Activation of corticostriatal circuitry relieves chronic neuropathic pain. - Wikidata - wikimedia - TriplyDB

Activation of corticostriatal circuitry relieves chronic neuropathic pain.

Activation of corticostriatal circuitry relieves chronic neuropathic pain.

http://www.wikidata.org/entity/Q35236871

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A randomized placebo-controlled pilot study of the efficacy and safety of D-cycloserine in people with chronic back pain.Corticolimbic anatomical characteristics predetermine risk for chronic pain Chronic pain induces generalized enhancement of aversion Corticostriatal Regulation of Acute Pain Neuropathic pain promotes adaptive changes in gene expression in brain networks involved in stress and depression Deactivation of excitatory neurons in the prelimbic cortex via Cdk5 promotes pain sensation and anxiety.Bidirectional modulation of hyperalgesia via the specific control of excitatory and inhibitory neuronal activity in the ACC Rescue of Impaired mGluR5-Driven Endocannabinoid Signaling Restores Prefrontal Cortical Output to Inhibit Pain in Arthritic Rats.Specific Targeting of the Basolateral Amygdala to Projectionally Defined Pyramidal Neurons in Prelimbic and Infralimbic Cortex.Dynamic Changes of Functional Pain Connectome in Women with Primary Dysmenorrhea.The indirect pathway of the nucleus accumbens shell amplifies neuropathic pain The Emotional Brain as a Predictor and Amplifier of Chronic Pain Interleukin-1β overproduction is a common cause for neuropathic pain, memory deficit, and depression following peripheral nerve injury in rodents Estrogen-dependent visceral hypersensitivity following stress in rats: An fMRI study Quantifying cerebral contributions to pain beyond nociception.Emerging Treatments for Neuropathic Pain.Modulation of pain, nociception, and analgesia by the brain reward center.Exercise-induced hypoalgesia: potential mechanisms in animal models of neuropathic pain.Brain activity for tactile allodynia: a longitudinal awake rat functional magnetic resonance imaging study tracking emergence of neuropathic pain.AMPAkines and morphine provide complementary analgesia.Will optogenetics be used to treat chronic pain patients?Corticolimbic circuitry in the modulation of chronic pain and substance abuse.Modeling Pain Using fMRI: From Regions to Biomarkers.Reduced Glutamatergic Currents and Dendritic Branching of Layer 5 Pyramidal Cells Contribute to Medial Prefrontal Cortex Deactivation in a Rat Model of Neuropathic Pain.Early life stress induces sex-dependent increases in phosphorylated extracellular signal-regulated kinase in brains of mice with neuropathic pain.Affective Circuitry Alterations in Patients with Trigeminal Neuralgia.Inhibition of anterior cingulate cortex excitatory neuronal activity induces conditioned place preference in a mouse model of chronic inflammatory pain.Ethanol Increases Mechanical Pain Sensitivity in Rats via Activation of GABAA Receptors in Medial Prefrontal Cortex.Identifying brain nociceptive information transmission in patients with chronic somatic pain.Loss of M1 Receptor Dependent Cholinergic Excitation Contributes to mPFC Deactivation in Neuropathic Pain.Spotlight on pain: optogenetic approaches for interrogating somatosensory circuits.AMPAkines Target the Nucleus Accumbens to Relieve Postoperative Pain.Mechanisms of placebo analgesia: A dual-process model informed by insights from cross-species comparisons.Deciphering neuronal population codes for acute thermal pain.Mu Opioid Receptors in Gamma-Aminobutyric Acidergic Forebrain Neurons Moderate Motivation for Heroin and Palatable Food.Real-time particle filtering and smoothing algorithms for detecting abrupt changes in neural ensemble spike activity.A pathway from midcingulate cortex to posterior insula gates nociceptive hypersensitivity.Sodium channels in pain disorders: pathophysiology and prospects for treatment.Hypersensitivity of Prelimbic Cortex Neurons Contributes to Aggravated Nociceptive Responses in Rats With Experience of Chronic Inflammatory Pain.Local field potential decoding of the onset and intensity of acute pain in rats.

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Activation of corticostriatal circuitry relieves chronic neuropathic pain.

http://www.wikidata.org/entity/Q35236871

description

2015 nî lūn-bûn

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2015 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2015 թվականի ապրիլին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

Activation of corticostriatal circuitry relieves chronic neuropathic pain.

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Activation of corticostriatal circuitry relieves chronic neuropathic pain.

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Activation of corticostriatal circuitry relieves chronic neuropathic pain.

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Activation of corticostriatal circuitry relieves chronic neuropathic pain.

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prefLabel

Activation of corticostriatal circuitry relieves chronic neuropathic pain.

@ast

Activation of corticostriatal circuitry relieves chronic neuropathic pain.

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JNEUROSCI.3494-14.2015

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Journal of Neuroscience

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Activation of corticostriatal circuitry relieves chronic neuropathic pain

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Hau Yueh Lin

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James D'amour

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Karl Deisseroth

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Michelle Lee

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Sarah E Eberle

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Toby R Manders

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P2860

Neuromodulation of thought: flexibilities and vulnerabilities in prefrontal cortical network synapses

Rapid regulation of depression-related behaviours by control of midbrain dopamine neurons

Neurocircuitry of addiction

Targeting abnormal neural circuits in mood and anxiety disorders: from the laboratory to the clinic

The brain in chronic CRPS pain: abnormal gray-white matter interactions in emotional and autonomic regions

Cognitive and emotional control of pain and its disruption in chronic pain

Efficacy of transcranial magnetic stimulation targets for depression is related to intrinsic functional connectivity with the subgenual cingulate

The development and application of optogenetics

Quantitative assessment of tactile allodynia in the rat paw

Animal models of neuropsychiatric disorders

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scholarly article

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10.1523/JNEUROSCI.3494-14.2015

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13

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Robert C Froemke

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neuropathic pain

chronic neuropathic pain

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