Susceptibility to arrhythmia in the infarcted heart depends on myofibroblast density. - Wikidata - wikimedia - TriplyDB

Susceptibility to arrhythmia in the infarcted heart depends on myofibroblast density.

Susceptibility to arrhythmia in the infarcted heart depends on myofibroblast density.

http://www.wikidata.org/entity/Q35224007

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Computational medicine: translating models to clinical care Computational Representations of Myocardial Infarct Scars and Implications for Arrhythmogenesis Computational Approaches to Understanding the Role of Fibroblast-Myocyte Interactions in Cardiac Arrhythmogenesis Cross talk between cardiac myocytes and fibroblasts: from multiscale investigative approaches to mechanisms and functional consequences Simulation of Cardiac Arrhythmias Using a 2D Heterogeneous Whole Heart Model Spiral-wave dynamics in a mathematical model of human ventricular tissue with myocytes and fibroblasts.Prospectively quantifying the propensity for atrial fibrillation: a mechanistic formulation Electrophysiology of Heart Failure Using a Rabbit Model: From the Failing Myocyte to Ventricular Fibrillation.Electrophysiological and structural remodeling in heart failure modulate arrhythmogenesis. 2D simulation study Nonequilibrium arrhythmic states and transitions in a mathematical model for diffuse fibrosis in human cardiac tissue Physiological Implications of Myocardial Scar Structure Images as drivers of progress in cardiac computational modelling.Computational modeling of cardiac fibroblasts and fibrosis Reentry and Ectopic Pacemakers Emerge in a Three-Dimensional Model for a Slab of Cardiac Tissue with Diffuse Microfibrosis near the Percolation Threshold.Optical mapping of optogenetically shaped cardiac action potentials Quantitative analysis of cardiac tissue including fibroblasts using three-dimensional confocal microscopy and image reconstruction: towards a basis for electrophysiological modeling.Exploring susceptibility to atrial and ventricular arrhythmias resulting from remodeling of the passive electrical properties in the heart: a simulation approach Fibroblast electrical remodeling in heart failure and potential effects on atrial fibrillation Curcumin suppresses transforming growth factor-β1-induced cardiac fibroblast differentiation via inhibition of Smad-2 and p38 MAPK signaling pathways.Virtual electrophysiological study of atrial fibrillation in fibrotic remodeling Electrophysiological and structural remodeling in heart failure modulate arrhythmogenesis. 1D simulation study.Computational modeling of cardiac optogenetics: Methodology overview & review of findings from simulations The Living Scar--Cardiac Fibroblasts and the Injured Heart.The crossroads of inflammation, fibrosis, and arrhythmia following myocardial infarction Precisely parameterized experimental and computational models of tissue organization.Myocyte-fibroblast communication in cardiac fibrosis and arrhythmias: Mechanisms and model systems.Mechanistic inquiry into the role of tissue remodeling in fibrotic lesions in human atrial fibrillation.Cardiac fibrosis and arrhythmogenesis: the road to repair is paved with perils.Fibroblast-myocyte electrotonic coupling: does it occur in native cardiac tissue?Advances in modeling ventricular arrhythmias: from mechanisms to the clinic.Multiscale computational analysis of the bioelectric consequences of myocardial ischaemia and infarction.A role for membrane shape and information processing in cardiac physiology.How computer simulations of the human heart can improve anti-arrhythmia therapy.Inverse estimation of cardiac activation times via gradient-based optimization.Can heart function lost to disease be regenerated by therapeutic targeting of cardiac scar tissue?Cardiac Fibroblast Activation Post-Myocardial Infarction: Current Knowledge Gaps.Computational rabbit models to investigate the initiation, perpetuation, and termination of ventricular arrhythmia.Tachycardia in post-infarction hearts: insights from 3D image-based ventricular models.Forced fusion of human ventricular scar cells with cardiomyocytes suppresses arrhythmogenicity in a co-culture model.Na(+) current expression in human atrial myofibroblasts: identity and functional roles.

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P2860

Susceptibility to arrhythmia in the infarcted heart depends on myofibroblast density.

http://www.wikidata.org/entity/Q35224007

description

2011 nî lūn-bûn

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2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Susceptibility to arrhythmia in the infarcted heart depends on myofibroblast density.

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Susceptibility to arrhythmia in the infarcted heart depends on myofibroblast density.

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type

label

Susceptibility to arrhythmia in the infarcted heart depends on myofibroblast density.

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Susceptibility to arrhythmia in the infarcted heart depends on myofibroblast density.

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prefLabel

Susceptibility to arrhythmia in the infarcted heart depends on myofibroblast density.

@ast

Susceptibility to arrhythmia in the infarcted heart depends on myofibroblast density.

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J.BPJ.2011.08.009

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Biophysical Journal

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Susceptibility to arrhythmia in the infarcted heart depends on myofibroblast density

@en

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Hermenegild J Arevalo

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Kathleen S McDowell

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P2860

Activation and inactivation of a non-selective cation conductance by local mechanical deformation of acutely isolated cardiac fibroblasts.

Towards predictive modelling of the electrophysiology of the heart.

Electrotonic myofibroblast-to-myocyte coupling increases propensity to reentrant arrhythmias in two-dimensional cardiac monolayers.

Organization of fibroblasts in the heart.

Automatically generated, anatomically accurate meshes for cardiac electrophysiology problems.

Magnetic resonance-based anatomical analysis of scar-related ventricular tachycardia: implications for catheter ablation.

Electrical remodeling in ischemia and infarction.

Mapping of regional myocardial strain and work during ventricular pacing: experimental study using magnetic resonance imaging tagging.

Image-based models of cardiac structure in health and disease.

Modulation of cardiac gap junction expression and arrhythmic susceptibility.

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1307-1315

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10.1016/J.BPJ.2011.08.009

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Mary M Maleckar

Natalia A Trayanova

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