Integrative analysis of type-I and type-II aberrations underscores the genetic heterogeneity of pediatric acute myeloid leukemia - Wikidata - wikimedia - TriplyDB

Integrative analysis of type-I and type-II aberrations underscores the genetic heterogeneity of pediatric acute myeloid leukemia

Integrative analysis of type-I and type-II aberrations underscores the genetic heterogeneity of pediatric acute myeloid leukemia

http://www.wikidata.org/entity/Q35265916

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Pediatric AML: From Biology to Clinical Management RAS mutations in early age leukaemia modulated by NQO1 rs1800566 (C609T) are associated with second-hand smoking exposures.Prognostic significance of FLT3-ITD in pediatric acute myeloid leukemia: a meta-analysis of cohort studies.Telomere length and telomerase complex mutations in pediatric acute myeloid leukemia.Regulation of CDX4 gene transcription by HoxA9, HoxA10, the Mll-Ell oncogene and Shp2 during leukemogenesis Global characteristics of childhood acute promyelocytic leukemia.Gene expression profiles associated with pediatric relapsed AML.Collaborative Efforts Driving Progress in Pediatric Acute Myeloid Leukemia.Recurrent deletions of IKZF1 in pediatric acute myeloid leukemia MicroRNA-106b~25 cluster is upregulated in relapsed MLL-rearranged pediatric acute myeloid leukemia.RNAi-mediated silencing of MLL-AF9 reveals leukemia-associated downstream targets and processes.Novel therapies for children with acute myeloid leukaemia.Relevance of leukemic stem cells in acute myeloid leukemia: heterogeneity and influence on disease monitoring, prognosis and treatment design.Targeting novel signaling pathways for resistant acute myeloid leukemia.Acute myeloid leukemia in infants: biology and treatment Insights in dynamic kinome reprogramming as a consequence of MEK inhibition in MLL-rearranged AML.Prognostic Significance of NRAS Gene Mutations in Children with Acute Myelogenous Leukemia.Recurrent translocation t(10;17)(p15;q21) in minimally differentiated acute myeloid leukemia results in ZMYND11/MBTD1 fusion.NPM1, FLT3 and CEBPA mutations in pediatric patients with AML from Argentina: incidence and prognostic value.The applicability of the WHO classification in paediatric AML. A NOPHO-AML study.BCOR and BCORL1 mutations in pediatric acute myeloid leukemia.Mapping epigenetic regulator gene mutations in cytogenetically normal pediatric acute myeloid leukemia Low frequency of type-I and type-II aberrations in myeloid leukemia of Down syndrome, underscoring the unique entity of this disease.The clinical relevance of BAALC and ERG expression levels in pediatric AML.The molecular landscape of pediatric acute myeloid leukemia reveals recurrent structural alterations and age-specific mutational interactions.Lentivirus-mediated RNA interference targeting FAMLF-1 inhibits cell growth and enhances cell differentiation of acute myeloid leukemia partially differentiated cells via inhibition of AKT and c-MYC.Prognostic value of genetic mutations in adolescent and young adults with acute myeloid leukemia.Analysis of class I and II aberrations in Iraqi childhood acute myeloid leukemia using filter paper cards.miR-9 is a tumor suppressor in pediatric AML with t(8;21).Childhood acute myeloid leukemia with bone marrow eosinophilia caused by t(16;21)(q24;q22).NUP98-NSD1 fusion in association with FLT3-ITD mutation identifies a prognostically relevant subgroup of pediatric acute myeloid leukemia patients suitable for monitoring by real time quantitative PCR.Unique BHLHB3 overexpression in pediatric acute myeloid leukemia with t(6;11)(q27;q23).Changes in cytogenetics and molecular genetics in acute myeloid leukemia from childhood to adult age groups.Cooperation of MLL/AF10(OM-LZ) with PTPN11 activating mutation induced monocytic leukemia with a shorter latency in a mouse bone marrow transplantation model.Clinical and prognostic significance of eosinophilia and inv(16)/t(16;16) in pediatric acute myelomonocytic leukemia (AML-M4).Clinical relevance of molecular aberrations in paediatric acute myeloid leukaemia at first relapse.Impact of mutations in FLT3, PTPN11 and RAS genes on the overall survival of pediatric B cell precursor acute lymphoblastic leukemia in Brazil.CBLmutations do not frequently occur in paediatric acute myeloid leukaemia

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Integrative analysis of type-I and type-II aberrations underscores the genetic heterogeneity of pediatric acute myeloid leukemia

http://www.wikidata.org/entity/Q35265916

description

2011 nî lūn-bûn

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2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2011 թվականի հուլիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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Integrative analysis of type-I ...... diatric acute myeloid leukemia

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Integrative analysis of type-I ...... diatric acute myeloid leukemia

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Integrative analysis of type-I ...... diatric acute myeloid leukemia

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Integrative analysis of type-I ...... diatric acute myeloid leukemia

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HAEMATOL.2010.038976

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Integrative analysis of type-I ...... diatric acute myeloid leukemia

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Andre Baruchel

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Anne R M von Bergh

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Brian V Balgobind

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C Michel Zwaan

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Dirk Reinhardt

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Gertjan J L Kaspers

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H Berna Beverloo

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Iris H I M Hollink

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Jacqueline Cloos

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Jan Stary

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P2860

The 2008 revision of the World Health Organization (WHO) classification of myeloid neoplasms and acute leukemia: rationale and important changes

Prevalence and prognostic implications of CEBPA mutations in pediatric acute myeloid leukemia (AML): a report from the Children's Oncology Group

DNMT3A Mutations in Acute Myeloid Leukemia

Activating mutation of D835 within the activation loop of FLT3 in human hematologic malignancies.

Molecular alterations of the IDH1 gene in AML: a Children's Oncology Group and Southwest Oncology Group study.

A FLT3-targeted tyrosine kinase inhibitor is cytotoxic to leukemia cells in vitro and in vivo

Knock-in of an internal tandem duplication mutation into murine FLT3 confers myeloproliferative disease in a mouse model

Prevalence and prognostic significance of Flt3 internal tandem duplication in pediatric acute myeloid leukemia.

Prevalence and prognostic significance of KIT mutations in pediatric patients with core binding factor AML enrolled on serial pediatric cooperative trials for de novo AML.

PML/RARalpha and FLT3-ITD induce an APL-like disease in a mouse model

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1478-1487

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10.3324/HAEMATOL.2010.038976

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10

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96

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Marry M van den Heuvel-Eibrink

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2011-07-26T00:00:00Z

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51523281

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21791472

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