Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function. - Wikidata - wikimedia - TriplyDB

Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function.

Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function.

http://www.wikidata.org/entity/Q35223050

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Cancer Stem Cells in Human Gastrointestinal Cancer Mitochondria, cholesterol and cancer cell metabolism Fundamentals of cancer metabolism Recommendations for myeloid-derived suppressor cell nomenclature and characterization standards Cancer stem cell metabolism Pancreatic cancer from bench to bedside: molecular pathways and treatment options Chronic Myeloid Leukemia and Hepatoblastoma: Two Cancer Models to Link Metabolism to Stem Cells Targeting dormant micrometastases: rationale, evidence to date and clinical implications Genetics and biology of pancreatic ductal adenocarcinoma Integration of Mitochondrial Targeting for Molecular Cancer Therapeutics Bypassing Mechanisms of Mitochondria-Mediated Cancer Stem Cells Resistance to Chemo- and Radiotherapy Neuroblastoma: oncogenic mechanisms and therapeutic exploitation of necroptosis Autophagy in colorectal cancer: An important switch from physiology to pathology Mitophagy programs: mechanisms and physiological implications of mitochondrial targeting by autophagy Pancreatic Cancer Metabolism: Breaking It Down to Build It Back Up Therapeutic strategies targeting cancer stem cells Metabolic reprogramming: the emerging concept and associated therapeutic strategies Cancer cells exploit adaptive mitochondrial dynamics to increase tumor cell invasion Treatment Strategies that Enhance the Efficacy and Selectivity of Mitochondria-Targeted Anticancer Agents Imaging and Therapy of Pancreatic Cancer with Phosphatidylserine-Targeted Nanovesicles Autophagy in malignant transformation and cancer progression Emerging strategies to effectively target autophagy in cancer Targeting mitochondria metabolism for cancer therapy Ductal pancreatic cancer modeling and drug screening using human pluripotent stem cell- and patient-derived tumor organoids.PI3K therapy reprograms mitochondrial trafficking to fuel tumor cell invasion.Survivin promotes oxidative phosphorylation, subcellular mitochondrial repositioning, and tumor cell invasion Metabolic plasticity underpins innate and acquired resistance to LDHA inhibition On metabolic reprogramming and tumor biology: A comprehensive survey of metabolism in breast cancer Imaging metabolic heterogeneity in cancer Cancer stem cell metabolism: a potential target for cancer therapy Concise Review: Stem Cells in Pancreatic Cancer: From Concept to Translation Metabolic pathways promoting cancer cell survival and growth Inhibition of Fatty Acid Oxidation Modulates Immunosuppressive Functions of Myeloid-Derived Suppressor Cells and Enhances Cancer Therapies Lacritin and other autophagy associated proteins in ocular surface health.Selective Vulnerability of Cancer Cells by Inhibition of Ca(2+) Transfer from Endoplasmic Reticulum to Mitochondria A neuronal network of mitochondrial dynamics regulates metastasis.Autocrine IGF1 Signaling Mediates Pancreatic Tumor Cell Dormancy in the Absence of Oncogenic Drivers.Targeted therapies for pancreatic adenocarcinoma: Where do we stand, how far can we go?Targeting autophagic cancer stem-cells to reverse chemoresistance in human triple negative breast cancer Diabetes, pancreatic cancer, and metformin therapy.

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P2860

Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function.

http://www.wikidata.org/entity/Q35223050

description

2014 nî lūn-bûn

@nan

2014 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի օգոստոսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function.

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Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function.

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type

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Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function.

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Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function.

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prefLabel

Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function.

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Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function.

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Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function.

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Andrea Viale

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Angela K Deem

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Avnish Kapoor

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Emanuela Brunetto

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Giannicola Genovese

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Giulio F Draetta

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Haoqiang Ying

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Huamin Wang

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Jason B Fleming

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Inhibition of Mutated, Activated BRAF in Metastatic Melanoma

Distinct populations of cancer stem cells determine tumor growth and metastatic activity in human pancreatic cancer

Identification of pancreatic cancer stem cells

Autophagy regulates lipid metabolism

Ras oncogenes: split personalities

Pancreatic cancers require autophagy for tumor growth

Pharmacologic inhibition of fatty acid oxidation sensitizes human leukemia cells to apoptosis induction

ALDH activity selectively defines an enhanced tumor-initiating cell population relative to CD133 expression in human pancreatic adenocarcinoma.

Generation of orthotopic and heterotopic human pancreatic cancer xenografts in immunodeficient mice

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7524

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Matteo Marchesini

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Alessandro Carugo

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264796234

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