Brg1 promotes both tumor-suppressive and oncogenic activities at distinct stages of pancreatic cancer formation. - Wikidata - wikimedia - TriplyDB

Brg1 promotes both tumor-suppressive and oncogenic activities at distinct stages of pancreatic cancer formation.

Brg1 promotes both tumor-suppressive and oncogenic activities at distinct stages of pancreatic cancer formation.

http://www.wikidata.org/entity/Q35228755

about

Genetics and biology of pancreatic ductal adenocarcinoma Developmental Pathways Direct Pancreatic Cancer Initiation from Its Cellular Origin Alterations of Epigenetic Regulators in Pancreatic Cancer and Their Clinical Implications Epigenetic treatment of pancreatic cancer: is there a therapeutic perspective on the horizon?Regulation of Cellular Identity in Cancer Epigenetic regulation of epithelial-mesenchymal transition BRG1 promotes hepatocarcinogenesis by regulating proliferation and invasiveness Pan-cancer analysis of somatic copy-number alterations implicates IRS4 and IGF2 in enhancer hijacking.Prognostic relevance of molecular subtypes and master regulators in pancreatic ductal adenocarcinoma Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based therapy for pancreatic ductal adenocarcinoma Targeting BET bromodomain proteins in solid tumors BET inhibitors block pancreatic stellate cell collagen I production and attenuate fibrosis in vivo.Stromal cues regulate the pancreatic cancer epigenome and metabolome.Neat1 is a p53-inducible lincRNA essential for transformation suppression.Targeting Transcriptional and Epigenetic Reprogramming in Stromal Cells in Fibrosis and Cancer.Combinatorial Screening of Pancreatic Adenocarcinoma Reveals Sensitivity to Drug Combinations Including Bromodomain Inhibitor Plus Neddylation Inhibitor.Exploiting receptor tyrosine kinase co-activation for cancer therapy.New insights in the development of pancreatic cancer.Cells of origin of pancreatic neoplasms.Pharmacotherapeutic Management of Pancreatic Ductal Adenocarcinoma: Current and Emerging Concepts.Differentiation and Inflammation: 'Best Enemies' in Gastrointestinal Carcinogenesis.ER stress protein AGR2 precedes and is involved in the regulation of pancreatic cancer initiation.Global Protease Activity Profiling Provides Differential Diagnosis of Pancreatic Cysts.ANGPTL4 promotes the progression of cutaneous melanoma to brain metastasis.Evaluation of the role of downregulation of SNF5/INI1 core subunit of SWI/SNF complex in clear cell renal cell carcinoma development.Arid1a Has Context-Dependent Oncogenic and Tumor Suppressor Functions in Liver Cancer.The BRG1 ATPase of human SWI/SNF chromatin remodeling enzymes as a driver of cancer.Systematic In Vivo Inactivation of Chromatin-Regulating Enzymes Identifies Setd2 as a Potent Tumor Suppressor in Lung Adenocarcinoma.Pancreatic cancer: A matter of timing.Arid1a restrains Kras-dependent changes in acinar cell identity.The BRG1/SOX9 axis is critical for acinar cell-derived pancreatic tumorigenesis Epigenetics of gastrointestinal diseases: notes from a workshop SCF-mediated degradation of Brg1 suppresses gastric cancer metastasis

P2860

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P2860

Brg1 promotes both tumor-suppressive and oncogenic activities at distinct stages of pancreatic cancer formation.

http://www.wikidata.org/entity/Q35228755

description

2015 nî lūn-bûn

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2015 թուականի Մարտին հրատարակուած գիտական յօդուած

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2015 թվականի մարտին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

Brg1 promotes both tumor-suppr ...... f pancreatic cancer formation.

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Brg1 promotes both tumor-suppr ...... f pancreatic cancer formation.

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type

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Brg1 promotes both tumor-suppr ...... f pancreatic cancer formation.

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Brg1 promotes both tumor-suppr ...... f pancreatic cancer formation.

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Brg1 promotes both tumor-suppr ...... f pancreatic cancer formation.

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Brg1 promotes both tumor-suppr ...... f pancreatic cancer formation.

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Genes & Development

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Brg1 promotes both tumor-suppr ...... f pancreatic cancer formation.

@en

P2093

Atsushi Urano

http://www.w3.org/2001/XMLSchema#string

David W Dawson

http://www.w3.org/2001/XMLSchema#string

E Scott Seeley

http://www.w3.org/2001/XMLSchema#string

Eric A Collisson

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Guido Von Figura

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Karina E Villanueva

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Matthias Hebrok

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Nilotpal Roy

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Shivani Malik

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Xinyuan Lu

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P2860

The spectrum of SWI/SNF mutations, ubiquitous in human cancers

Purification and characterization of mSin3A-containing Brg1 and hBrm chromatin remodeling complexes

Kras(G12D) and Smad4/Dpc4 haploinsufficiency cooperate to induce mucinous cystic neoplasms and invasive adenocarcinoma of the pancreas

Selective inhibition of BET bromodomains

HMGA2 and Smads co-regulate SNAIL1 expression during induction of epithelial-to-mesenchymal transition

PANTHER in 2013: modeling the evolution of gene function, and other gene attributes, in the context of phylogenetic trees

Convergent structural alterations define SWItch/Sucrose NonFermentable (SWI/SNF) chromatin remodeler as a central tumor suppressive complex in pancreatic cancer

TopHat: discovering splice junctions with RNA-Seq

mSin3A/histone deacetylase 2- and PRMT5-containing Brg1 complex is involved in transcriptional repression of the Myc target gene cad

Pancreatic ductal cells in development, regeneration, and neoplasia

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658-671

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scholarly article

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10.1101/GAD.256628.114

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6

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29

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2015-03-01T00:00:00Z

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273778599

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25792600

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4378197

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