Advanced glycation end products are direct modulators of β-cell function. - Wikidata - wikimedia - TriplyDB

Advanced glycation end products are direct modulators of β-cell function.

Advanced glycation end products are direct modulators of β-cell function.

http://www.wikidata.org/entity/Q35227180

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Dietary Advanced Glycation End Products and Risk Factors for Chronic Disease: A Systematic Review of Randomised Controlled Trials Effects of Two Different Dietary Patterns on Inflammatory Markers, Advanced Glycation End Products and Lipids in Subjects without Type 2 Diabetes: A Randomised Crossover Study.TRB3 mediates advanced glycation end product-induced apoptosis of pancreatic β-cells through the protein kinase C β pathway.Effect of dietary prebiotic supplementation on advanced glycation, insulin resistance and inflammatory biomarkers in adults with pre-diabetes: a study protocol for a double-blind placebo-controlled randomised crossover clinical trial.Age-related impairment of pancreatic Beta-cell function: pathophysiological and cellular mechanisms.Pre-pregnancy fried food consumption and the risk of gestational diabetes mellitus: a prospective cohort study Acute exposure to a precursor of advanced glycation end products induces a dual effect on the rat pancreatic islet function.Advanced glycation end products and oxidative stress in type 2 diabetes mellitus.Sesamin Ameliorates Advanced Glycation End Products-Induced Pancreatic β-Cell Dysfunction and Apoptosis.Glycotoxin and autoantibodies are additive environmentally determined predictors of type 1 diabetes: a twin and population study.ATP synthase subunit-β down-regulation aggravates diabetic nephropathy.Oral advanced glycation endproducts (AGEs) promote insulin resistance and diabetes by depleting the antioxidant defenses AGE receptor-1 and sirtuin 1.Dietary Advanced Glycation End Products Consumption as a Direct Modulator of Insulin Sensitivity in Overweight Humans: A Study Protocol for a Double-Blind, Randomized, Two Period Cross-Over Trial.Heparanase induced by advanced glycation end products (AGEs) promotes macrophage migration involving RAGE and PI3K/AKT pathway Receptor for advanced glycation endproducts signaling cascades are activated in pancreatic fibroblasts, but not in the INS1E insulinoma cell line: Are mesenchymal cells major players in chronic inflammation?The pathological role of advanced glycation end products-downregulated heat shock protein 60 in islet β-cell hypertrophy and dysfunction Advanced glycation end products (AGE) and diabetes: cause, effect, or both?Coming full circle in diabetes mellitus: from complications to initiation.Dietary advanced glycation end-product restriction for the attenuation of insulin resistance, oxidative stress and endothelial dysfunction: a systematic review.Intracellular and extracellular adenosine triphosphate in regulation of insulin secretion from pancreatic β cells (β).A novel PPARgamma agonist monascin's potential application in diabetes prevention.Glycoxidation of biological macromolecules: a critical approach to halt the menace of glycation.Advanced Glycation End Products, Inflammation, and Chronic Metabolic Diseases: Links in a Chain?Induction of receptor for advanced glycation end products by insufficient leptin action triggers pancreatic β-cell failure in type 2 diabetes.Effect of diet-derived advanced glycation end products on inflammation.Inhibition of tumor suppressor p53 preserves glycation-serum induced pancreatic beta-cell demise.Physiological and therapeutic effects of carnosine on cardiometabolic risk and disease.Nε-(carboxymethyl) lysine-induced mitochondrial fission and mitophagy cause decreased insulin secretion from β-cells.Diabetic kidney disease: a role for advanced glycation end-product receptor 1 (AGE-R1)?Targeting advanced glycation with pharmaceutical agents: where are we now?Dietary factors in the development of type 1 diabetes.Receptor for Advanced Glycation End Products (RAGE) in Type 1 Diabetes Pathogenesis.Pancreatic islet inflammation: an emerging role for chemokines.Dietary Advanced Glycation End Products and Cardiometabolic Risk.Maternal intake of fried foods and risk of gestational diabetes mellitus.High-Sensitivity Cardiac Troponin T (hs-cTnT) as a Predictor of Incident Diabetes in the Atherosclerosis Risk in Communities Study.Is vitamin D deficiency related to accumulation of advanced glycation end products, markers of inflammation, and oxidative stress in diabetic subjects?High Serum Advanced Glycation End Products Are Associated with Decreased Insulin Secretion in Patients with Type 2 Diabetes: A Brief Report.Effect of Nɛ-carboxymethyllysine on oxidative stress and the glutathione system in beta cells.N-acetylcysteine Counteracts Adipose Tissue Macrophage Infiltration and Insulin Resistance Elicited by Advanced Glycated Albumin in Healthy Rats.

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Advanced glycation end products are direct modulators of β-cell function.

http://www.wikidata.org/entity/Q35227180

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2011 nî lūn-bûn

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2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Advanced glycation end products are direct modulators of β-cell function.

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Brian G Drew

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P2860

Tobacco smoke is a source of toxic reactive glycation products

HLA DR-DQ-encoded genetic determinants of childhood-onset type 1 diabetes in Finland: an analysis of 622 nuclear families

Advanced glycation end products cause epithelial-myofibroblast transdifferentiation via the receptor for advanced glycation end products (RAGE)

Stable expression of manganese superoxide dismutase (MnSOD) in insulinoma cells prevents IL-1beta- induced cytotoxicity and reduces nitric oxide production

Impaired mitochondrial activity in the insulin-resistant offspring of patients with type 2 diabetes

Renoprotective effects of a novel inhibitor of advanced glycation.

Maternally transmitted and food-derived glycotoxins: a factor preconditioning the young to diabetes?

Differential effect of inbred mouse strain (C57BL/6, DBA/2, 129T2) on insulin secretory function in response to a high fat diet.

Structure elucidation of a senescence cross-link from human extracellular matrix. Implication of pentoses in the aging process.

Accumulation of Maillard reaction products in skin collagen in diabetes and aging.

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51641315

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