Global analysis of fungal morphology exposes mechanisms of host cell escape. - Wikidata - wikimedia - TriplyDB

Global analysis of fungal morphology exposes mechanisms of host cell escape.

Global analysis of fungal morphology exposes mechanisms of host cell escape.

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Adaptations of the Secretome of Candida albicans in Response to Host-Related Environmental Conditions Systems Level Dissection of Candida Recognition by Dectins: A Matter of Fungal Morphology and Site of Infection Turning Up the Heat: Inflammasome Activation by Fungal Pathogens Characterization of Virulence-Related Phenotypes in Candida Species of the CUG Clade.Cell biology of Candida albicans-host interactions Mitochondrial Activity and Cyr1 Are Key Regulators of Ras1 Activation of C. albicans Virulence Pathways Functional Divergence of Hsp90 Genetic Interactions in Biofilm and Planktonic Cellular States Global Analysis of the Fungal Microbiome in Cystic Fibrosis Patients Reveals Loss of Function of the Transcriptional Repressor Nrg1 as a Mechanism of Pathogen Adaptation Mapping the Hsp90 Genetic Network Reveals Ergosterol Biosynthesis and Phosphatidylinositol-4-Kinase Signaling as Core Circuitry Governing Cellular Stress Metal Chelation as a Powerful Strategy to Probe Cellular Circuitry Governing Fungal Drug Resistance and Morphogenesis Opportunistic pathogen Candida albicans elicits a temporal response in primary human mast cells.The Candida albicans ATO Gene Family Promotes Neutralization of the Macrophage Phagolysosome Signaling through Lrg1, Rho1 and Pkc1 Governs Candida albicans Morphogenesis in Response to Diverse Cues.Functional Genomic Analysis of Candida albicans Adherence Reveals a Key Role for the Arp2/3 Complex in Cell Wall Remodelling and Biofilm Formation.Bypass of Candida albicans Filamentation/Biofilm Regulators through Diminished Expression of Protein Kinase Cak1.Genome-wide functional analysis in Candida albicans.The Endoplasmic Reticulum-Mitochondrion Tether ERMES Orchestrates Fungal Immune Evasion, Illuminating Inflammasome Responses to Hyphal Signals.The synthesis, regulation, and functions of sterols in Candida albicans: Well-known but still lots to learn.Candida albicans Dbf4-dependent Cdc7 kinase plays a novel role in the inhibition of hyphal development.Robust Extracellular pH Modulation by Candida albicans during Growth in Carboxylic Acids.Genome-Wide Screen for Haploinsufficient Cell Size Genes in the Opportunistic Yeast Candida albicans.Extensive functional redundancy in the regulation of Candida albicans drug resistance and morphogenesis by lysine deacetylases Hos2, Hda1, Rpd3 and Rpd31.Specific pathways mediating inflammasome activation by Candida parapsilosis.Staurosporine Induces Filamentation in the Human Fungal Pathogen Candida albicans via Signaling through Cyr1 and Protein Kinase A.Phagosomal Neutralization by the Fungal Pathogen Candida albicans Induces Macrophage Pyroptosis.Strategies in the discovery of novel antifungal scaffolds.Alarmin(g) the innate immune system to invasive fungal infections Filamentation protects Candida albicans from amphotericin B-induced programmed cell death via a mechanism involving the yeast metacaspase, MCA1.N-Acetylglucosamine Metabolism Promotes Survival of Candida albicans in the Phagosome.Chemical inhibitors of Candida albicans hyphal morphogenesis target endocytosis.A functional link between hyphal maintenance and quorum sensing in Candida albicans.IL-1α is Critical for Resistance Against Highly Virulent Aspergillus fumigatus Isolates.Ydj1 governs fungal morphogenesis and stress response, and facilitates mitochondrial protein import via Mas1 and Mas2.Chemogenomic profiling of the fungal pathogen Candida albicans.Filamentation Involves Two Overlapping, but Distinct, Programs of Filamentation in the Pathogenic Fungus Candida albicans.Morphology Changes in Human Fungal Pathogens upon Interaction with the Host.Candida albicans Hyphae: From Growth Initiation to Invasion.Negative control of Candida albicans filamentation-associated gene expression by essential protein kinase gene KIN28.Tuning Hsf1 levels drives distinct fungal morphogenetic programs with depletion impairing Hsp90 function and overexpression expanding the target space.Candida albicans-epithelial interactions and induction of mucosal innate immunity.

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P2860

Global analysis of fungal morphology exposes mechanisms of host cell escape.

http://www.wikidata.org/entity/Q35246478

description

2015 nî lūn-bûn

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2015 թուականի Մարտին հրատարակուած գիտական յօդուած

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2015 թվականի մարտին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

Global analysis of fungal morphology exposes mechanisms of host cell escape.

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Global analysis of fungal morphology exposes mechanisms of host cell escape.

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type

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Global analysis of fungal morphology exposes mechanisms of host cell escape.

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Global analysis of fungal morphology exposes mechanisms of host cell escape.

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prefLabel

Global analysis of fungal morphology exposes mechanisms of host cell escape.

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Global analysis of fungal morphology exposes mechanisms of host cell escape.

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Nature Communications

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Global analysis of fungal morphology exposes mechanisms of host cell escape.

@en

P2093

Bo Jiang

http://www.w3.org/2001/XMLSchema#string

Terry Roemer

http://www.w3.org/2001/XMLSchema#string

Troy Ketela

http://www.w3.org/2001/XMLSchema#string

P2860

Plasmodium falciparum erythrocyte invasion: combining function with immune evasion

A human-curated annotation of the Candida albicans genome.

The Cryptococcus neoformans capsule: a sword and a shield.

Functional characterization of the S. cerevisiae genome by gene deletion and parallel analysis

Translocation of lipid-linked oligosaccharides across the ER membrane requires Rft1 protein.

Genome-wide fitness test and mechanism-of-action studies of inhibitory compounds in Candida albicans

Fitness trade-offs restrict the evolution of resistance to amphotericin B

Nonfilamentous C. albicans mutants are avirulent

Forward genetics in Candida albicans that reveals the Arp2/3 complex is required for hyphal formation, but not endocytosis.

Systematic screens of a Candida albicans homozygous deletion library decouple morphogenetic switching and pathogenicity.

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6741

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scholarly article

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10.1038/NCOMMS7741

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6

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Teresa R O'Meara

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2015-03-31T00:00:00Z

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274317110

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1026536887

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25824284

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4382923

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