Inhibition of keratinocyte differentiation by the synergistic effect of IL-17A, IL-22, IL-1α, TNFα and oncostatin M. - Wikidata - wikimedia - TriplyDB

Inhibition of keratinocyte differentiation by the synergistic effect of IL-17A, IL-22, IL-1α, TNFα and oncostatin M.

Inhibition of keratinocyte differentiation by the synergistic effect of IL-17A, IL-22, IL-1α, TNFα and oncostatin M.

http://www.wikidata.org/entity/Q35204519

about

The Changing Landscape of Alopecia Areata: The Therapeutic Paradigm.IL22/IL-22R pathway induces cell survival in human glioblastoma cells.Interleukin-22: immunobiology and pathology.Langerhans Cells Facilitate UVB-Induced Epidermal Carcinogenesis.Asymmetric stem-cell division ensures sustained keratinocyte hyperproliferation in psoriatic skin lesions Antibody blockade of IL-17 family cytokines in immunity to acute murine oral mucosal candidiasis.Distinct expression of interleukin (IL)-36α, β and γ, their antagonist IL-36Ra and IL-38 in psoriasis, rheumatoid arthritis and Crohn's disease.A novel therapeutic paradigm for patients with extensive alopecia areata.Interleukin 17 is a chief orchestrator of immunity.Interleukin-17A-induced production of acute serum amyloid A by keratinocytes contributes to psoriasis pathogenesis.Development of a new model of reconstituted mouse epidermis and characterization of its response to proinflammatory cytokines.Inherent differences in keratinocyte function in hidradenitis suppurativa: Evidence for the role of IL-22 in disease pathogenesis.PFKFB3, a Direct Target of p63, Is Required for Proliferation and Inhibits Differentiation in Epidermal Keratinocytes.IMQ-induced skin inflammation in mice is dependent on IL-1R1 and MyD88 signaling but independent of the NLRP3 inflammasome.Oncostatin M overexpression induces skin inflammation but is not required in the mouse model of imiquimod-induced psoriasis-like inflammation.Topical application of a linoleic acid-ceramide containing moisturizer exhibit therapeutic and preventive benefits for psoriasis vulgaris: a randomized controlled trial.Role of Fibroblast Growth Factor Receptor 2b in the Cross Talk between Autophagy and Differentiation: Involvement of Jun N-Terminal Protein Kinase Signaling.IL-38 has an anti-inflammatory action in psoriasis and its expression correlates with disease severity and therapeutic response to anti-IL-17A treatment

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Inhibition of keratinocyte differentiation by the synergistic effect of IL-17A, IL-22, IL-1α, TNFα and oncostatin M.

http://www.wikidata.org/entity/Q35204519

description

2014 nî lūn-bûn

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2014 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2014 թվականի հուլիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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Inhibition of keratinocyte dif ...... IL-1α, TNFα and oncostatin M.

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Inhibition of keratinocyte dif ...... IL-1α, TNFα and oncostatin M.

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Inhibition of keratinocyte dif ...... IL-1α, TNFα and oncostatin M.

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JOURNAL.PONE.0101937

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Inhibition of keratinocyte dif ...... IL-1α, TNFα and oncostatin M.

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Christine Barrault

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François-Xavier Bernard

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Gérard Guillet

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Hanitriniaina Rabeony

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Isabelle Petit-Paris

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Jean-Claude Lecron

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Jean-François Jegou

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Julien Garnier

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Karline Guilloteau

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Nathalie Pedretti

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P2860

Interleukin (IL)-22 and IL-17 are coexpressed by Th17 cells and cooperatively enhance expression of antimicrobial peptides

Accurate normalization of real-time quantitative RT-PCR data by geometric averaging of multiple internal control genes

Development, cytokine profile and function of human interleukin 17-producing helper T cells

Interleukin-22, a T(H)17 cytokine, mediates IL-23-induced dermal inflammation and acanthosis

Inflammatory skin disease in transgenic mice that express high levels of interleukin 1 alpha in basal epidermis

Genome-wide expression profiling of five mouse models identifies similarities and differences with human psoriasis.

Interleukin-17 and interferon-gamma synergize in the enhancement of proinflammatory cytokine production by human keratinocytes.

Epidermal barrier formation and recovery in skin disorders.

IL-23 stimulates epidermal hyperplasia via TNF and IL-20R2-dependent mechanisms with implications for psoriasis pathogenesis

Recent advances in the IL-17 cytokine family.

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e101937

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scholarly article

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10.1371/JOURNAL.PONE.0101937

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4092099

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