Bone morphogenetic protein inhibition promotes neurological recovery after intraventricular hemorrhage. - Wikidata - wikimedia - TriplyDB

Bone morphogenetic protein inhibition promotes neurological recovery after intraventricular hemorrhage.

Bone morphogenetic protein inhibition promotes neurological recovery after intraventricular hemorrhage.

http://www.wikidata.org/entity/Q35196642

about

Oligodendrogenesis in the normal and pathological central nervous system Bone Morphogenetic Protein 4 Signalling in Neural Stem and Progenitor Cells during Development and after Injury Postnatal glucocorticoid-induced hypomyelination, gliosis, and neurologic deficits are dose-dependent, preparation-specific, and reversible AMPA-Kainate Receptor Inhibition Promotes Neurologic Recovery in Premature Rabbits with Intraventricular Hemorrhage Delayed myelination in an intrauterine growth retardation model is mediated by oxidative stress upregulating bone morphogenetic protein 4 Comparison of the global gene expression of choroid plexus and meninges and associated vasculature under control conditions and after pronounced hyperthermia or amphetamine toxicity Intraventricular hemorrhage induces deposition of proteoglycans in premature rabbits, but their in vivo degradation with chondroitinase does not restore myelination, ventricle size and neurological recovery Mesenchymal stem cells transplantation for neuroprotection in preterm infants with severe intraventricular hemorrhage.Migration of oligodendrocyte progenitor cells is controlled by transforming growth factor β family proteins during corticogenesis.Investigation of sequential growth factor delivery during cuprizone challenge in mice aimed to enhance oligodendrogliogenesis and myelin repair.Neuroprotection by rAAV-mediated gene transfer of bone morphogenic protein 7 Hyaluronidase and Hyaluronan Oligosaccharides Promote Neurological Recovery after Intraventricular Hemorrhage.Regulation of endogenous neural stem/progenitor cells for neural repair-factors that promote neurogenesis and gliogenesis in the normal and damaged brain Older Age Results in Differential Gene Expression after Mild Traumatic Brain Injury and Is Linked to Imaging Differences at Acute Follow-up.Mechanisms of hydrocephalus after neonatal and adult intraventricular hemorrhage.Treatment with thyroxine restores myelination and clinical recovery after intraventricular hemorrhage.Nanomedicine in cerebral palsy.Knockdown of both p120 catenin and Kaiso promotes expansion of human corneal endothelial monolayers via RhoA-ROCK-noncanonical BMP-NFκB pathway Signalling pathways that inhibit the capacity of precursor cells for myelin repair.Pharmacological approaches to intervention in hypomyelinating and demyelinating white matter pathology.Extracellular cues influencing oligodendrocyte differentiation and (re)myelination Mesenchymal Stem Cells: The Magic Cure for Intraventricular Hemorrhage?Epidermal growth factor preserves myelin and promotes astrogliosis after intraventricular hemorrhage.Olig1 is required for noggin-induced neonatal myelin repair.Disruption of Interneuron Neurogenesis in Premature Newborns and Reversal with Estrogen Treatment.From the Cover: Arsenic Induces Hippocampal Neuronal Apoptosis and Cognitive Impairments via an Up-Regulated BMP2/Smad-Dependent Reduced BDNF/TrkB Signaling in Rats.Distribution of the creatine transporter throughout the human brain reveals a spectrum of creatine transporter immunoreactivity.Strategies for working with a preterm rabbit model of glycerol-induced intraventricular hemorrhage: strengths and limitations.Intrathecal Noggin administration in rats temporally ameliorates mechanical allodynia induced by a chronic constriction injury.The effect of Noggin supplementation in Matrigel nanofiber-based cell culture system for derivation of neural-like cells from human endometrial-derived stromal cells.The Potentials and Caveats of Mesenchymal Stromal Cell-Based Therapies in the Preterm Infant.Injury-induced perivascular niche supports alternative differentiation of adult rodent CNS progenitor cells

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Bone morphogenetic protein inhibition promotes neurological recovery after intraventricular hemorrhage.

http://www.wikidata.org/entity/Q35196642

description

2011 nî lūn-bûn

@nan

2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

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2011 թվականի օգոստոսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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Bone morphogenetic protein inh ...... r intraventricular hemorrhage.

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Bone morphogenetic protein inh ...... r intraventricular hemorrhage.

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Bone morphogenetic protein inh ...... r intraventricular hemorrhage.

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JNEUROSCI.0013-11.2011

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Journal of Neuroscience

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Bone morphogenetic protein inh ...... r intraventricular hemorrhage.

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Anna Csiszar

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Caroline Chua

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Furong Hu

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Govindaiah Vinukonda

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Joshua C Brumberg

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Krishna Dummula

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Peter Mouton

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Philip Chu

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Praveen Ballabh

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Rashmi Bansal

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P2860

Regeneration beyond the glial scar

Expression pattern of the transcription factor Olig2 in response to brain injuries: implications for neuronal repair.

Interactions between ID and OLIG proteins mediate the inhibitory effects of BMP4 on oligodendroglial differentiation

Versican is upregulated in CNS injury and is a product of oligodendrocyte lineage cells

Bone morphogenetic proteins promote gliosis in demyelinating spinal cord lesions

A potential role for bone morphogenetic protein signalling in glial cell fate determination following adult central nervous system injury in vivo

Common developmental requirement for Olig function indicates a motor neuron/oligodendrocyte connection

Applications of small molecule BMP inhibitors in physiology and disease.

Chordin-induced lineage plasticity of adult SVZ neuroblasts after demyelination.

The oligodendrocyte and its many cellular processes.

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12068-12082

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10.1523/JNEUROSCI.0013-11.2011

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21865450

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