerbates lung fibrosis in mice.
@en
prefLabel
The vitronectin-binding function of PAI-1 exacerbates lung fibrosis in mice.
@ast
The vitronectin-binding function of PAI-1 exacerbates lung fibrosis in mice.
@en
P2093
P2860
P1433
P1476
The vitronectin-binding function of PAI-1 exacerbates lung fibrosis in mice.
@en
P2093
Andrew K Cunningham
Anthony J Courey
Daniel A Lawrence
Jeffrey C Horowitz
Kevin K Kim
Margaret L Novak
Mark Warnock
Monica P Goldklang
Natalya Subbotina
P2860
P304
P356
10.1182/BLOOD-2010-12-324574
P407
P577
2011-07-06T00:00:00Z