The role of the tripartite glutamatergic synapse in the pathophysiology of Alzheimer's disease. - Wikidata - wikimedia - TriplyDB

The role of the tripartite glutamatergic synapse in the pathophysiology of Alzheimer's disease.

The role of the tripartite glutamatergic synapse in the pathophysiology of Alzheimer's disease.

http://www.wikidata.org/entity/Q35195234

about

Danggui-Shaoyao-San: New Hope for Alzheimer's Disease Dysfunction of NMDA receptors in Alzheimer's disease Knocking down of heat-shock protein 27 directs differentiation of functional glutamatergic neurons from placenta-derived multipotent cells.Role of Pro-Inflammatory Cytokines and Vitamin D in Probable Alzheimer's Disease with Depression.Signaling pathways regulating neuron-glia interaction and their implications in Alzheimer's disease.The Markers of Glutamate Metabolism in Peripheral Blood Mononuclear Cells and Neurological Complications in Lung Cancer Patients.Gartanin Protects Neurons against Glutamate-Induced Cell Death in HT22 Cells: Independence of Nrf-2 but Involvement of HO-1 and AMPK.Neuroprotective Effects of Etidronate and 2,3,3-Trisphosphonate Against Glutamate-Induced Toxicity in PC12 Cells.Genetic Studies on the Tripartite Glutamate Synapse in the Pathophysiology and Therapeutics of Mood Disorders.Stress-Induced Synaptic Dysfunction and Neurotransmitter Release in Alzheimer's Disease: Can Neurotransmitters and Neuromodulators be Potential Therapeutic Targets?Mechanistic comparison of current pharmacological treatments and novel phytochemicals to target amyloid peptides in Alzheimer's and neurodegenerative diseases.Discrete pools of oligomeric amyloid-β track with spatial learning deficits in a mouse model of Alzheimer amyloidosis.Non-Fibrillar Oligomeric Amyloid-β within Synapses.Troxerutin protects hippocampal neurons against amyloid beta-induced oxidative stress and apoptosis.SPARCL1 Accelerates Symptom Onset in Alzheimer's Disease and Influences Brain Structure and Function During Aging.Diversity of astroglial responses across human neurodegenerative disorders and brain aging.Neuron-astrocyte signaling is preserved in the aging brain.Peripheral Interventions Enhancing Brain Glutamate Homeostasis Relieve Amyloid β- and TNFα- Mediated Synaptic Plasticity Disruption in the Rat Hippocampus.Potential and Challenges for the Clinical Use of d-Serine As a Cognitive Enhancer.Is riluzole a new drug for Alzheimer's disease?Reactive Astrocytes as Drug Target in Alzheimer's Disease.

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P2860

The role of the tripartite glutamatergic synapse in the pathophysiology of Alzheimer's disease.

http://www.wikidata.org/entity/Q35195234

description

2015 nî lūn-bûn

@nan

2015 թուականի Մարտին հրատարակուած գիտական յօդուած

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2015 թվականի մարտին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

@zh-hk

2015年論文

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2015年論文

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2015年论文

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name

The role of the tripartite glu ...... iology of Alzheimer's disease.

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The role of the tripartite glu ...... iology of Alzheimer's disease.

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The role of the tripartite glu ...... iology of Alzheimer's disease.

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The role of the tripartite glu ...... iology of Alzheimer's disease.

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The role of the tripartite glu ...... iology of Alzheimer's disease.

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Aging and Disease

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The role of the tripartite glu ...... iology of Alzheimer's disease.

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Carolyn C Rudy

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Daniel S Weitzner

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Holly C Hunsberger

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Miranda N Reed

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P2860

A Oligomer-Induced Aberrations in Synapse Composition, Shape, and Density Provide a Molecular Basis for Loss of Connectivity in Alzheimer's Disease

A synaptic model of memory: long-term potentiation in the hippocampus

The synaptic vesicle cycle

Characterisation of the human NMDA receptor subunit NR3A glycine binding site

Excitatory amino acid transporter 5, a retinal glutamate transporter coupled to a chloride conductance

Metabolism of ketonic acids in animal tissues

Long-lasting potentiation of synaptic transmission in the dentate area of the anaesthetized rabbit following stimulation of the perforant path

Long-lasting potentiation of synaptic transmission in the dentate area of the unanaestetized rabbit following stimulation of the perforant path

Memantine preferentially blocks extrasynaptic over synaptic NMDA receptor currents in hippocampal autapses

Amyloid-beta protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory

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10.14336/AD.2014.0423

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2015-03-10T00:00:00Z

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Alzheimer's disease

pathophysiology

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