Truncated peroxisome proliferator-activated receptor-γ coactivator 1α splice variant is severely altered in Huntington's disease - Wikidata - wikimedia - TriplyDB

Truncated peroxisome proliferator-activated receptor-γ coactivator 1α splice variant is severely altered in Huntington's disease

Truncated peroxisome proliferator-activated receptor-γ coactivator 1α splice variant is severely altered in Huntington's disease

http://www.wikidata.org/entity/Q35268092

about

Electron Transport Disturbances and Neurodegeneration: From Albert Szent-Györgyi's Concept (Szeged) till Novel Approaches to Boost Mitochondrial Bioenergetics PGC-1α, mitochondrial dysfunction, and Huntington's disease Shaping the role of mitochondria in the pathogenesis of Huntington's disease Mitochondrial dysfunction in neurodegenerative diseases Effect of exercise intensity on isoform-specific expressions of NT-PGC-1 α mRNA in mouse skeletal muscle A greatly extended PPARGC1A genomic locus encodes several new brain-specific isoforms and influences Huntington disease age of onset.Brain diabetic neurodegeneration segregates with low intrinsic aerobic capacity Pharmacologic activation of mitochondrial biogenesis exerts widespread beneficial effects in a transgenic mouse model of Huntington's disease Hypothalamic overexpression of mutant huntingtin causes dysregulation of brown adipose tissue.Potential roles of PINK1 for increased PGC-1α-mediated mitochondrial fatty acid oxidation and their associations with Alzheimer disease and diabetes.Enhanced mitochondrial biogenesis ameliorates disease phenotype in a full-length mouse model of Huntington's disease.Antioxidants in Huntington's disease.Prospects for neuroprotective therapies in prodromal Huntington's disease.Transcriptional coactivator NT-PGC-1α promotes gluconeogenic gene expression and enhances hepatic gluconeogenesis.mRNA expression levels of PGC-1α in a transgenic and a toxin model of Huntington's disease.Transcriptional Dysregulation and Post-translational Modifications in Polyglutamine Diseases: From Pathogenesis to Potential Therapeutic Strategies.Mitochondrial Quality Control in Neurodegenerative Diseases: Focus on Parkinson's Disease and Huntington's Disease.Mitochondrial Morphology, Function and Homeostasis Are Impaired by Expression of an N-terminal Calpain Cleavage Fragment of Ataxin-3

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P2860

Truncated peroxisome proliferator-activated receptor-γ coactivator 1α splice variant is severely altered in Huntington's disease

http://www.wikidata.org/entity/Q35268092

description

2011 nî lūn-bûn

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2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի հուլիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Truncated peroxisome prolifera ...... ltered in Huntington's disease

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Truncated peroxisome prolifera ...... ltered in Huntington's disease

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type

label

Truncated peroxisome prolifera ...... ltered in Huntington's disease

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Truncated peroxisome prolifera ...... ltered in Huntington's disease

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Truncated peroxisome prolifera ...... ltered in Huntington's disease

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Truncated peroxisome prolifera ...... ltered in Huntington's disease

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Neurodegenerative Diseases

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Truncated peroxisome prolifera ...... ltered in Huntington's disease

@en

P2093

Abhijeet Sharma

http://www.w3.org/2001/XMLSchema#string

Abhishek Chandra

http://www.w3.org/2001/XMLSchema#string

Ashu Johri

http://www.w3.org/2001/XMLSchema#string

Lichuan Yang

http://www.w3.org/2001/XMLSchema#string

M Flint Beal

http://www.w3.org/2001/XMLSchema#string

Sara Orobello

http://www.w3.org/2001/XMLSchema#string

Thomas Hennessey

http://www.w3.org/2001/XMLSchema#string

P2860

Nutrient control of glucose homeostasis through a complex of PGC-1alpha and SIRT1

Peroxisome proliferator-activated receptor gamma coactivator 1 coactivators, energy homeostasis, and metabolism

Metabolic control of muscle mitochondrial function and fatty acid oxidation through SIRT1/PGC-1alpha

Defects in adaptive energy metabolism with CNS-linked hyperactivity in PGC-1alpha null mice

Exon 1 of the HD gene with an expanded CAG repeat is sufficient to cause a progressive neurological phenotype in transgenic mice

Isoform-specific increases in murine skeletal muscle peroxisome proliferator-activated receptor-gamma coactivator-1alpha (PGC-1alpha) mRNA in response to beta2-adrenergic receptor activation and exercise.

Reduced creatine kinase as a central and peripheral biomarker in Huntington's disease

Mitochondrial loss, dysfunction and altered dynamics in Huntington's disease

The energetics of Huntington's disease.

Transcriptional abnormalities in Huntington disease.

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496-503

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scholarly article

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10.1159/000327910

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6

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8

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Ferdinando Squitieri

Anatoly Starkov

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2011-07-15T00:00:00Z

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51493663

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21757867

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Huntington disease

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3186722

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