Analysis of a Caenorhabditis elegans Twist homolog identifies conserved and divergent aspects of mesodermal patterning.
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A multiparameter network reveals extensive divergence between C. elegans bHLH transcription factorsPhylogenetic analysis of the human basic helix-loop-helix proteinsA Genetic-Pathophysiological Framework for CraniosynostosisSomatic muscle specification during embryonic and post-embryonic development in the nematode C. elegansdbl-1/TGF-β and daf-12/NHR Signaling Mediate Cell-Nonautonomous Effects of daf-16/FOXO on Starvation-Induced Developmental ArrestThe HLH-6 transcription factor regulates C. elegans pharyngeal gland development and functionThe Caenorhabditis elegans T-box factor MLS-1 requires Groucho co-repressor interaction for uterine muscle specificationA survey of TWIST for mutations in craniosynostosis reveals a variable length polyglycine tract in asymptomatic individualsThe basic helix-loop-helix factor, HAND2, functions as a transcriptional activator by binding to E-boxes as a heterodimerThe T-box factor MLS-1 acts as a molecular switch during specification of nonstriated muscle in C. elegansMultiple Wnts redundantly control polarity orientation in Caenorhabditis elegans epithelial stem cellsMechanism of transcriptional activation by the proto-oncogene Twist1The C. elegans SoxC protein SEM-2 opposes differentiation factors to promote a proliferative blast cell fate in the postembryonic mesoderm.Myrf ER-Bound Transcription Factors Drive C. elegans Synaptic Plasticity via Cleavage-Dependent Nuclear Translocation.Lipoprotein receptors and a disabled family cytoplasmic adaptor protein regulate EGL-17/FGF export in C. elegans.Localized TWIST1 and TWIST2 basic domain substitutions cause four distinct human diseases that can be modeled in Caenorhabditis elegansTBC-2 is required for embryonic yolk protein storage and larval survival during L1 diapause in Caenorhabditis elegans.Dopamine signaling in C. elegans is mediated in part by HLH-17-dependent regulation of extracellular dopamine levels.Mutations in the human TWIST gene.Isolation and culture of larval cells from C. elegans.Distinct Caenorhabditis elegans HLH-8/twist-containing dimers function in the mesoderm.C. elegans twist gene expression in differentiated cell types is controlled by autoregulation through intron elementsLIN-12/Notch activation leads to microRNA-mediated down-regulation of Vav in C. elegans.A biochemist's guide to Caenorhabditis elegansCaenorhabditis elegans Galphaq regulates egg-laying behavior via a PLCbeta-independent and serotonin-dependent signaling pathway and likely functions both in the nervous system and in muscle.A novel mitochondrial outer membrane protein, MOMA-1, that affects cristae morphology in Caenorhabditis elegansEstablishment of a tissue-specific RNAi system in C. elegans.The GEX-2 and GEX-3 proteins are required for tissue morphogenesis and cell migrations in C. elegansThe basic helix-loop-helix protein family: comparative genomics and phylogenetic analysis.Neuroblast migration along the anteroposterior axis of C. elegans is controlled by opposing gradients of Wnts and a secreted Frizzled-related protein.Mesodermal gene expression during the embryonic and larval development of the articulate brachiopod Terebratalia transversa.Ce-emerin and LEM-2: essential roles in Caenorhabditis elegans development, muscle function, and mitosis.Dimerization-driven degradation of C. elegans and human E proteins.Transcription factor redundancy and tissue-specific regulation: evidence from functional and physical network connectivity.Nuclear hormone receptor CHR3 is a critical regulator of all four larval molts of the nematode Caenorhabditis elegansDorsoventral patterning of the C. elegans postembryonic mesoderm requires both LIN-12/Notch and TGFbeta signalingContributions of mRNA abundance, ribosome loading, and post- or peri-translational effects to temporal repression of C. elegans heterochronic miRNA targets.Analysis of cell migration using Caenorhabditis elegans as a model system.LIN-12/Notch signaling instructs postsynaptic muscle arm development by regulating UNC-40/DCC and MADD-2 in Caenorhabditis elegans.Mesodermal expression of the C. elegans HMX homolog mls-2 requires the PBC homolog CEH-20
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P2860
Analysis of a Caenorhabditis elegans Twist homolog identifies conserved and divergent aspects of mesodermal patterning.
description
1998 nî lūn-bûn
@nan
1998 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Analysis of a Caenorhabditis e ...... ects of mesodermal patterning.
@ast
Analysis of a Caenorhabditis e ...... ects of mesodermal patterning.
@en
type
label
Analysis of a Caenorhabditis e ...... ects of mesodermal patterning.
@ast
Analysis of a Caenorhabditis e ...... ects of mesodermal patterning.
@en
prefLabel
Analysis of a Caenorhabditis e ...... ects of mesodermal patterning.
@ast
Analysis of a Caenorhabditis e ...... ects of mesodermal patterning.
@en
P2093
P2860
P356
P1433
P1476
Analysis of a Caenorhabditis e ...... ects of mesodermal patterning.
@en
P2093
P2860
P304
P356
10.1101/GAD.12.16.2623
P577
1998-08-01T00:00:00Z