Thioredoxin-2 inhibits mitochondrial reactive oxygen species generation and apoptosis stress kinase-1 activity to maintain cardiac function. - Wikidata - wikimedia - TriplyDB

Thioredoxin-2 inhibits mitochondrial reactive oxygen species generation and apoptosis stress kinase-1 activity to maintain cardiac function.

Thioredoxin-2 inhibits mitochondrial reactive oxygen species generation and apoptosis stress kinase-1 activity to maintain cardiac function.

http://www.wikidata.org/entity/Q35217815

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Oxidative Stress after Surgery on the Immature Heart Mitochondrial Redox Signaling and Tumor Progression Mitochondrial dysfunction in cardiac aging Paradoxical Roles of Antioxidant Enzymes: Basic Mechanisms and Health Implications Reactive Oxygen Species/Nitric Oxide Mediated Inter-Organ Communication in Skeletal Muscle Wasting Diseases.The Role of NOX4 and TRX2 in Angiogenesis and Their Potential Cross-Talk.Modulation of signaling mechanisms in the heart by thioredoxin 1.Role of Epithelial-Mesenchyme Transition in Chlamydia Pathogenesis.Identification of potential genes for human ischemic cardiomyopathy based on RNA-Seq data.Detoxification of Mitochondrial Oxidants and Apoptotic Signaling Are Facilitated by Thioredoxin-2 and Peroxiredoxin-3 during Hyperoxic Injury.Thidoredxin-2 overexpression fails to rescue chronic high calorie diet induced hippocampal dysfunction.ROS activates JNK-mediated autophagy to counteract apoptosis in mouse mesenchymal stem cells in vitro.Translocase of Inner Membrane 50 Functions as a Novel Protective Regulator of Pathological Cardiac Hypertrophy.Thioredoxin-2 protects against oxygen-glucose deprivation/reperfusion injury by inhibiting autophagy and apoptosis in H9c2 cardiomyocytes.Mitochondrial ROS control of cancer.Mitochondria in Structural and Functional Cardiac Remodeling.Thioredoxin 2 Offers Protection against Mitochondrial Oxidative Stress in H9c2 Cells and against Myocardial Hypertrophy Induced by Hyperglycemia.ASK1-dependent endothelial cell activation is critical in ovarian cancer growth and metastasis.Beneficial effects of mycophenolate mofetil on cardiotoxicity induced by tacrolimus in wistar rats.Thioredoxin Reductase 2 (Txnrd2) Regulates Mitochondrial Integrity in the Progression of Age-Related Heart Failure.Melatonin and the electron transport chain.Mechanistic Role of Thioredoxin 2 in Heart Failure.The metabolic syndrome induces early changes in the swine renal medullary mitochondria.Hypoxia inhibits expression and function of mitochondrial thioredoxin 2 to promote pulmonary hypertension.Differences in Reperfusion-Induced Mitochondrial Oxidative Stress and Cell Death Between Hippocampal CA1 and CA3 Subfields Are Due to the Mitochondrial Thioredoxin System.SIRT4 accelerates Ang II-induced pathological cardiac hypertrophy by inhibiting manganese superoxide dismutase activity.Cardioprotective and nonprotective regimens of chronic hypoxia diversely affect the myocardial antioxidant systems.Intramyocardial injection of thioredoxin 2-expressing lentivirus alleviates myocardial ischemia-reperfusion injury in rats.The deubiquitinating enzyme TNFAIP3 mediates inactivation of hepatic ASK1 and ameliorates nonalcoholic steatohepatitis.ASK family in cardiovascular biology and medicine.An Extract from the Plant Deschampsia antarctica Protects Fibroblasts from Senescence Induced by Hydrogen Peroxide.Subcellular Reactive Oxygen Species (ROS) in Cardiovascular Pathophysiology.Temporal Effects of Combined Birinapant and Paclitaxel on Pancreatic Cancer Cells Investigated via Large-scale, Ion-Current-Based Quantitative Proteomics (IonStar).Cold exposure induced oxidative stress and apoptosis in the myocardium by inhibiting the Nrf2-Keap1 signaling pathway.Resolvin D1 attenuates liver ischaemia/reperfusion injury through modulating thioredoxin 2-mediated mitochondrial quality control.Mitochondria, Oxidative Stress and Innate Immunity Genetic Alterations in Oxidant and Anti-Oxidant Enzymes in the Vascular System

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Thioredoxin-2 inhibits mitochondrial reactive oxygen species generation and apoptosis stress kinase-1 activity to maintain cardiac function.

http://www.wikidata.org/entity/Q35217815

description

2015 nî lūn-bûn

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2015 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի հունվարին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

Thioredoxin-2 inhibits mitocho ...... to maintain cardiac function.

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Thioredoxin-2 inhibits mitocho ...... to maintain cardiac function.

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type

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Thioredoxin-2 inhibits mitocho ...... to maintain cardiac function.

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Thioredoxin-2 inhibits mitocho ...... to maintain cardiac function.

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Thioredoxin-2 inhibits mitocho ...... to maintain cardiac function.

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Thioredoxin-2 inhibits mitocho ...... to maintain cardiac function.

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Ford Hinojosa-Kirschenbaum

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Frank J Giordano

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George Tellides

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Grant R Budas

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Haifeng Zhang

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Huanjiao Jenny Zhou

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Lina Yao

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Luiz Belardinelli

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Peidong Fan

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Qunhua Huang

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P2860

Essential role for mitochondrial thioredoxin reductase in hematopoiesis, heart development, and heart function

Thioredoxin and ventricular remodeling

Mechanisms controlling mitochondrial biogenesis and respiration through the thermogenic coactivator PGC-1

Auranofin increases apoptosis and ischaemia-reperfusion injury in the rat isolated heart

A redox-dependent pathway for regulating class II HDACs and cardiac hypertrophy

Cytoplasmic thioredoxin reductase is essential for embryogenesis but dispensable for cardiac development

An apoptosis signal-regulating kinase 1 inhibitor reduces cardiomyocyte apoptosis and infarct size in a rat ischemia-reperfusion model

Regulation of cardiac hypertrophy by intracellular signalling pathways

Oxygen, oxidative stress, hypoxia, and heart failure.

A role for peroxisome proliferator-activated receptor γ coactivator-1 in the control of mitochondrial dynamics during postnatal cardiac growth

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1082-1097

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12

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2015-01-27T00:00:00Z

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25628390

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4374031

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