Purkinje cell vulnerability and autism: a possible etiological connection.
about
The neuropathology of autism: where do we stand?Statistical distribution of blood serotonin as a predictor of early autistic brain abnormalities.Inflammatory cytokines: potential biomarkers of immunologic dysfunction in autism spectrum disordersEvidence of toxicity, oxidative stress, and neuronal insult in autismPre-clinical models of neurodevelopmental disorders: focus on the cerebellumActivation of the maternal immune system alters cerebellar development in the offspring.Loss of the tuberous sclerosis complex protein tuberin causes Purkinje cell degenerationAltered posterior cingulate cortical cyctoarchitecture, but normal density of neurons and interneurons in the posterior cingulate cortex and fusiform gyrus in autism.Anatomical likelihood estimation meta-analysis of grey and white matter anomalies in autism spectrum disorders.Early pharmacological treatment of autism: a rationale for developmental treatment.Brain region-specific deficit in mitochondrial electron transport chain complexes in children with autism.Auditory hypersensitivity in the autistic spectrum disorder.Is neonatal jaundice associated with Autism Spectrum Disorders: a systematic review.Exploring the multifactorial nature of autism through computational systems biology: calcium and the Rho GTPase RAC1 under the spotlight.Injury of the developing cerebellum: a brief review of the effects of endotoxin and asphyxial challenges in the late gestation sheep fetus.Bergmann Glia, Long-Term Depression, and Autism Spectrum Disorder.An MRI and proton spectroscopy study of the thalamus in children with autism.Assessment and comparison of neural morphology through metrical feature extraction and analysis in neuron and neuron-glia cultures.Further evidence that infantile autism is a chronic psychosis distinguished by a deficient delayed response function affecting the connections between hippocampus and singulum in its center, the SMA and the inhibitory Purkinje cells in cerebellum.Heterogeneous dysregulation of microRNAs across the autism spectrum.Calretinin interneuron density in the caudate nucleus is lower in autism spectrum disorder.Expression of mutant DISC1 in Purkinje cells increases their spontaneous activity and impairs cognitive and social behaviors in mice.Stem cell fusion in the brain.A novel pathogenic mechanism for cerebellar lesions produced by Solanum bonariense in cattle.A diffusion tensor imaging study of the cerebellar pathways in children with autism spectrum disorder.Effects of rat prenatal exposure to valproic acid on behaviour and neuro-anatomy.Brain region-specific glutathione redox imbalance in autism.Analysis of toddlers' gait after six months of independent walking to identify autism: a preliminary study.
P2860
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P2860
Purkinje cell vulnerability and autism: a possible etiological connection.
description
2003 nî lūn-bûn
@nan
2003 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Purkinje cell vulnerability and autism: a possible etiological connection.
@ast
Purkinje cell vulnerability and autism: a possible etiological connection.
@en
type
label
Purkinje cell vulnerability and autism: a possible etiological connection.
@ast
Purkinje cell vulnerability and autism: a possible etiological connection.
@en
prefLabel
Purkinje cell vulnerability and autism: a possible etiological connection.
@ast
Purkinje cell vulnerability and autism: a possible etiological connection.
@en
P1476
Purkinje cell vulnerability and autism: a possible etiological connection.
@en
P2093
Janet Kinnear Kern
P304
P356
10.1016/S0387-7604(03)00056-1
P577
2003-09-01T00:00:00Z