NLRC4 and TLR5 each contribute to host defense in respiratory melioidosis.
about
The role of innate immunity receptors in the pathogenesis of inflammatory bowel diseaseTherapeutic Administration of a Monoclonal Anti-Il-1β Antibody Protects Against Experimental Melioidosis.Role of Canonical and Non-canonical Inflammasomes During Burkholderia Infection.Type III Secretion in the Melioidosis Pathogen Burkholderia pseudomallei.NLRC4 regulates caspase-1 and IL-1beta production in a CD11blowLy6Glow population of cells required for resistance to Pseudomonas aeruginosa keratitis.Immune response to recombinant Burkholderia pseudomallei FliC.
P2860
NLRC4 and TLR5 each contribute to host defense in respiratory melioidosis.
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2014 nî lūn-bûn
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2014 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի սեպտեմբերին հրատարակված գիտական հոդված
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年论文
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name
NLRC4 and TLR5 each contribute to host defense in respiratory melioidosis.
@ast
NLRC4 and TLR5 each contribute to host defense in respiratory melioidosis.
@en
type
label
NLRC4 and TLR5 each contribute to host defense in respiratory melioidosis.
@ast
NLRC4 and TLR5 each contribute to host defense in respiratory melioidosis.
@en
prefLabel
NLRC4 and TLR5 each contribute to host defense in respiratory melioidosis.
@ast
NLRC4 and TLR5 each contribute to host defense in respiratory melioidosis.
@en
P2093
P2860
P50
P1476
NLRC4 and TLR5 each contribute to host defense in respiratory melioidosis
@en
P2093
Direk Limmathurotsakul
H Denny Liggitt
Narisara Chantratita
Nicolle D Myers
Sharon J Peacock
Shawn J Skerrett
Vanaporn Wuthiekanun
P2860
P356
10.1371/JOURNAL.PNTD.0003178
P407
P577
2014-09-18T00:00:00Z