Aberrant contraction of antigen-specific CD4 T cells after infection in the absence of gamma interferon or its receptor.
about
Salmonella as a model for non-cognate Th1 cell stimulationDifferential role of gamma interferon in inhibiting pulmonary eosinophilia and exacerbating systemic disease in fusion protein-immunized mice undergoing challenge infection with respiratory syncytial virusMice with a selective impairment of IFN-gamma signaling in macrophage lineage cells demonstrate the critical role of IFN-gamma-activated macrophages for the control of protozoan parasitic infections in vivoRole of PD-1 in regulating acute infections.CD4 memory T cells divide poorly in response to antigen because of their cytokine profileInduction and function of virus-specific CD4+ T cell responses.Surviving the crash: transitioning from effector to memory CD8+ T cell.Regulation of neutrophils by interferon-γ limits lung inflammation during tuberculosis infection.Anti-gamma interferon antibodies enhance the immunogenicity of recombinant adenovirus vectors.Perforin plays an unexpected role in regulating T-cell contraction during prolonged Listeria monocytogenes infection.IFN-gamma acts directly on activated CD4+ T cells during mycobacterial infection to promote apoptosis by inducing components of the intracellular apoptosis machinery and by inducing extracellular proapoptotic signals.T-cell death and cancer immune tolerance.Deviation from a strong Th1-dominated to a modest Th17-dominated CD4 T cell response in the absence of IL-12p40 and type I IFNs sustains protective CD8 T cells.Interleukin-18-related genes are induced during the contraction phase but do not play major roles in regulating the dynamics or function of the T-cell response to Listeria monocytogenes infectionIL-7 receptor expression provides the potential for long-term survival of both CD62Lhigh central memory T cells and Th1 effector cells during Leishmania major infection.Gene expression changes in the mesenteric lymph nodes of rats after oral peanut extract exposure.Regulation of CD4+ T-cell contraction during pathogen challengeTNF signalling drives expansion of bone marrow CD4+ T cells responsible for HSC exhaustion in experimental visceral leishmaniasis.Influence of Inflammation in the Process of T Lymphocyte Differentiation: Proliferative, Metabolic, and Oxidative Changes.
P2860
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P2860
Aberrant contraction of antigen-specific CD4 T cells after infection in the absence of gamma interferon or its receptor.
description
2006 nî lūn-bûn
@nan
2006 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Aberrant contraction of antige ...... ma interferon or its receptor.
@ast
Aberrant contraction of antige ...... ma interferon or its receptor.
@en
type
label
Aberrant contraction of antige ...... ma interferon or its receptor.
@ast
Aberrant contraction of antige ...... ma interferon or its receptor.
@en
prefLabel
Aberrant contraction of antige ...... ma interferon or its receptor.
@ast
Aberrant contraction of antige ...... ma interferon or its receptor.
@en
P2860
P356
P1476
Aberrant contraction of antige ...... mma interferon or its receptor
@en
P2093
Jodie S Haring
P2860
P304
P356
10.1128/IAI.00847-06
P407
P50
P577
2006-09-11T00:00:00Z