Mitochondrial Ca(2+) uptake is essential for synaptic plasticity in pain.
about
Ionotropic glutamate receptors and voltage-gated Ca²⁺ channels in long-term potentiation of spinal dorsal horn synapses and pain hypersensitivityCell signaling and mitochondrial dynamics: Implications for neuronal function and neurodegenerative diseaseReactive Oxygen Species: Physiological and Physiopathological Effects on Synaptic PlasticityUse of Capsaicin to Treat Pain: Mechanistic and Therapeutic ConsiderationsUptake and mitochondrial dysfunction of alpha-synuclein in human astrocytes, cortical neurons and fibroblasts.Carbonic anhydrase-8 regulates inflammatory pain by inhibiting the ITPR1-cytosolic free calcium pathway.Induction of long-term potentiation and long-term depression is cell-type specific in the spinal cord.Ca2+-mediated mitochondrial reactive oxygen species metabolism augments Wnt/β-catenin pathway activation to facilitate cell differentiation.Drp1-mediated mitochondrial abnormalities link to synaptic injury in diabetes model.Mechanisms of G protein-coupled estrogen receptor-mediated spinal nociception.HSV vector-mediated GAD67 suppresses neuropathic pain induced by perineural HIV gp120 in rats through inhibition of ROS and Wnt5aInvolvement of reactive oxygen species in cocaine-taking behaviors in ratsSex differences in opioid analgesia and addiction: interactions among opioid receptors and estrogen receptors.Oxidative Stress as a Physiological Pain Response in Full-Term NewbornsMitochondrial CaMKII inhibition in airway epithelium protects against allergic asthma.Deconstructing the neuropathic pain phenotype to reveal neural mechanisms.Neuronal calcium signaling in chronic pain.Reactive oxygen species affect spinal cell type-specific synaptic plasticity in a model of neuropathic pain.Mitochondrial dynamics in neuronal injury, development and plasticity.MnSOD mediated by HSV vectors in the periaqueductal gray suppresses morphine withdrawal in rats.Pharmacological Modulation of the Mitochondrial Electron Transport Chain in Paclitaxel-Induced Painful Peripheral Neuropathy.Miro1-dependent mitochondrial positioning drives the rescaling of presynaptic Ca2+ signals during homeostatic plasticity.The role of TRPM2 in hydrogen peroxide-induced expression of inflammatory cytokine and chemokine in rat trigeminal ganglia.Reactive oxygen species (ROS) modulate AMPA receptor phosphorylation and cell-surface localization in concert with pain-related behavior.Gene therapy with HSV encoding p55TNFR gene for HIV neuropathic pain: an evidence-based mini-review.Phosphorylated CCAAT/Enhancer Binding Protein β Contributes to Rat HIV-Related Neuropathic Pain: In Vitro and In Vivo Studies.Sciatic nerve ligation causes impairment of mitochondria associated with changes in distribution, respiration, and cardiolipin composition in related spinal cord neurons in rats.
P2860
Q26823531-67A32954-DB1F-46D5-971A-ACDCC5FD2B8BQ26853356-9D48BF73-3680-4216-8227-53B42ABC0D7FQ28070007-5D0FC279-4D4E-4C9F-A6FC-F3EF8C563AC0Q28073741-18675D88-1B17-43AA-A263-FA82074DDA21Q35008102-714170A8-D0D6-44D2-AE44-48FF29D300BBQ35142140-02CDB3AD-28F8-4894-B74A-8469C79E52AFQ35193964-D49905D7-5F24-45BE-99A4-70DA12925D04Q35225170-CA24DD57-B248-4D93-A4C7-73511842DA69Q35532253-A47C7D70-D35D-4440-BE45-55583B10504BQ36140716-D96A03FE-BFDF-4169-9608-26DA683E3225Q36777541-CEAD4935-18FD-4087-92A7-76D61CD0309BQ36832327-B0C8B6FF-F222-4E79-826B-225A97164C50Q37352118-1A64DF07-DF6A-4062-B34A-4465539BFBE6Q37590841-147B3F26-0A79-4DD2-AA52-17DBDE356CCEQ37624358-BF0508FC-724F-4A81-ACE3-9E0B7378760AQ37988158-1DD9CF6B-61CB-4B0A-B5D9-84BF8A7D886BQ38228215-5847EAD4-84BC-4BC3-AB82-3347EAD444A1Q38678930-96B23057-A4D6-4527-8637-E01EEC459500Q39116596-24E255BC-7C08-41F7-A03D-73656E1CFB2FQ40262488-4A3F92F1-9489-4549-BE03-4BE106240F65Q41184407-261237E5-DEE1-4FE2-831C-13A1A7600E04Q41865556-7EDD7C07-868F-470C-A7C4-CA0836288CC1Q42143373-28335638-EB4C-4692-90DB-707C3E9E78ECQ42265352-D3486A0A-7952-4C6F-BC46-EF1188CE82D0Q45874354-6356BFC6-49FC-456D-ACE7-468DC0A33D5FQ47340505-6862EBE8-0C06-4F55-A39D-FCD089F2BE0BQ50230679-8866AA7E-132D-42BD-AF4C-47F1E9333653
P2860
Mitochondrial Ca(2+) uptake is essential for synaptic plasticity in pain.
description
2011 nî lūn-bûn
@nan
2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Mitochondrial Ca(2+) uptake is essential for synaptic plasticity in pain.
@ast
Mitochondrial Ca(2+) uptake is essential for synaptic plasticity in pain.
@en
type
label
Mitochondrial Ca(2+) uptake is essential for synaptic plasticity in pain.
@ast
Mitochondrial Ca(2+) uptake is essential for synaptic plasticity in pain.
@en
prefLabel
Mitochondrial Ca(2+) uptake is essential for synaptic plasticity in pain.
@ast
Mitochondrial Ca(2+) uptake is essential for synaptic plasticity in pain.
@en
P2093
P2860
P1476
Mitochondrial Ca(2+) uptake is essential for synaptic plasticity in pain.
@en
P2093
Hee Young Kim
Jigong Wang
Jin Mo Chung
Kwan Yeop Lee
Kyungsoon Chung
Sang Jeong Kim
P2860
P304
12982-12991
P356
10.1523/JNEUROSCI.3093-11.2011
P407
P577
2011-09-01T00:00:00Z