The outcome of polymicrobial sepsis is independent of T and B cells.
about
Immunosuppression after sepsis: systemic inflammation and sepsis induce a loss of naïve T-cells but no enduring cell-autonomous defects in T-cell functionTyrosine kinase 2 promotes sepsis-associated lethality by facilitating production of interleukin-27A historical perspective on sepsis.Potential role of myeloid cell/eosinophil-derived IL-17 in LPS-induced endotoxin shockRegulatory T cells are protective in systemic inflammation response syndrome induced by zymosan in miceMyD88-dependent production of IL-17F is modulated by the anaphylatoxin C5a via the Akt signaling pathway.Evidence for anti-inflammatory effects of C5a on the innate IL-17A/IL-23 axisAnti-inflammatory effects of β2 adrenergic receptor agonists in experimental acute lung injury.Complement activation product C5a is a selective suppressor of TLR4-induced, but not TLR3-induced, production of IL-27(p28) from macrophages.Th17 cells: critical mediators of host responses to burn injury and sepsisFingerprinting of the TLR4-induced acute inflammatory response.Experimental design of complement component 5a-induced acute lung injury (C5a-ALI): a role of CC-chemokine receptor type 5 during immune activation by anaphylatoxin.CD11c+ alveolar macrophages are a source of IL-23 during lipopolysaccharide-induced acute lung injury.Candida albicans-Staphylococcus aureus polymicrobial peritonitis modulates host innate immunity.WAVE1 mediates suppression of phagocytosis by phospholipid-derived DAMPs.The interaction between C5a and both C5aR and C5L2 receptors is required for production of G-CSF during acute inflammation.The immune response to severe bacterial infections: consequences for therapy.New insights for C5a and C5a receptors in sepsis.Pathways mediating resolution of inflammation: when enough is too much.Regulation of IL-17 family members by adrenal hormones during experimental sepsis in mice.
P2860
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P2860
The outcome of polymicrobial sepsis is independent of T and B cells.
description
2011 nî lūn-bûn
@nan
2011 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
The outcome of polymicrobial sepsis is independent of T and B cells.
@ast
The outcome of polymicrobial sepsis is independent of T and B cells.
@en
type
label
The outcome of polymicrobial sepsis is independent of T and B cells.
@ast
The outcome of polymicrobial sepsis is independent of T and B cells.
@en
prefLabel
The outcome of polymicrobial sepsis is independent of T and B cells.
@ast
The outcome of polymicrobial sepsis is independent of T and B cells.
@en
P2093
P2860
P1433
P1476
The outcome of polymicrobial sepsis is independent of T and B cells.
@en
P2093
J Vidya Sarma
Markus Bosmann
Norman F Russkamp
Vinay R Patel
P2860
P304
P356
10.1097/SHK.0B013E3182295F5F
P577
2011-10-01T00:00:00Z