Adenosine monophosphate-activated kinase alpha1 promotes endothelial barrier repair. - Wikidata - wikimedia - TriplyDB

Adenosine monophosphate-activated kinase alpha1 promotes endothelial barrier repair.

Adenosine monophosphate-activated kinase alpha1 promotes endothelial barrier repair.

http://www.wikidata.org/entity/Q35225087

about

Pseudomonas aeruginosa exotoxin Y is a promiscuous cyclase that increases endothelial tau phosphorylation and permeability Adenosine A1 receptors promote vasa vasorum endothelial cell barrier integrity via Gi and Akt-dependent actin cytoskeleton remodeling AMPK: a master energy regulator for gonadal function.N-cadherin coordinates AMP kinase-mediated lung vascular repair.Store-operated Ca2+ entry (SOCE) induced by protease-activated receptor-1 mediates STIM1 protein phosphorylation to inhibit SOCE in endothelial cells through AMP-activated protein kinase and p38β mitogen-activated protein kinase Neuronal Wiskott-Aldrich syndrome protein regulates TGF-β1-mediated lung vascular permeability AMP-dependent kinase inhibits oxidative stress-induced caveolin-1 phosphorylation and endocytosis by suppressing the dissociation between c-Abl and Prdx1 proteins in endothelial cells Adenosine monophosphate-activated protein kinase is required for pulmonary artery smooth muscle cell survival and the development of hypoxic pulmonary hypertension Interaction of membrane/lipid rafts with the cytoskeleton: impact on signaling and function: membrane/lipid rafts, mediators of cytoskeletal arrangement and cell signaling.Metformin-stimulated AMPK-α1 promotes microvascular repair in acute lung injury.Connecting metabolism to intestinal barrier function: The role of leptin.Cerulein hyperstimulation decreases AMP-activated protein kinase levels at the site of maximal zymogen activation Factors controlling vascular permeability: transmitting mechanical signals. Focus on "Mechanical induction of group V phospholipase A₂ causes lung inflammation and acute lung injury".Targeting therapeutic effects: subcellular location matters. Focus on "Pharmacological AMP-kinase activators have compartment-specific effects on cell physiology".Permeability and calcium signaling in lung endothelium: unpack the box….Instillation of Hyaluronan Reverses Acid Instillation Injury to the Mammalian Blood Gas Barrier.Synergistic effect of smoking with genetic variants in the AMPKα1 gene on the risk of coronary artery disease in type 2 diabetes.

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Adenosine monophosphate-activated kinase alpha1 promotes endothelial barrier repair.

http://www.wikidata.org/entity/Q35225087

description

2011 nî lūn-bûn

@nan

2011 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի հունիսին հրատարակված գիտական հոդված

@hy

2011年の論文

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2011年学术文章

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2011年学术文章

@zh-cn

2011年学术文章

@zh-hans

2011年学术文章

@zh-my

2011年学术文章

@zh-sg

2011年學術文章

@yue

name

Adenosine monophosphate-activated kinase alpha1 promotes endothelial barrier repair.

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Adenosine monophosphate-activated kinase alpha1 promotes endothelial barrier repair.

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type

label

Adenosine monophosphate-activated kinase alpha1 promotes endothelial barrier repair.

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Adenosine monophosphate-activated kinase alpha1 promotes endothelial barrier repair.

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prefLabel

Adenosine monophosphate-activated kinase alpha1 promotes endothelial barrier repair.

@ast

Adenosine monophosphate-activated kinase alpha1 promotes endothelial barrier repair.

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Adenosine monophosphate-activated kinase alpha1 promotes endothelial barrier repair.

@en

P2093

Judy Creighton

http://www.w3.org/2001/XMLSchema#string

Mikhail Alexeyev

http://www.w3.org/2001/XMLSchema#string

MingYuan Jian

http://www.w3.org/2001/XMLSchema#string

Paul A Insel

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Sarah Sayner

http://www.w3.org/2001/XMLSchema#string

P2860

A high-throughput cell migration assay using scratch wound healing, a comparison of image-based readout methods

Spectrin-anchored phosphodiesterase 4D4 restricts cAMP from disrupting microtubules and inducing endothelial cell gap formation

Cav3.1 (alpha1G) controls von Willebrand factor secretion in rat pulmonary microvascular endothelial cells.

Production of high-titer helper-free retroviruses by transient transfection

Wound repair and regeneration

AMP-activated protein kinase (AMPK) activation and glycogen synthase kinase-3β (GSK-3β) inhibition induce Ca2+-independent deposition of tight junction components at the plasma membrane.

Contact-dependent promotion of cell migration by the OL-protocadherin-Nap1 interaction

Cell-cell adhesion in human fibroblasts requires calcium signaling.

N-cadherin association with lipid rafts regulates its dynamic assembly at cell-cell junctions in C2C12 myoblasts.

AMP-activated protein kinase activation as a strategy for protecting vascular endothelial function.

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3356-3365

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scholarly article

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10.1096/FJ.10-179218

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10

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25

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2011-06-16T00:00:00Z

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51226472

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21680893

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3177581

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